Signals straightened out

The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The s...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Peter K Jackson [verfasserIn]

Format:	Artikel
Sprache:	Englisch

Erschienen:	2016

Schlagwörter:	Kidney diseases Proteins Metabolites Calcium Mutation Signal transduction

Übergeordnetes Werk:	Enthalten in: Nature - London : Macmillan Publishers Limited, part of Springer Nature, 1869, 531(2016), 7596, Seite 582
Übergeordnetes Werk:	volume:531 ; year:2016 ; number:7596 ; pages:582

Links:	Link aufrufen

Katalog-ID:	OLC1972878166

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650		4	\|a Kidney diseases
650		4	\|a Proteins
650		4	\|a Metabolites
650		4	\|a Calcium
650		4	\|a Mutation
650		4	\|a Signal transduction
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spelling	PQ20160430 (DE-627)OLC1972878166 (DE-599)GBVOLC1972878166 (PRQ)p572-5f049ffe80ef3d81df02641bdbfdf6f4a9bf911929504f756bcf4ac5e23edf70 (KEY)0072945020160000531759600582signalsstraightenedout DE-627 ger DE-627 rakwb eng 070 500 DNB 500 AVZ BIODIV fid Peter K Jackson verfasserin aut Signals straightened out 2016 Text txt rdacontent ohne Hilfsmittel zu benutzen n rdamedia Band nc rdacarrier The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement. Kidney diseases Proteins Metabolites Calcium Mutation Signal transduction Enthalten in Nature London : Macmillan Publishers Limited, part of Springer Nature, 1869 531(2016), 7596, Seite 582 (DE-627)129292834 (DE-600)120714-3 (DE-576)014473941 0028-0836 nnns volume:531 year:2016 number:7596 pages:582 http://search.proquest.com/docview/1778751132 GBV_USEFLAG_A SYSFLAG_A GBV_OLC FID-BIODIV SSG-OLC-PHY SSG-OLC-CHE SSG-OLC-MAT SSG-OLC-FOR SSG-OLC-SPO SSG-OLC-PHA SSG-OLC-DE-84 SSG-OPC-FOR GBV_ILN_11 GBV_ILN_22 GBV_ILN_30 GBV_ILN_40 GBV_ILN_47 GBV_ILN_55 GBV_ILN_59 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_101 GBV_ILN_110 GBV_ILN_120 GBV_ILN_154 GBV_ILN_168 GBV_ILN_170 GBV_ILN_171 GBV_ILN_211 GBV_ILN_290 GBV_ILN_294 GBV_ILN_601 GBV_ILN_647 GBV_ILN_754 GBV_ILN_2001 GBV_ILN_2002 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2015 GBV_ILN_2016 GBV_ILN_2018 GBV_ILN_2020 GBV_ILN_2026 GBV_ILN_2095 GBV_ILN_2116 GBV_ILN_2120 GBV_ILN_2121 GBV_ILN_2219 GBV_ILN_2221 GBV_ILN_2279 GBV_ILN_2286 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4125 GBV_ILN_4219 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4302 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4314 GBV_ILN_4317 GBV_ILN_4320 GBV_ILN_4324 GBV_ILN_4700 AR 531 2016 7596 582
allfields_unstemmed	PQ20160430 (DE-627)OLC1972878166 (DE-599)GBVOLC1972878166 (PRQ)p572-5f049ffe80ef3d81df02641bdbfdf6f4a9bf911929504f756bcf4ac5e23edf70 (KEY)0072945020160000531759600582signalsstraightenedout DE-627 ger DE-627 rakwb eng 070 500 DNB 500 AVZ BIODIV fid Peter K Jackson verfasserin aut Signals straightened out 2016 Text txt rdacontent ohne Hilfsmittel zu benutzen n rdamedia Band nc rdacarrier The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement. Kidney diseases Proteins Metabolites Calcium Mutation Signal transduction Enthalten in Nature London : Macmillan Publishers Limited, part of Springer Nature, 1869 531(2016), 7596, Seite 582 (DE-627)129292834 (DE-600)120714-3 (DE-576)014473941 0028-0836 nnns volume:531 year:2016 number:7596 pages:582 http://search.proquest.com/docview/1778751132 GBV_USEFLAG_A SYSFLAG_A GBV_OLC FID-BIODIV SSG-OLC-PHY SSG-OLC-CHE SSG-OLC-MAT SSG-OLC-FOR SSG-OLC-SPO SSG-OLC-PHA SSG-OLC-DE-84 SSG-OPC-FOR GBV_ILN_11 GBV_ILN_22 GBV_ILN_30 GBV_ILN_40 GBV_ILN_47 GBV_ILN_55 GBV_ILN_59 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_101 GBV_ILN_110 GBV_ILN_120 GBV_ILN_154 GBV_ILN_168 GBV_ILN_170 GBV_ILN_171 GBV_ILN_211 GBV_ILN_290 GBV_ILN_294 GBV_ILN_601 GBV_ILN_647 GBV_ILN_754 GBV_ILN_2001 GBV_ILN_2002 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2015 GBV_ILN_2016 GBV_ILN_2018 GBV_ILN_2020 GBV_ILN_2026 GBV_ILN_2095 GBV_ILN_2116 GBV_ILN_2120 GBV_ILN_2121 GBV_ILN_2219 GBV_ILN_2221 GBV_ILN_2279 GBV_ILN_2286 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4125 GBV_ILN_4219 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4302 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4314 GBV_ILN_4317 GBV_ILN_4320 GBV_ILN_4324 GBV_ILN_4700 AR 531 2016 7596 582
