Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome:
Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-l...
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HISAMATSU, KENICHI - M.D. KUSANO, KENGO FUKUSHIMA - M.D. MORITA, HIROSHI - M.D. TAKENAKA, SHIHO - M.D. NAGASE, SATOSHI - M.D. NAKAMURA, KAZUFUMI - M.D. EMORI, TETSURO - M.D. MATSUBARA, HIROMI - M.D. MIKOUCHI, HIROSHI - M.D. NISHIZAKI, YOSHITOMO - M.D. OHE, TOHRU - M.D. |
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E-Artikel |
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350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK .: Blackwell Science Inc ; 2004 |
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Online-Ressource |
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2004 ; Blackwell Publishing Journal Backfiles 1879-2005 |
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In: Journal of cardiovascular electrophysiology - Oxford : Wiley-Blackwell, 1990, 15(2004), 8, Seite 0 |
| Übergeordnetes Werk: |
volume:15 ; year:2004 ; number:8 ; pages:0 |
| Links: |
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| DOI / URN: |
10.1046/j.1540-8167.2004.03675.x |
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NLEJ243345763 |
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| 245 | 1 | 0 | |a Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: |
| 264 | 1 | |a 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . |b Blackwell Science Inc |c 2004 | |
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| 520 | |a Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. | ||
| 533 | |d 2004 |f Blackwell Publishing Journal Backfiles 1879-2005 |7 |2004|||||||||| | ||
| 700 | 1 | |a HISAMATSU, KENICHI |c M.D. |4 oth | |
| 700 | 1 | |a KUSANO, KENGO FUKUSHIMA |c M.D. |4 oth | |
| 700 | 1 | |a MORITA, HIROSHI |c M.D. |4 oth | |
| 700 | 1 | |a TAKENAKA, SHIHO |c M.D. |4 oth | |
| 700 | 1 | |a NAGASE, SATOSHI |c M.D. |4 oth | |
| 700 | 1 | |a NAKAMURA, KAZUFUMI |c M.D. |4 oth | |
| 700 | 1 | |a EMORI, TETSURO |c M.D. |4 oth | |
| 700 | 1 | |a MATSUBARA, HIROMI |c M.D. |4 oth | |
| 700 | 1 | |a MIKOUCHI, HIROSHI |c M.D. |4 oth | |
| 700 | 1 | |a NISHIZAKI, YOSHITOMO |c M.D. |4 oth | |
| 700 | 1 | |a OHE, TOHRU |c M.D. |4 oth | |
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10.1046/j.1540-8167.2004.03675.x doi (DE-627)NLEJ243345763 DE-627 ger DE-627 rakwb Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . Blackwell Science Inc 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| HISAMATSU, KENICHI M.D. oth KUSANO, KENGO FUKUSHIMA M.D. oth MORITA, HIROSHI M.D. oth TAKENAKA, SHIHO M.D. oth NAGASE, SATOSHI M.D. oth NAKAMURA, KAZUFUMI M.D. oth EMORI, TETSURO M.D. oth MATSUBARA, HIROMI M.D. oth MIKOUCHI, HIROSHI M.D. oth NISHIZAKI, YOSHITOMO M.D. oth OHE, TOHRU M.D. oth In Journal of cardiovascular electrophysiology Oxford : Wiley-Blackwell, 1990 15(2004), 8, Seite 0 Online-Ressource (DE-627)NLEJ243926243 (DE-600)2037519-0 1540-8167 nnns volume:15 year:2004 number:8 pages:0 http://dx.doi.org/10.1046/j.1540-8167.2004.03675.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 15 2004 8 0 |
| spelling |
