Environmental exposures: an underrecognized contribution to noncommunicable diseases
Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports hav...
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De Gruyter ; 2013 |
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7 |
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Walter de Gruyter Online Zeitschriften |
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Enthalten in: Reviews on environmental health - Berlin : de Gruyter, 1986, 28(2013), 1 vom: 04. Apr., Seite 59-65 |
| Übergeordnetes Werk: |
volume:28 ; year:2013 ; number:1 ; day:04 ; month:04 ; pages:59-65 ; extent:7 |
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10.1515/reveh-2012-0033 |
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| 520 | |a Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. | ||
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10.1515/reveh-2012-0033 doi artikel_Grundlieferung.pp (DE-627)NLEJ247534617 DE-627 ger DE-627 rakwb Environmental exposures: an underrecognized contribution to noncommunicable diseases De Gruyter 2013 7 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. Walter de Gruyter Online Zeitschriften burden of disease children environment exposure noncommunicable diseases Norman, Rosana E. oth Carpenter, David O. oth Scott, James oth Brune, Marie Noel oth Sly, Peter D. oth Enthalten in Reviews on environmental health Berlin : de Gruyter, 1986 28(2013), 1 vom: 04. Apr., Seite 59-65 (DE-627)NLEJ248236687 (DE-600)2609347-9 2191-0308 nnns volume:28 year:2013 number:1 day:04 month:04 pages:59-65 extent:7 https://doi.org/10.1515/reveh-2012-0033 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 28 2013 1 04 04 59-65 7 |
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10.1515/reveh-2012-0033 doi artikel_Grundlieferung.pp (DE-627)NLEJ247534617 DE-627 ger DE-627 rakwb Environmental exposures: an underrecognized contribution to noncommunicable diseases De Gruyter 2013 7 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. Walter de Gruyter Online Zeitschriften burden of disease children environment exposure noncommunicable diseases Norman, Rosana E. oth Carpenter, David O. oth Scott, James oth Brune, Marie Noel oth Sly, Peter D. oth Enthalten in Reviews on environmental health Berlin : de Gruyter, 1986 28(2013), 1 vom: 04. Apr., Seite 59-65 (DE-627)NLEJ248236687 (DE-600)2609347-9 2191-0308 nnns volume:28 year:2013 number:1 day:04 month:04 pages:59-65 extent:7 https://doi.org/10.1515/reveh-2012-0033 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 28 2013 1 04 04 59-65 7 |
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10.1515/reveh-2012-0033 doi artikel_Grundlieferung.pp (DE-627)NLEJ247534617 DE-627 ger DE-627 rakwb Environmental exposures: an underrecognized contribution to noncommunicable diseases De Gruyter 2013 7 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. Walter de Gruyter Online Zeitschriften burden of disease children environment exposure noncommunicable diseases Norman, Rosana E. oth Carpenter, David O. oth Scott, James oth Brune, Marie Noel oth Sly, Peter D. oth Enthalten in Reviews on environmental health Berlin : de Gruyter, 1986 28(2013), 1 vom: 04. Apr., Seite 59-65 (DE-627)NLEJ248236687 (DE-600)2609347-9 2191-0308 nnns volume:28 year:2013 number:1 day:04 month:04 pages:59-65 extent:7 https://doi.org/10.1515/reveh-2012-0033 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 28 2013 1 04 04 59-65 7 |
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10.1515/reveh-2012-0033 doi artikel_Grundlieferung.pp (DE-627)NLEJ247534617 DE-627 ger DE-627 rakwb Environmental exposures: an underrecognized contribution to noncommunicable diseases De Gruyter 2013 7 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. Walter de Gruyter Online Zeitschriften burden of disease children environment exposure noncommunicable diseases Norman, Rosana E. oth Carpenter, David O. oth Scott, James oth Brune, Marie Noel oth Sly, Peter D. oth Enthalten in Reviews on environmental health Berlin : de Gruyter, 1986 28(2013), 1 vom: 04. Apr., Seite 59-65 (DE-627)NLEJ248236687 (DE-600)2609347-9 2191-0308 nnns volume:28 year:2013 number:1 day:04 month:04 pages:59-65 extent:7 https://doi.org/10.1515/reveh-2012-0033 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 28 2013 1 04 04 59-65 7 |
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10.1515/reveh-2012-0033 doi artikel_Grundlieferung.pp (DE-627)NLEJ247534617 DE-627 ger DE-627 rakwb Environmental exposures: an underrecognized contribution to noncommunicable diseases De Gruyter 2013 7 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. Walter de Gruyter Online Zeitschriften burden of disease children environment exposure noncommunicable diseases Norman, Rosana E. oth Carpenter, David O. oth Scott, James oth Brune, Marie Noel oth Sly, Peter D. oth Enthalten in Reviews on environmental health Berlin : de Gruyter, 1986 28(2013), 1 vom: 04. Apr., Seite 59-65 (DE-627)NLEJ248236687 (DE-600)2609347-9 2191-0308 nnns volume:28 year:2013 number:1 day:04 month:04 pages:59-65 extent:7 https://doi.org/10.1515/reveh-2012-0033 Deutschlandweit zugänglich GBV_USEFLAG_U ZDB-1-DGR GBV_NL_ARTICLE AR 28 2013 1 04 04 59-65 7 |