allfieldsGer	PQ20160430 (DE-627)OLC1972878166 (DE-599)GBVOLC1972878166 (PRQ)p572-5f049ffe80ef3d81df02641bdbfdf6f4a9bf911929504f756bcf4ac5e23edf70 (KEY)0072945020160000531759600582signalsstraightenedout DE-627 ger DE-627 rakwb eng 070 500 DNB 500 AVZ BIODIV fid Peter K Jackson verfasserin aut Signals straightened out 2016 Text txt rdacontent ohne Hilfsmittel zu benutzen n rdamedia Band nc rdacarrier The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement. Kidney diseases Proteins Metabolites Calcium Mutation Signal transduction Enthalten in Nature London : Macmillan Publishers Limited, part of Springer Nature, 1869 531(2016), 7596, Seite 582 (DE-627)129292834 (DE-600)120714-3 (DE-576)014473941 0028-0836 nnns volume:531 year:2016 number:7596 pages:582 http://search.proquest.com/docview/1778751132 GBV_USEFLAG_A SYSFLAG_A GBV_OLC FID-BIODIV SSG-OLC-PHY SSG-OLC-CHE SSG-OLC-MAT SSG-OLC-FOR SSG-OLC-SPO SSG-OLC-PHA SSG-OLC-DE-84 SSG-OPC-FOR GBV_ILN_11 GBV_ILN_22 GBV_ILN_30 GBV_ILN_40 GBV_ILN_47 GBV_ILN_55 GBV_ILN_59 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_101 GBV_ILN_110 GBV_ILN_120 GBV_ILN_154 GBV_ILN_168 GBV_ILN_170 GBV_ILN_171 GBV_ILN_211 GBV_ILN_290 GBV_ILN_294 GBV_ILN_601 GBV_ILN_647 GBV_ILN_754 GBV_ILN_2001 GBV_ILN_2002 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2015 GBV_ILN_2016 GBV_ILN_2018 GBV_ILN_2020 GBV_ILN_2026 GBV_ILN_2095 GBV_ILN_2116 GBV_ILN_2120 GBV_ILN_2121 GBV_ILN_2219 GBV_ILN_2221 GBV_ILN_2279 GBV_ILN_2286 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4125 GBV_ILN_4219 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4302 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4314 GBV_ILN_4317 GBV_ILN_4320 GBV_ILN_4324 GBV_ILN_4700 AR 531 2016 7596 582
allfieldsSound	PQ20160430 (DE-627)OLC1972878166 (DE-599)GBVOLC1972878166 (PRQ)p572-5f049ffe80ef3d81df02641bdbfdf6f4a9bf911929504f756bcf4ac5e23edf70 (KEY)0072945020160000531759600582signalsstraightenedout DE-627 ger DE-627 rakwb eng 070 500 DNB 500 AVZ BIODIV fid Peter K Jackson verfasserin aut Signals straightened out 2016 Text txt rdacontent ohne Hilfsmittel zu benutzen n rdamedia Band nc rdacarrier The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement. Kidney diseases Proteins Metabolites Calcium Mutation Signal transduction Enthalten in Nature London : Macmillan Publishers Limited, part of Springer Nature, 1869 531(2016), 7596, Seite 582 (DE-627)129292834 (DE-600)120714-3 (DE-576)014473941 0028-0836 nnns volume:531 year:2016 number:7596 pages:582 http://search.proquest.com/docview/1778751132 GBV_USEFLAG_A SYSFLAG_A GBV_OLC FID-BIODIV SSG-OLC-PHY SSG-OLC-CHE SSG-OLC-MAT SSG-OLC-FOR SSG-OLC-SPO SSG-OLC-PHA SSG-OLC-DE-84 SSG-OPC-FOR GBV_ILN_11 GBV_ILN_22 GBV_ILN_30 GBV_ILN_40 GBV_ILN_47 GBV_ILN_55 GBV_ILN_59 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_101 GBV_ILN_110 GBV_ILN_120 GBV_ILN_154 GBV_ILN_168 GBV_ILN_170 GBV_ILN_171 GBV_ILN_211 GBV_ILN_290 GBV_ILN_294 GBV_ILN_601 GBV_ILN_647 GBV_ILN_754 GBV_ILN_2001 GBV_ILN_2002 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2015 GBV_ILN_2016 GBV_ILN_2018 GBV_ILN_2020 GBV_ILN_2026 GBV_ILN_2095 GBV_ILN_2116 GBV_ILN_2120 GBV_ILN_2121 GBV_ILN_2219 GBV_ILN_2221 GBV_ILN_2279 GBV_ILN_2286 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4125 GBV_ILN_4219 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4302 GBV_ILN_4305 GBV_ILN_4313 GBV_ILN_4314 GBV_ILN_4317 GBV_ILN_4320 GBV_ILN_4324 GBV_ILN_4700 AR 531 2016 7596 582
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source	Enthalten in Nature 531(2016), 7596, Seite 582 volume:531 year:2016 number:7596 pages:582
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author	Peter K Jackson
spellingShingle	Peter K Jackson ddc 070 ddc 500 fid BIODIV misc Kidney diseases misc Proteins misc Metabolites misc Calcium misc Mutation misc Signal transduction Signals straightened out
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abstract	The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement.
abstractGer	The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement.
abstract_unstemmed	The hypothesis8 that forces generated by urinary flow elicit ciliary mechanosensation in the kidney dates to 2001, and defects in this process have been proposed to cause adult polycystic kidney disease. A technical tour de force has allowed Delling et al. to question this long-standing model. The strength of their argument lies in their use of recently developed Ca2+-signalling reporters (described above) and their ability to rapidly visualize Ca2+ signalling while controlling for ciliary movement.
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score	7.3994074

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