10.1046/j.1540-8167.2004.03675.x doi (DE-627)NLEJ243345763 DE-627 ger DE-627 rakwb Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . Blackwell Science Inc 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| HISAMATSU, KENICHI M.D. oth KUSANO, KENGO FUKUSHIMA M.D. oth MORITA, HIROSHI M.D. oth TAKENAKA, SHIHO M.D. oth NAGASE, SATOSHI M.D. oth NAKAMURA, KAZUFUMI M.D. oth EMORI, TETSURO M.D. oth MATSUBARA, HIROMI M.D. oth MIKOUCHI, HIROSHI M.D. oth NISHIZAKI, YOSHITOMO M.D. oth OHE, TOHRU M.D. oth In Journal of cardiovascular electrophysiology Oxford : Wiley-Blackwell, 1990 15(2004), 8, Seite 0 Online-Ressource (DE-627)NLEJ243926243 (DE-600)2037519-0 1540-8167 nnns volume:15 year:2004 number:8 pages:0 http://dx.doi.org/10.1046/j.1540-8167.2004.03675.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 15 2004 8 0 |
| allfields_unstemmed |
10.1046/j.1540-8167.2004.03675.x doi (DE-627)NLEJ243345763 DE-627 ger DE-627 rakwb Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . Blackwell Science Inc 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| HISAMATSU, KENICHI M.D. oth KUSANO, KENGO FUKUSHIMA M.D. oth MORITA, HIROSHI M.D. oth TAKENAKA, SHIHO M.D. oth NAGASE, SATOSHI M.D. oth NAKAMURA, KAZUFUMI M.D. oth EMORI, TETSURO M.D. oth MATSUBARA, HIROMI M.D. oth MIKOUCHI, HIROSHI M.D. oth NISHIZAKI, YOSHITOMO M.D. oth OHE, TOHRU M.D. oth In Journal of cardiovascular electrophysiology Oxford : Wiley-Blackwell, 1990 15(2004), 8, Seite 0 Online-Ressource (DE-627)NLEJ243926243 (DE-600)2037519-0 1540-8167 nnns volume:15 year:2004 number:8 pages:0 http://dx.doi.org/10.1046/j.1540-8167.2004.03675.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 15 2004 8 0 |
| allfieldsGer |
10.1046/j.1540-8167.2004.03675.x doi (DE-627)NLEJ243345763 DE-627 ger DE-627 rakwb Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . Blackwell Science Inc 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| HISAMATSU, KENICHI M.D. oth KUSANO, KENGO FUKUSHIMA M.D. oth MORITA, HIROSHI M.D. oth TAKENAKA, SHIHO M.D. oth NAGASE, SATOSHI M.D. oth NAKAMURA, KAZUFUMI M.D. oth EMORI, TETSURO M.D. oth MATSUBARA, HIROMI M.D. oth MIKOUCHI, HIROSHI M.D. oth NISHIZAKI, YOSHITOMO M.D. oth OHE, TOHRU M.D. oth In Journal of cardiovascular electrophysiology Oxford : Wiley-Blackwell, 1990 15(2004), 8, Seite 0 Online-Ressource (DE-627)NLEJ243926243 (DE-600)2037519-0 1540-8167 nnns volume:15 year:2004 number:8 pages:0 http://dx.doi.org/10.1046/j.1540-8167.2004.03675.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 15 2004 8 0 |
| allfieldsSound |
10.1046/j.1540-8167.2004.03675.x doi (DE-627)NLEJ243345763 DE-627 ger DE-627 rakwb Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: 350 Main Street , Malden , MA 02148-5018 , USA , and 9600 Garsington Road , Oxford OX4 2DQ , UK . Blackwell Science Inc 2004 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. 2004 Blackwell Publishing Journal Backfiles 1879-2005 |2004|||||||||| HISAMATSU, KENICHI M.D. oth KUSANO, KENGO FUKUSHIMA M.D. oth MORITA, HIROSHI M.D. oth TAKENAKA, SHIHO M.D. oth NAGASE, SATOSHI M.D. oth NAKAMURA, KAZUFUMI M.D. oth EMORI, TETSURO M.D. oth MATSUBARA, HIROMI M.D. oth MIKOUCHI, HIROSHI M.D. oth NISHIZAKI, YOSHITOMO M.D. oth OHE, TOHRU M.D. oth In Journal of cardiovascular electrophysiology Oxford : Wiley-Blackwell, 1990 15(2004), 8, Seite 0 Online-Ressource (DE-627)NLEJ243926243 (DE-600)2037519-0 1540-8167 nnns volume:15 year:2004 number:8 pages:0 http://dx.doi.org/10.1046/j.1540-8167.2004.03675.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 15 2004 8 0 |
