Periodontal epidemiology: towards social science or molecular biology?
Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Baelum, Vibeke [verfasserIn] Lopez, Rodrigo [verfasserIn] |
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| Format: |
E-Artikel |
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| Erschienen: |
Oxford, UK: Munksgaard International Publishers ; 2004 |
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| Schlagwörter: |
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| Umfang: |
Online-Ressource |
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| Reproduktion: |
2004 ; Blackwell Publishing Journal Backfiles 1879-2005 |
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| Übergeordnetes Werk: |
In: Community dentistry and oral epidemiology - Oxford [u.a.] : Blackwell, 1973, 32(2004), 4, Seite 0 |
| Übergeordnetes Werk: |
volume:32 ; year:2004 ; number:4 ; pages:0 |
| Links: |
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| DOI / URN: |
10.1111/j.1600-0528.2004.00159.x |
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| 520 | |a Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. | ||
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10.1111/j.1600-0528.2004.00159.x doi (DE-627)NLEJ242629628 DE-627 ger DE-627 rakwb Baelum, Vibeke verfasserin aut Periodontal epidemiology: towards social science or molecular biology? Oxford, UK Munksgaard International Publishers 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| epidemiology Lopez, Rodrigo verfasserin aut In Community dentistry and oral epidemiology Oxford [u.a.] : Blackwell, 1973 32(2004), 4, Seite 0 Online-Ressource (DE-627)NLEJ243927207 (DE-600)2027101-3 1600-0528 nnns volume:32 year:2004 number:4 pages:0 http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 32 2004 4 0 |
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10.1111/j.1600-0528.2004.00159.x doi (DE-627)NLEJ242629628 DE-627 ger DE-627 rakwb Baelum, Vibeke verfasserin aut Periodontal epidemiology: towards social science or molecular biology? Oxford, UK Munksgaard International Publishers 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| epidemiology Lopez, Rodrigo verfasserin aut In Community dentistry and oral epidemiology Oxford [u.a.] : Blackwell, 1973 32(2004), 4, Seite 0 Online-Ressource (DE-627)NLEJ243927207 (DE-600)2027101-3 1600-0528 nnns volume:32 year:2004 number:4 pages:0 http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 32 2004 4 0 |
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10.1111/j.1600-0528.2004.00159.x doi (DE-627)NLEJ242629628 DE-627 ger DE-627 rakwb Baelum, Vibeke verfasserin aut Periodontal epidemiology: towards social science or molecular biology? Oxford, UK Munksgaard International Publishers 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| epidemiology Lopez, Rodrigo verfasserin aut In Community dentistry and oral epidemiology Oxford [u.a.] : Blackwell, 1973 32(2004), 4, Seite 0 Online-Ressource (DE-627)NLEJ243927207 (DE-600)2027101-3 1600-0528 nnns volume:32 year:2004 number:4 pages:0 http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 32 2004 4 0 |
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10.1111/j.1600-0528.2004.00159.x doi (DE-627)NLEJ242629628 DE-627 ger DE-627 rakwb Baelum, Vibeke verfasserin aut Periodontal epidemiology: towards social science or molecular biology? Oxford, UK Munksgaard International Publishers 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| epidemiology Lopez, Rodrigo verfasserin aut In Community dentistry and oral epidemiology Oxford [u.a.] : Blackwell, 1973 32(2004), 4, Seite 0 Online-Ressource (DE-627)NLEJ243927207 (DE-600)2027101-3 1600-0528 nnns volume:32 year:2004 number:4 pages:0 http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 32 2004 4 0 |
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10.1111/j.1600-0528.2004.00159.x doi (DE-627)NLEJ242629628 DE-627 ger DE-627 rakwb Baelum, Vibeke verfasserin aut Periodontal epidemiology: towards social science or molecular biology? Oxford, UK Munksgaard International Publishers 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| epidemiology Lopez, Rodrigo verfasserin aut In Community dentistry and oral epidemiology Oxford [u.a.] : Blackwell, 1973 32(2004), 4, Seite 0 Online-Ressource (DE-627)NLEJ243927207 (DE-600)2027101-3 1600-0528 nnns volume:32 year:2004 number:4 pages:0 http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 32 2004 4 0 |
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Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. |
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Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. |
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Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes. |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">NLEJ242629628</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230505205639.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">120427s2004 xx |||||o 00| ||und c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1111/j.1600-0528.2004.00159.x</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)NLEJ242629628</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Baelum, Vibeke</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Periodontal epidemiology: towards social science or molecular biology?</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="a">Oxford, UK</subfield><subfield code="b">Munksgaard International Publishers</subfield><subfield code="c">2004</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Abstract – Terms such as ‘molecular epidemiology’ and ‘genetic epidemiology’ have been coined to depict the change from ‘traditional epidemiology’, concerned with disease determinants at the community or society level, over to ‘modern epidemiology’, which is concerned with determinants operating at the individual level or even below, i.e. at the organ, tissue, cell, or molecular level. In this commentary, we point out to the limitations of this development and suggest that more emphasis is placed on making the presumed causal disease models explicit, when investigating the relationship between putative determinants and disease. Understanding the disease processes at the micro-level is insufficient for understanding disease at the individual level; and disease patterns at the population level cannot be understood unless it is realized that individuals exist in a variety of contexts that cannot be reduced to individual attributes.</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="d">2004</subfield><subfield code="f">Blackwell Publishing Journal Backfiles 1879-2005</subfield><subfield code="7">|2004||||||||||</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">epidemiology</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Lopez, Rodrigo</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">In</subfield><subfield code="t">Community dentistry and oral epidemiology</subfield><subfield code="d">Oxford [u.a.] : Blackwell, 1973</subfield><subfield code="g">32(2004), 4, Seite 0</subfield><subfield code="h">Online-Ressource</subfield><subfield code="w">(DE-627)NLEJ243927207</subfield><subfield code="w">(DE-600)2027101-3</subfield><subfield code="x">1600-0528</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:32</subfield><subfield code="g">year:2004</subfield><subfield code="g">number:4</subfield><subfield code="g">pages:0</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">http://dx.doi.org/10.1111/j.1600-0528.2004.00159.x</subfield><subfield code="q">text/html</subfield><subfield code="x">Verlag</subfield><subfield code="z">Deutschlandweit zugänglich</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-DJB</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">32</subfield><subfield code="j">2004</subfield><subfield code="e">4</subfield><subfield code="h">0</subfield></datafield></record></collection>
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