A Case Study: Identifying a New Case of Wilson's Disease
To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient edu...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Noble, Jeannette A. - RN, BSN [verfasserIn] |
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| Format: |
E-Artikel |
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| Erschienen: |
Oxford, UK; Malden, USA: Blackwell Science Inc ; 2005 |
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| Schlagwörter: |
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| Umfang: |
Online-Ressource |
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| Reproduktion: |
2005 ; Blackwell Publishing Journal Backfiles 1879-2005 |
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| Übergeordnetes Werk: |
In: Journal of the American Academy of Nurse Practitioners - Malden, Mass. [u.a.] : Wiley-Blackwell, 1989, 17(2005), 12, Seite 0 |
| Übergeordnetes Werk: |
volume:17 ; year:2005 ; number:12 ; pages:0 |
| Links: |
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| DOI / URN: |
10.1111/j.1745-7599.2005.00089.x |
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NLEJ243494971 |
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| 520 | |a To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. | ||
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10.1111/j.1745-7599.2005.00089.x doi (DE-627)NLEJ243494971 DE-627 ger DE-627 rakwb Noble, Jeannette A. RN, BSN verfasserin aut A Case Study: Identifying a New Case of Wilson's Disease Oxford, UK; Malden, USA Blackwell Science Inc 2005 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. 2005 Blackwell Publishing Journal Backfiles 1879-2005 |2005|||||||||| Wilson's disease In Journal of the American Academy of Nurse Practitioners Malden, Mass. [u.a.] : Wiley-Blackwell, 1989 17(2005), 12, Seite 0 (DE-627)NLEJ243926340 (DE-600)2116099-5 1745-7599 nnns volume:17 year:2005 number:12 pages:0 http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 17 2005 12 0 |
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10.1111/j.1745-7599.2005.00089.x doi (DE-627)NLEJ243494971 DE-627 ger DE-627 rakwb Noble, Jeannette A. RN, BSN verfasserin aut A Case Study: Identifying a New Case of Wilson's Disease Oxford, UK; Malden, USA Blackwell Science Inc 2005 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. 2005 Blackwell Publishing Journal Backfiles 1879-2005 |2005|||||||||| Wilson's disease In Journal of the American Academy of Nurse Practitioners Malden, Mass. [u.a.] : Wiley-Blackwell, 1989 17(2005), 12, Seite 0 (DE-627)NLEJ243926340 (DE-600)2116099-5 1745-7599 nnns volume:17 year:2005 number:12 pages:0 http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 17 2005 12 0 |
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10.1111/j.1745-7599.2005.00089.x doi (DE-627)NLEJ243494971 DE-627 ger DE-627 rakwb Noble, Jeannette A. RN, BSN verfasserin aut A Case Study: Identifying a New Case of Wilson's Disease Oxford, UK; Malden, USA Blackwell Science Inc 2005 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. 2005 Blackwell Publishing Journal Backfiles 1879-2005 |2005|||||||||| Wilson's disease In Journal of the American Academy of Nurse Practitioners Malden, Mass. [u.a.] : Wiley-Blackwell, 1989 17(2005), 12, Seite 0 (DE-627)NLEJ243926340 (DE-600)2116099-5 1745-7599 nnns volume:17 year:2005 number:12 pages:0 http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 17 2005 12 0 |
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10.1111/j.1745-7599.2005.00089.x doi (DE-627)NLEJ243494971 DE-627 ger DE-627 rakwb Noble, Jeannette A. RN, BSN verfasserin aut A Case Study: Identifying a New Case of Wilson's Disease Oxford, UK; Malden, USA Blackwell Science Inc 2005 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. 2005 Blackwell Publishing Journal Backfiles 1879-2005 |2005|||||||||| Wilson's disease In Journal of the American Academy of Nurse Practitioners Malden, Mass. [u.a.] : Wiley-Blackwell, 1989 17(2005), 12, Seite 0 (DE-627)NLEJ243926340 (DE-600)2116099-5 1745-7599 nnns volume:17 year:2005 number:12 pages:0 http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 17 2005 12 0 |
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10.1111/j.1745-7599.2005.00089.x doi (DE-627)NLEJ243494971 DE-627 ger DE-627 rakwb Noble, Jeannette A. RN, BSN verfasserin aut A Case Study: Identifying a New Case of Wilson's Disease Oxford, UK; Malden, USA Blackwell Science Inc 2005 Online-Ressource nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. 2005 Blackwell Publishing Journal Backfiles 1879-2005 |2005|||||||||| Wilson's disease In Journal of the American Academy of Nurse Practitioners Malden, Mass. [u.a.] : Wiley-Blackwell, 1989 17(2005), 12, Seite 0 (DE-627)NLEJ243926340 (DE-600)2116099-5 1745-7599 nnns volume:17 year:2005 number:12 pages:0 http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x text/html Verlag Deutschlandweit zugänglich Volltext GBV_USEFLAG_U ZDB-1-DJB GBV_NL_ARTICLE AR 17 2005 12 0 |
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To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. |
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To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. |
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To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage. |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">NLEJ243494971</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230505193233.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">120427s2005 xx |||||o 00| ||und c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1111/j.1745-7599.2005.00089.x</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)NLEJ243494971</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Noble, Jeannette A.</subfield><subfield code="c">RN, BSN</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">A Case Study: Identifying a New Case of Wilson's Disease</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="a">Oxford, UK; Malden, USA</subfield><subfield code="b">Blackwell Science Inc</subfield><subfield code="c">2005</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zzz</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">z</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">nicht spezifiziert</subfield><subfield code="b">zu</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">To present a case of Wilson's disease that presented with fatigue, nausea, abdominal pain, and splenomegaly. Patient information, diagnostic tests, etiology, anatomy and physiology, pathophysiology, assessment, signs and symptoms, diagnosis, medical treatment, nursing interventions, patient education, and research findings related to Wilson's disease are discussed.<section xml:id="abs1-2"><title type="main">Data SourcesCase study and scientific literature from Internet, journals, and medical textbooks.<section xml:id="abs1-3"><title type="main">ConclusionsWilson's disease is a hereditary, autosomal-recessive disease affecting copper excretion. As copper accumulates, signs and symptoms appear. Individuals often present with nonspecific findings, making diagnosis difficult.<section xml:id="abs1-4"><title type="main">Implications for PracticeThis article reviews this rare but potentially devastating disease. Early diagnosis is vital to prevent copper accumulation leading to hepatic cirrhosis, basal ganglia degeneration, and irreversible organ damage.</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="d">2005</subfield><subfield code="f">Blackwell Publishing Journal Backfiles 1879-2005</subfield><subfield code="7">|2005||||||||||</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Wilson's disease</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">In</subfield><subfield code="t">Journal of the American Academy of Nurse Practitioners</subfield><subfield code="d">Malden, Mass. [u.a.] : Wiley-Blackwell, 1989</subfield><subfield code="g">17(2005), 12, Seite 0</subfield><subfield code="w">(DE-627)NLEJ243926340</subfield><subfield code="w">(DE-600)2116099-5</subfield><subfield code="x">1745-7599</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:17</subfield><subfield code="g">year:2005</subfield><subfield code="g">number:12</subfield><subfield code="g">pages:0</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">http://dx.doi.org/10.1111/j.1745-7599.2005.00089.x</subfield><subfield code="q">text/html</subfield><subfield code="x">Verlag</subfield><subfield code="z">Deutschlandweit zugänglich</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-DJB</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">17</subfield><subfield code="j">2005</subfield><subfield code="e">12</subfield><subfield code="h">0</subfield></datafield></record></collection>
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