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HISAMATSU, KENICHI @@oth@@ KUSANO, KENGO FUKUSHIMA @@oth@@ MORITA, HIROSHI @@oth@@ TAKENAKA, SHIHO @@oth@@ NAGASE, SATOSHI @@oth@@ NAKAMURA, KAZUFUMI @@oth@@ EMORI, TETSURO @@oth@@ MATSUBARA, HIROMI @@oth@@ MIKOUCHI, HIROSHI @@oth@@ NISHIZAKI, YOSHITOMO @@oth@@ OHE, TOHRU @@oth@@ |
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Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. 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Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome |
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relationships between depolarization abnormality and repolarization abnormality in patients with brugada syndrome |
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Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: |
| abstract |
Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. |
| abstractGer |
Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. |
| abstract_unstemmed |
Introduction: Repolarization and depolarization abnormalities have been reported to be related to Brugada syndrome. Methods and Results: We evaluated the relationships between repolarization abnormality and depolarization abnormality using 48-lead unipolar signal-averaged electrocardiograms and 87-lead unipolar body surface maps in 15 patients with Brugada-type ECGs. Data were compared with those from healthy control subjects (n = 5) and within subgroups of Brugada syndrome with (n = 8) and without (n = 7) ventricular arrhythmias (VA) induced by programmed electrical stimulation (PES). Eighty-seven-lead body surface maps were recorded, and potential maps were constructed to evaluate elevation of the ST segment 20 ms after the J point. Forty-eight-lead signal-averaged ECGs were recorded, and isochronal maps of duration of the delayed potential (dDP) were constructed to evaluate the dDP in each lead. Potential maps showed that patients with Brugada-type ECG, especially those with VA induced by programmed electrical stimulation, had greater elevation of the ST segment in the right ventricular outflow tract, especially at E5. Isochronal maps of dDP in the Brugada-type ECG group showed that maximum dDP was located at E5 and that the area with long dDP was larger than that in the control subjects. The dDPs at E7, E5, F7, and F5 in the VA-inducible group were significantly longer than those in the VA-noninducible group. These results showed that the location of greater elevation in the ST segment coincided with the location of longer dDP. Conclusion: Repolarization abnormality and depolarization abnormality in the walls of both ventricles, especially in the right ventricular outflow tract, are related to the VA of Brugada syndrome. |
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Relationships Between Depolarization Abnormality and Repolarization Abnormality in Patients with Brugada Syndrome: |
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HISAMATSU, KENICHI M.D. KUSANO, KENGO FUKUSHIMA M.D. MORITA, HIROSHI M.D. TAKENAKA, SHIHO M.D. NAGASE, SATOSHI M.D. NAKAMURA, KAZUFUMI M.D. EMORI, TETSURO M.D. MATSUBARA, HIROMI M.D. MIKOUCHI, HIROSHI M.D. NISHIZAKI, YOSHITOMO M.D. OHE, TOHRU M.D. |
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HISAMATSU, KENICHI M.D. KUSANO, KENGO FUKUSHIMA M.D. MORITA, HIROSHI M.D. TAKENAKA, SHIHO M.D. NAGASE, SATOSHI M.D. NAKAMURA, KAZUFUMI M.D. EMORI, TETSURO M.D. MATSUBARA, HIROMI M.D. MIKOUCHI, HIROSHI M.D. NISHIZAKI, YOSHITOMO M.D. OHE, TOHRU M.D. |
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