Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis
Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW...
Ausführliche Beschreibung
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| Autor*in: |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1990 |
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| Umfang: |
4 |
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| Reproduktion: |
Springer Online Journal Archives 1860-2002 |
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| Übergeordnetes Werk: |
in: Pediatric nephrology - 1987, 4(1990) vom: Juni, Seite 593-596 |
| Übergeordnetes Werk: |
volume:4 ; year:1990 ; month:06 ; pages:593-596 ; extent:4 |
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| 520 | |a Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. | ||
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(DE-627)NLEJ205953921 DE-627 ger DE-627 rakwb eng Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis 1990 4 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. Springer Online Journal Archives 1860-2002 Igarashi, Takashi oth Kawato, Hidehiko oth Kamoshita, Shigehiko oth in Pediatric nephrology 1987 4(1990) vom: Juni, Seite 593-596 (DE-627)NLEJ188983775 (DE-600)1463004-7 1432-198X nnns volume:4 year:1990 month:06 pages:593-596 extent:4 http://dx.doi.org/10.1007/BF00858629 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 4 1990 6 593-596 4 |
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(DE-627)NLEJ205953921 DE-627 ger DE-627 rakwb eng Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis 1990 4 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. Springer Online Journal Archives 1860-2002 Igarashi, Takashi oth Kawato, Hidehiko oth Kamoshita, Shigehiko oth in Pediatric nephrology 1987 4(1990) vom: Juni, Seite 593-596 (DE-627)NLEJ188983775 (DE-600)1463004-7 1432-198X nnns volume:4 year:1990 month:06 pages:593-596 extent:4 http://dx.doi.org/10.1007/BF00858629 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 4 1990 6 593-596 4 |
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(DE-627)NLEJ205953921 DE-627 ger DE-627 rakwb eng Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis 1990 4 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. Springer Online Journal Archives 1860-2002 Igarashi, Takashi oth Kawato, Hidehiko oth Kamoshita, Shigehiko oth in Pediatric nephrology 1987 4(1990) vom: Juni, Seite 593-596 (DE-627)NLEJ188983775 (DE-600)1463004-7 1432-198X nnns volume:4 year:1990 month:06 pages:593-596 extent:4 http://dx.doi.org/10.1007/BF00858629 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 4 1990 6 593-596 4 |
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(DE-627)NLEJ205953921 DE-627 ger DE-627 rakwb eng Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis 1990 4 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. Springer Online Journal Archives 1860-2002 Igarashi, Takashi oth Kawato, Hidehiko oth Kamoshita, Shigehiko oth in Pediatric nephrology 1987 4(1990) vom: Juni, Seite 593-596 (DE-627)NLEJ188983775 (DE-600)1463004-7 1432-198X nnns volume:4 year:1990 month:06 pages:593-596 extent:4 http://dx.doi.org/10.1007/BF00858629 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 4 1990 6 593-596 4 |
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(DE-627)NLEJ205953921 DE-627 ger DE-627 rakwb eng Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis 1990 4 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. Springer Online Journal Archives 1860-2002 Igarashi, Takashi oth Kawato, Hidehiko oth Kamoshita, Shigehiko oth in Pediatric nephrology 1987 4(1990) vom: Juni, Seite 593-596 (DE-627)NLEJ188983775 (DE-600)1463004-7 1432-198X nnns volume:4 year:1990 month:06 pages:593-596 extent:4 http://dx.doi.org/10.1007/BF00858629 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 4 1990 6 593-596 4 |
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reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis |
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Reversible low-molecular-weight proteinuria in patients with distal renal tubular acidosis |
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Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. |
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Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. |
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Abstract Four patients with untreated renal tubular acidosis had a urinary excretion of low-molecular-weight (LMW) proteins which was restored to normal by alkali therapy. Hypokalaemic proximal tubular damage in untreated patients with distal renal tubular acidosis is believed to be the cause of LMW proteinuria. An examination of urinary excretion of LMW proteins is useful for determining hypokalaemic proximal tubular dysfunction, as well as the efficiency of alkali therapy. |
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