The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response
Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,7...
Ausführliche Beschreibung
Autor*in: |
Kacin, Alan [verfasserIn] Golja, Petra [verfasserIn] Eiken, Ola [verfasserIn] Tipton, Michael J. [verfasserIn] Mekjavic, Igor B. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2007 |
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Schlagwörter: |
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Übergeordnetes Werk: |
Enthalten in: European journal of applied physiology - Berlin : Springer, 1928, 99(2007), 5 vom: 23. Jan., Seite 557-566 |
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Übergeordnetes Werk: |
volume:99 ; year:2007 ; number:5 ; day:23 ; month:01 ; pages:557-566 |
Links: |
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DOI / URN: |
10.1007/s00421-006-0364-9 |
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Katalog-ID: |
SPR005514797 |
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245 | 1 | 4 | |a The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
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520 | |a Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. | ||
650 | 4 | |a Exercise thermoregulation |7 (dpeaa)DE-He213 | |
650 | 4 | |a Non-thermal sweating |7 (dpeaa)DE-He213 | |
650 | 4 | |a Intermittent normobaric hypoxia |7 (dpeaa)DE-He213 | |
650 | 4 | |a Perceived exertion |7 (dpeaa)DE-He213 | |
650 | 4 | |a Relative work rate |7 (dpeaa)DE-He213 | |
700 | 1 | |a Golja, Petra |e verfasserin |4 aut | |
700 | 1 | |a Eiken, Ola |e verfasserin |4 aut | |
700 | 1 | |a Tipton, Michael J. |e verfasserin |4 aut | |
700 | 1 | |a Mekjavic, Igor B. |e verfasserin |4 aut | |
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10.1007/s00421-006-0364-9 doi (DE-627)SPR005514797 (SPR)s00421-006-0364-9-e DE-627 ger DE-627 rakwb eng 610 ASE 44.37 bkl Kacin, Alan verfasserin aut The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 Golja, Petra verfasserin aut Eiken, Ola verfasserin aut Tipton, Michael J. verfasserin aut Mekjavic, Igor B. verfasserin aut Enthalten in European journal of applied physiology Berlin : Springer, 1928 99(2007), 5 vom: 23. Jan., Seite 557-566 (DE-627)253722780 (DE-600)1459054-2 1439-6327 nnns volume:99 year:2007 number:5 day:23 month:01 pages:557-566 https://dx.doi.org/10.1007/s00421-006-0364-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_647 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.37 ASE AR 99 2007 5 23 01 557-566 |
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10.1007/s00421-006-0364-9 doi (DE-627)SPR005514797 (SPR)s00421-006-0364-9-e DE-627 ger DE-627 rakwb eng 610 ASE 44.37 bkl Kacin, Alan verfasserin aut The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 Golja, Petra verfasserin aut Eiken, Ola verfasserin aut Tipton, Michael J. verfasserin aut Mekjavic, Igor B. verfasserin aut Enthalten in European journal of applied physiology Berlin : Springer, 1928 99(2007), 5 vom: 23. Jan., Seite 557-566 (DE-627)253722780 (DE-600)1459054-2 1439-6327 nnns volume:99 year:2007 number:5 day:23 month:01 pages:557-566 https://dx.doi.org/10.1007/s00421-006-0364-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_647 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.37 ASE AR 99 2007 5 23 01 557-566 |
allfields_unstemmed |
10.1007/s00421-006-0364-9 doi (DE-627)SPR005514797 (SPR)s00421-006-0364-9-e DE-627 ger DE-627 rakwb eng 610 ASE 44.37 bkl Kacin, Alan verfasserin aut The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 Golja, Petra verfasserin aut Eiken, Ola verfasserin aut Tipton, Michael J. verfasserin aut Mekjavic, Igor B. verfasserin aut Enthalten in European journal of applied physiology Berlin : Springer, 1928 99(2007), 5 vom: 23. Jan., Seite 557-566 (DE-627)253722780 (DE-600)1459054-2 1439-6327 nnns volume:99 year:2007 number:5 day:23 month:01 pages:557-566 https://dx.doi.org/10.1007/s00421-006-0364-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_647 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.37 ASE AR 99 2007 5 23 01 557-566 |
allfieldsGer |
10.1007/s00421-006-0364-9 doi (DE-627)SPR005514797 (SPR)s00421-006-0364-9-e DE-627 ger DE-627 rakwb eng 610 ASE 44.37 bkl Kacin, Alan verfasserin aut The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 Golja, Petra verfasserin aut Eiken, Ola verfasserin aut Tipton, Michael J. verfasserin aut Mekjavic, Igor B. verfasserin aut Enthalten in European journal of applied physiology Berlin : Springer, 1928 99(2007), 5 vom: 23. Jan., Seite 557-566 (DE-627)253722780 (DE-600)1459054-2 1439-6327 nnns volume:99 year:2007 number:5 day:23 month:01 pages:557-566 https://dx.doi.org/10.1007/s00421-006-0364-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_647 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.37 ASE AR 99 2007 5 23 01 557-566 |
allfieldsSound |
10.1007/s00421-006-0364-9 doi (DE-627)SPR005514797 (SPR)s00421-006-0364-9-e DE-627 ger DE-627 rakwb eng 610 ASE 44.37 bkl Kacin, Alan verfasserin aut The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 Golja, Petra verfasserin aut Eiken, Ola verfasserin aut Tipton, Michael J. verfasserin aut Mekjavic, Igor B. verfasserin aut Enthalten in European journal of applied physiology Berlin : Springer, 1928 99(2007), 5 vom: 23. Jan., Seite 557-566 (DE-627)253722780 (DE-600)1459054-2 1439-6327 nnns volume:99 year:2007 number:5 day:23 month:01 pages:557-566 https://dx.doi.org/10.1007/s00421-006-0364-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_647 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4277 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.37 ASE AR 99 2007 5 23 01 557-566 |
language |
English |
source |
Enthalten in European journal of applied physiology 99(2007), 5 vom: 23. Jan., Seite 557-566 volume:99 year:2007 number:5 day:23 month:01 pages:557-566 |
sourceStr |
Enthalten in European journal of applied physiology 99(2007), 5 vom: 23. Jan., Seite 557-566 volume:99 year:2007 number:5 day:23 month:01 pages:557-566 |
