Inter-individual variation of inflammatory markers of cardiovascular risks and diseases
Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological...
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E-Artikel |
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Walter de Gruyter ; 2005 |
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©2005 by Walter de Gruyter Berlin New York |
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14 |
| Reproduktion: |
Walter de Gruyter Online Zeitschriften |
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Enthalten in: Clinical chemistry and laboratory medicine - Berlin [u.a.] : De Gruyter, 1998, 43(2005), 7 vom: 22. Aug., Seite 671-684 |
| Übergeordnetes Werk: |
volume:43 ; year:2005 ; number:7 ; day:22 ; month:08 ; pages:671-684 ; extent:14 |
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| DOI / URN: |
10.1515/CCLM.2005.116 |
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NLEJ246714670 |
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| 520 | |a Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. | ||
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10.1515/CCLM.2005.116 doi artikel_Grundlieferung.pp (DE-627)NLEJ246714670 DE-627 ger DE-627 rakwb Inter-individual variation of inflammatory markers of cardiovascular risks and diseases Walter de Gruyter 2005 14 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier ©2005 by Walter de Gruyter Berlin New York Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. Walter de Gruyter Online Zeitschriften atherosclerosis environmental factors intermediate phenotypes of inflammation polymorphisms Berrahmoune, Hind oth Lamont, John oth Fitzgerald, Peter oth Visvikis-Siest, Sophie oth Enthalten in Clinical chemistry and laboratory medicine Berlin [u.a.] : De Gruyter, 1998 43(2005), 7 vom: 22. Aug., Seite 671-684 (DE-627)NLEJ248235222 (DE-600)1492732-9 1437-4331 nnns volume:43 year:2005 number:7 day:22 month:08 pages:671-684 extent:14 https://doi.org/10.1515/CCLM.2005.116 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 43 2005 7 22 08 671-684 14 |
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10.1515/CCLM.2005.116 doi artikel_Grundlieferung.pp (DE-627)NLEJ246714670 DE-627 ger DE-627 rakwb Inter-individual variation of inflammatory markers of cardiovascular risks and diseases Walter de Gruyter 2005 14 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier ©2005 by Walter de Gruyter Berlin New York Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. Walter de Gruyter Online Zeitschriften atherosclerosis environmental factors intermediate phenotypes of inflammation polymorphisms Berrahmoune, Hind oth Lamont, John oth Fitzgerald, Peter oth Visvikis-Siest, Sophie oth Enthalten in Clinical chemistry and laboratory medicine Berlin [u.a.] : De Gruyter, 1998 43(2005), 7 vom: 22. Aug., Seite 671-684 (DE-627)NLEJ248235222 (DE-600)1492732-9 1437-4331 nnns volume:43 year:2005 number:7 day:22 month:08 pages:671-684 extent:14 https://doi.org/10.1515/CCLM.2005.116 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 43 2005 7 22 08 671-684 14 |
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Inter-individual variation of inflammatory markers of cardiovascular risks and diseases |
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Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. ©2005 by Walter de Gruyter Berlin New York |
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Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. ©2005 by Walter de Gruyter Berlin New York |
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Cardiovascular diseases are a real public health problem and have multifactorial origin. Full comprehension of these diseases is very difficult because of their clinical and biological heterogeneity. The best way to understand the development of these diseases is to first investigate each biological system involved in the diseases and secondly, interactions between them. Studying intermediate phenotype variation for these biological systems is a new and promising approach for their elucidation. Among these metabolic systems, inflammation is known to be implicated in the occurrence and worsening of cardiovascular diseases. For this review, we chose to report the most important results concerning environmental, genetic and epigenetic determinants that influence intermediate phenotypes (protein and RNA levels) of inflammation. Indeed, numerous studies have investigated these determinants in healthy populations, in other words, without the influence of either disease or medication. This new approach will help to better understand the regulation of these intermediate phenotypes and to identify a panel of risk markers, which may be useful in predicting and warning those at risk and in giving adequate treatment to each patient. ©2005 by Walter de Gruyter Berlin New York |
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