Hepatorenal syndrome / Hepatorenales Syndrom
This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting bot...
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Gespeichert in:
| Autor*in: |
Demırtaş, Selda [verfasserIn] Can, Murat [verfasserIn] Yarpuzlu, Ayşegül [verfasserIn] |
|---|
| Format: |
E-Artikel |
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| Erschienen: |
Walter de Gruyter ; 2006 |
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| Schlagwörter: |
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| Anmerkung: |
©2006 by Walter de Gruyter Berlin New York |
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| Umfang: |
8 |
| Reproduktion: |
Walter de Gruyter Online Zeitschriften |
|---|---|
| Übergeordnetes Werk: |
In: 0025-8466 - 30(2006), 5 vom: 15. Okt., Seite 272-279 |
| Übergeordnetes Werk: |
In: 30(2006), 5 vom: 15. Okt., Seite 272-279 volume:30 ; year:2006 ; number:5 ; day:15 ; month:10 ; pages:272-279 ; extent:8 |
| Links: |
|---|
| DOI / URN: |
10.1515/JLM.2006.998 |
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NLEJ247095494 |
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| 520 | |a This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. | ||
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10.1515/JLM.2006.998 doi artikel_Grundlieferung.pp (DE-627)NLEJ247095494 DE-627 ger DE-627 rakwb Demırtaş, Selda verfasserin aut Hepatorenal syndrome / Hepatorenales Syndrom Walter de Gruyter 2006 8 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier ©2006 by Walter de Gruyter Berlin New York This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. Walter de Gruyter Online Zeitschriften cystatin C hepatorenal syndrome laboratory assessment Cystatin C Hepatorenales Syndrom Labortests Can, Murat verfasserin aut Yarpuzlu, Ayşegül verfasserin aut In 30(2006), 5 vom: 15. Okt., Seite 272-279 0025-8466 volume:30 year:2006 number:5 day:15 month:10 pages:272-279 extent:8 https://doi.org/10.1515/JLM.2006.998 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 30 2006 5 15 10 272-279 8 |
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10.1515/JLM.2006.998 doi artikel_Grundlieferung.pp (DE-627)NLEJ247095494 DE-627 ger DE-627 rakwb Demırtaş, Selda verfasserin aut Hepatorenal syndrome / Hepatorenales Syndrom Walter de Gruyter 2006 8 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier ©2006 by Walter de Gruyter Berlin New York This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. Walter de Gruyter Online Zeitschriften cystatin C hepatorenal syndrome laboratory assessment Cystatin C Hepatorenales Syndrom Labortests Can, Murat verfasserin aut Yarpuzlu, Ayşegül verfasserin aut In 30(2006), 5 vom: 15. Okt., Seite 272-279 0025-8466 volume:30 year:2006 number:5 day:15 month:10 pages:272-279 extent:8 https://doi.org/10.1515/JLM.2006.998 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 30 2006 5 15 10 272-279 8 |
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10.1515/JLM.2006.998 doi artikel_Grundlieferung.pp (DE-627)NLEJ247095494 DE-627 ger DE-627 rakwb Demırtaş, Selda verfasserin aut Hepatorenal syndrome / Hepatorenales Syndrom Walter de Gruyter 2006 8 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier ©2006 by Walter de Gruyter Berlin New York This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. Walter de Gruyter Online Zeitschriften cystatin C hepatorenal syndrome laboratory assessment Cystatin C Hepatorenales Syndrom Labortests Can, Murat verfasserin aut Yarpuzlu, Ayşegül verfasserin aut In 30(2006), 5 vom: 15. Okt., Seite 272-279 0025-8466 volume:30 year:2006 number:5 day:15 month:10 pages:272-279 extent:8 https://doi.org/10.1515/JLM.2006.998 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 30 2006 5 15 10 272-279 8 |
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This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. ©2006 by Walter de Gruyter Berlin New York |
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This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. ©2006 by Walter de Gruyter Berlin New York |
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This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches. ©2006 by Walter de Gruyter Berlin New York |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">NLEJ247095494</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230506114650.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">220814s2006 xx |||||o 00| ||und c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1515/JLM.2006.998</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">artikel_Grundlieferung.pp</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)NLEJ247095494</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Demırtaş, Selda</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Hepatorenal syndrome / Hepatorenales Syndrom</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="b">Walter de Gruyter</subfield><subfield code="c">2006</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">8</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="500" ind1=" " ind2=" "><subfield code="a">©2006 by Walter de Gruyter Berlin New York</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">This article summarizes the literature on current definition, suggested pathogenetic mechanisms and the role of laboratory assessment in the differential diagnosis of hepatorenal syndrome (HRS) from other causes of renal disease that may arise during hepatic cirrhosis and some diseases affecting both liver and kidney. It should be remembered that the main theory suggested for the pathogenesis of HRS is the arterial vasodilation hypothesis of portal hypertension, ending in type 1 and type 2 HRS, but there is no consensus supporting either mechanism as a solid theory for initiation of HRS pathogenesis to date. No laboratory test can firmly establish a diagnosis of HRS, which is mainly based on the absence of any specific cause of renal failure. Laboratory and ultrasonographic tests based on non-invasive techniques are being investigated as possible diagnostic approaches.</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="f">Walter de Gruyter Online Zeitschriften</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">cystatin C</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">hepatorenal syndrome</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">laboratory assessment</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Cystatin C</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Hepatorenales Syndrom</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Labortests</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Can, Murat</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Yarpuzlu, Ayşegül</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">In</subfield><subfield code="g">30(2006), 5 vom: 15. Okt., Seite 272-279</subfield><subfield code="x">0025-8466</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:30</subfield><subfield code="g">year:2006</subfield><subfield code="g">number:5</subfield><subfield code="g">day:15</subfield><subfield code="g">month:10</subfield><subfield code="g">pages:272-279</subfield><subfield code="g">extent:8</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1515/JLM.2006.998</subfield><subfield code="z">Deutschlandweit zugänglich</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-DGR</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">30</subfield><subfield code="j">2006</subfield><subfield code="e">5</subfield><subfield code="b">15</subfield><subfield code="c">10</subfield><subfield code="h">272-279</subfield><subfield code="g">8</subfield></datafield></record></collection>
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