The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases
[Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disord...
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E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
s.l.: Nature Publishing Group ; 1993 |
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| Umfang: |
8 |
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| Reproduktion: |
Nature Archives 1869 - 2009 |
|---|---|
| Übergeordnetes Werk: |
In: Nature |
| Übergeordnetes Werk: |
volume:361 ; year:1993 ; number:6409 ; month:02 ; pages:226-233 ; extent:8 |
| Links: |
|---|
| DOI / URN: |
10.1038/361226a0 |
|---|
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| 520 | |a [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... | ||
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10.1038/361226a0 doi (DE-627)NLEJ235138886 DE-627 ger DE-627 rakwb eng The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases [s.l.] Nature Publishing Group 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... Nature Archives 1869 - 2009 Vetrie, David oth Vořechovský, Igor oth Sideras, Paschalis oth Holland, Jill oth Davies, Angela oth Flinter, Frances oth Hammarström, Lennart oth Kinnon, Christine oth Levinsky, Roland oth Bobrow, Martin oth Smith, C. I. Edvard oth Bentley, David R. oth In Nature <London> London [u.a.] : Nature Publising Group, 1869 361(1993), 6409 vom: Feb., Seite 226-233 (DE-627)NLEJ227682335 (DE-600)1413423-8 1476-4687 nnns volume:361 year:1993 number:6409 month:02 pages:226-233 extent:8 http://dx.doi.org/10.1038/361226a0 application/pdf Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-NTA GBV_NL_ARTICLE AR 361 1993 6409 2 226-233 8 |
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10.1038/361226a0 doi (DE-627)NLEJ235138886 DE-627 ger DE-627 rakwb eng The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases [s.l.] Nature Publishing Group 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... Nature Archives 1869 - 2009 Vetrie, David oth Vořechovský, Igor oth Sideras, Paschalis oth Holland, Jill oth Davies, Angela oth Flinter, Frances oth Hammarström, Lennart oth Kinnon, Christine oth Levinsky, Roland oth Bobrow, Martin oth Smith, C. I. Edvard oth Bentley, David R. oth In Nature <London> London [u.a.] : Nature Publising Group, 1869 361(1993), 6409 vom: Feb., Seite 226-233 (DE-627)NLEJ227682335 (DE-600)1413423-8 1476-4687 nnns volume:361 year:1993 number:6409 month:02 pages:226-233 extent:8 http://dx.doi.org/10.1038/361226a0 application/pdf Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-NTA GBV_NL_ARTICLE AR 361 1993 6409 2 226-233 8 |
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10.1038/361226a0 doi (DE-627)NLEJ235138886 DE-627 ger DE-627 rakwb eng The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases [s.l.] Nature Publishing Group 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... Nature Archives 1869 - 2009 Vetrie, David oth Vořechovský, Igor oth Sideras, Paschalis oth Holland, Jill oth Davies, Angela oth Flinter, Frances oth Hammarström, Lennart oth Kinnon, Christine oth Levinsky, Roland oth Bobrow, Martin oth Smith, C. I. Edvard oth Bentley, David R. oth In Nature <London> London [u.a.] : Nature Publising Group, 1869 361(1993), 6409 vom: Feb., Seite 226-233 (DE-627)NLEJ227682335 (DE-600)1413423-8 1476-4687 nnns volume:361 year:1993 number:6409 month:02 pages:226-233 extent:8 http://dx.doi.org/10.1038/361226a0 application/pdf Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-NTA GBV_NL_ARTICLE AR 361 1993 6409 2 226-233 8 |
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10.1038/361226a0 doi (DE-627)NLEJ235138886 DE-627 ger DE-627 rakwb eng The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases [s.l.] Nature Publishing Group 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... Nature Archives 1869 - 2009 Vetrie, David oth Vořechovský, Igor oth Sideras, Paschalis oth Holland, Jill oth Davies, Angela oth Flinter, Frances oth Hammarström, Lennart oth Kinnon, Christine oth Levinsky, Roland oth Bobrow, Martin oth Smith, C. I. Edvard oth Bentley, David R. oth In Nature <London> London [u.a.] : Nature Publising Group, 1869 361(1993), 6409 vom: Feb., Seite 226-233 (DE-627)NLEJ227682335 (DE-600)1413423-8 1476-4687 nnns volume:361 year:1993 number:6409 month:02 pages:226-233 extent:8 http://dx.doi.org/10.1038/361226a0 application/pdf Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-NTA GBV_NL_ARTICLE AR 361 1993 6409 2 226-233 8 |
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10.1038/361226a0 doi (DE-627)NLEJ235138886 DE-627 ger DE-627 rakwb eng The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases [s.l.] Nature Publishing Group 1993 8 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier [Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... Nature Archives 1869 - 2009 Vetrie, David oth Vořechovský, Igor oth Sideras, Paschalis oth Holland, Jill oth Davies, Angela oth Flinter, Frances oth Hammarström, Lennart oth Kinnon, Christine oth Levinsky, Roland oth Bobrow, Martin oth Smith, C. I. Edvard oth Bentley, David R. oth In Nature <London> London [u.a.] : Nature Publising Group, 1869 361(1993), 6409 vom: Feb., Seite 226-233 (DE-627)NLEJ227682335 (DE-600)1413423-8 1476-4687 nnns volume:361 year:1993 number:6409 month:02 pages:226-233 extent:8 http://dx.doi.org/10.1038/361226a0 application/pdf Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-NTA GBV_NL_ARTICLE AR 361 1993 6409 2 226-233 8 |
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[Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... |
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[Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... |
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[Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ... |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">NLEJ235138886</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20210706210644.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">110225s1993 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1038/361226a0</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)NLEJ235138886</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">The gene involved in X-linked agammaglobulinaemia is a member of the src family of protein-tyrosine kinases</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="a">[s.l.]</subfield><subfield code="b">Nature Publishing Group</subfield><subfield code="c">1993</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">8</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">[Auszug] X-linked agammaglobulinaemia (XLA) is a human immunodeficiency caused by failure of pre-B cells in the bone marrow to develop into circulating mature B cells. A novel gene has been isolated which maps to the XLA locus, is expressed in B cells, and shows mutations in families with the disorder. The ...</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="f">Nature Archives 1869 - 2009</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Vetrie, David</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Vořechovský, Igor</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Sideras, Paschalis</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Holland, Jill</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Davies, Angela</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Flinter, Frances</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Hammarström, Lennart</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Kinnon, Christine</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Levinsky, Roland</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Bobrow, Martin</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Smith, C. I. Edvard</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Bentley, David R.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">In</subfield><subfield code="t">Nature <London></subfield><subfield code="d">London [u.a.] : Nature Publising Group, 1869</subfield><subfield code="g">361(1993), 6409 vom: Feb., Seite 226-233</subfield><subfield code="w">(DE-627)NLEJ227682335</subfield><subfield code="w">(DE-600)1413423-8</subfield><subfield code="x">1476-4687</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:361</subfield><subfield code="g">year:1993</subfield><subfield code="g">number:6409</subfield><subfield code="g">month:02</subfield><subfield code="g">pages:226-233</subfield><subfield code="g">extent:8</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">http://dx.doi.org/10.1038/361226a0</subfield><subfield code="q">application/pdf</subfield><subfield code="z">Deutschlandweit zugänglich</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-NTA</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">361</subfield><subfield code="j">1993</subfield><subfield code="e">6409</subfield><subfield code="c">2</subfield><subfield code="h">226-233</subfield><subfield code="g">8</subfield></datafield></record></collection>
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