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Environmental exposures: an underrecognized contribution to noncommunicable diseases |
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Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. |
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Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. |
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Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks. |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">NLEJ247534617</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230506005048.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">220814s2013 xx |||||o 00| ||und c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1515/reveh-2012-0033</subfield><subfield code="2">doi</subfield></datafield><datafield tag="028" ind1="5" ind2="2"><subfield code="a">artikel_Grundlieferung.pp</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)NLEJ247534617</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Environmental exposures: an underrecognized contribution to noncommunicable diseases</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="b">De Gruyter</subfield><subfield code="c">2013</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">7</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Previous attempts to determine the degree to which exposure to environmental factors contribute to noncommunicable diseases (NCDs) have been very conservative and have significantly underestimated the actual contribution of the environment for at least two reasons. Firstly, most previous reports have excluded the contribution of lifestyle behavioral risk factors, but these usually involve significant exposure to environmental chemicals that increase risk of disease. Secondly, early life exposure to chemical contaminants is now clearly associated with an elevated risk of several diseases later in life, but these connections are often difficult to discern. This is especially true for asthma and neurodevelopmental conditions, but there is also a major contribution to the development of obesity and chronic diseases. Most cancers are caused by environmental exposures in genetically susceptible individuals. In addition, new information shows significant associations between cardiovascular diseases and diabetes and exposure to environmental chemicals present in air, food, and water. These relationships likely reflect the combination of epigenetic effects and gene induction. Environmental factors contribute significantly more to NCDs than previous reports have suggested. Prevention needs to shift focus from individual responsibility to societal responsibility and an understanding that effective prevention of NCDs ultimately relies on improved environmental management to reduce exposure to modifiable risks.</subfield></datafield><datafield tag="533" ind1=" " ind2=" "><subfield code="f">Walter de Gruyter Online Zeitschriften</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">burden of disease</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">children</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">environment</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">exposure</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">noncommunicable diseases</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Norman, Rosana E.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Carpenter, David O.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Scott, James</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Brune, Marie Noel</subfield><subfield code="4">oth</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Sly, Peter D.</subfield><subfield code="4">oth</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Reviews on environmental health</subfield><subfield code="d">Berlin : de Gruyter, 1986</subfield><subfield code="g">28(2013), 1 vom: 04. Apr., Seite 59-65</subfield><subfield code="w">(DE-627)NLEJ248236687</subfield><subfield code="w">(DE-600)2609347-9</subfield><subfield code="x">2191-0308</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:28</subfield><subfield code="g">year:2013</subfield><subfield code="g">number:1</subfield><subfield code="g">day:04</subfield><subfield code="g">month:04</subfield><subfield code="g">pages:59-65</subfield><subfield code="g">extent:7</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1515/reveh-2012-0033</subfield><subfield code="z">Deutschlandweit zugänglich</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_U</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-1-DGR</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_NL_ARTICLE</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">28</subfield><subfield code="j">2013</subfield><subfield code="e">1</subfield><subfield code="b">04</subfield><subfield code="c">04</subfield><subfield code="h">59-65</subfield><subfield code="g">7</subfield></datafield></record></collection>
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