format_phy_str_mv |
Article |
institution |
findex.gbv.de |
topic_facet |
Exercise thermoregulation Non-thermal sweating Intermittent normobaric hypoxia Perceived exertion Relative work rate |
dewey-raw |
610 |
isfreeaccess_bool |
false |
container_title |
European journal of applied physiology |
authorswithroles_txt_mv |
Kacin, Alan @@aut@@ Golja, Petra @@aut@@ Eiken, Ola @@aut@@ Tipton, Michael J. @@aut@@ Mekjavic, Igor B. @@aut@@ |
publishDateDaySort_date |
2007-01-23T00:00:00Z |
hierarchy_top_id |
253722780 |
dewey-sort |
3610 |
id |
SPR005514797 |
language_de |
englisch |
fullrecord |
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Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. 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|
author |
Kacin, Alan |
spellingShingle |
Kacin, Alan ddc 610 bkl 44.37 misc Exercise thermoregulation misc Non-thermal sweating misc Intermittent normobaric hypoxia misc Perceived exertion misc Relative work rate The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
authorStr |
Kacin, Alan |
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610 - Medicine & health |
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illustrated |
Not Illustrated |
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610 ASE 44.37 bkl The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response Exercise thermoregulation (dpeaa)DE-He213 Non-thermal sweating (dpeaa)DE-He213 Intermittent normobaric hypoxia (dpeaa)DE-He213 Perceived exertion (dpeaa)DE-He213 Relative work rate (dpeaa)DE-He213 |
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ddc 610 bkl 44.37 misc Exercise thermoregulation misc Non-thermal sweating misc Intermittent normobaric hypoxia misc Perceived exertion misc Relative work rate |
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ddc 610 bkl 44.37 misc Exercise thermoregulation misc Non-thermal sweating misc Intermittent normobaric hypoxia misc Perceived exertion misc Relative work rate |
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ddc 610 bkl 44.37 misc Exercise thermoregulation misc Non-thermal sweating misc Intermittent normobaric hypoxia misc Perceived exertion misc Relative work rate |
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European journal of applied physiology |
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The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
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The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
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Kacin, Alan |
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European journal of applied physiology |
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European journal of applied physiology |
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Kacin, Alan Golja, Petra Eiken, Ola Tipton, Michael J. Mekjavic, Igor B. |
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610 ASE 44.37 bkl |
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610 |
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influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
title_auth |
The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
abstract |
Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. |
abstractGer |
Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. |
abstract_unstemmed |
Abstract The effect of acute and 23 days of intermittent exposures to normobaric hypoxia on the forehead sweating response during steady-state exercise was investigated. Eight endurance athletes slept in a normobaric hypoxic room for a minimum of 8 h per day at a simulated altitude equivalent to 2,700 m for 23 days (sleep high–train low regimen). Peak oxygen uptake %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} } \right)} %$ and peak work rate ($ WR_{peak} $) were determined under normoxic (20.9%$ O_{2} $) and hypoxic (13.5%$ O_{2} $) conditions prior to (pre-IHE), and immediately after (post-IHE) the intermittent hypoxic exposures (IHE). Also, each subject performed three 30-min cycle-ergometry bouts: (1) normoxic exercise at 50% $ WR_{peak} $ attained in normoxia (control trial; CT); (2) hypoxic exercise at 50% $ WR_{peak} $ attained in hypoxia (hypoxic relative trial; HRT) and (3) hypoxic exercise at the same absolute work rate as in CT (hypoxic absolute trial; HAT). Exposure to hypoxia induced a 33 and 37% decrease (P < 0.001) in %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{V}{\text{O}}_{{2{\text{peak}}}} %$ pre-IHE and post-IHE, respectively. Despite similar relative oxygen uptake during HAT pre-IHE and post-IHE, the ratings of perceived whole-body exertion decreased substantially (P < 0.05) post-IHE. Pre-IHE the sweat secretion on the forehead %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f} \right)} %$ was greater (P < 0.01) in the HAT (2.60 (0.80) mg $ cm^{−2} $ $ min^{−1} $) compared to the other two trials (CT = 1.87 (1.09) mg $ cm^{−2} $ $ min^{−1} $; HRT = 1.57 (0.82) mg $ cm^{−2} $ $ min^{−1} $) despite a similar exercise-induced elevation in body temperatures, resulting in an augmented (P < 0.01) gain of the sweating response %$ {\left( {\ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} } \right)}. %$ The augmented %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f %$ and %$ \ifmmode\expandafter\dot\else\expandafter\.\fi{m}_{{{\text{sw}}}} f/\Delta T_{{{\text{re}}}} %$ during the HAT were no longer evident post-IHE. Thus, it appears that exercise sweating on the forehead is potentiated by acute exposure to hypoxia, an effect which can be abolished by 23 days of intermittent hypoxic exposures. |
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The influence of acute and 23 days of intermittent hypoxic exposures on the exercise-induced forehead sweating response |
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|
score |
7.398575 |