Experimentelle Therapie des Hodgkin-Lymphoms

Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wi...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Böll, B. [verfasserIn] Borchmann, P. [verfasserIn] Engert, A. [verfasserIn]

Format:	E-Artikel
Sprache:	Deutsch

Erschienen:	2009

Schlagwörter:	Immunotherapy Small molecular substances Targeted therapy H-RS cell Relapsed Hodgkin‘s lymphoma

Übergeordnetes Werk:	Enthalten in: Der Onkologe - Berlin : Springer, 1996, 16(2009), 1 vom: 23. Dez., Seite 48-54
Übergeordnetes Werk:	volume:16 ; year:2009 ; number:1 ; day:23 ; month:12 ; pages:48-54

Links:	Volltext

DOI / URN:	10.1007/s00761-009-1765-2

Katalog-ID:	SPR007780826

Internformat


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520			\|a Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen.
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912			\|a GBV_ILN_32
912			\|a GBV_ILN_39
912			\|a GBV_ILN_40
912			\|a GBV_ILN_60
912			\|a GBV_ILN_62
912			\|a GBV_ILN_63
912			\|a GBV_ILN_65
912			\|a GBV_ILN_69
912			\|a GBV_ILN_70
912			\|a GBV_ILN_73
912			\|a GBV_ILN_74
912			\|a GBV_ILN_90
912			\|a GBV_ILN_95
912			\|a GBV_ILN_100
912			\|a GBV_ILN_101
912			\|a GBV_ILN_105
912			\|a GBV_ILN_110
912			\|a GBV_ILN_120
912			\|a GBV_ILN_138
912			\|a GBV_ILN_151
912			\|a GBV_ILN_152
912			\|a GBV_ILN_161
912			\|a GBV_ILN_170
912			\|a GBV_ILN_171
912			\|a GBV_ILN_187
912			\|a GBV_ILN_213
912			\|a GBV_ILN_224
912			\|a GBV_ILN_230
912			\|a GBV_ILN_250
912			\|a GBV_ILN_267
912			\|a GBV_ILN_281
912			\|a GBV_ILN_285
912			\|a GBV_ILN_293
912			\|a GBV_ILN_370
912			\|a GBV_ILN_602
912			\|a GBV_ILN_636
912			\|a GBV_ILN_702
912			\|a GBV_ILN_711
912			\|a GBV_ILN_2001
912			\|a GBV_ILN_2003
912			\|a GBV_ILN_2004
912			\|a GBV_ILN_2005
912			\|a GBV_ILN_2006
912			\|a GBV_ILN_2007
912			\|a GBV_ILN_2008
912			\|a GBV_ILN_2009
912			\|a GBV_ILN_2010
912			\|a GBV_ILN_2011
912			\|a GBV_ILN_2014
912			\|a GBV_ILN_2015
912			\|a GBV_ILN_2020
912			\|a GBV_ILN_2021
912			\|a GBV_ILN_2025
912			\|a GBV_ILN_2026
912			\|a GBV_ILN_2027
912			\|a GBV_ILN_2031
912			\|a GBV_ILN_2034
912			\|a GBV_ILN_2037
912			\|a GBV_ILN_2038
912			\|a GBV_ILN_2039
912			\|a GBV_ILN_2044
912			\|a GBV_ILN_2048
912			\|a GBV_ILN_2049
912			\|a GBV_ILN_2050
912			\|a GBV_ILN_2055
912			\|a GBV_ILN_2057
912			\|a GBV_ILN_2059
912			\|a GBV_ILN_2061
912			\|a GBV_ILN_2064
912			\|a GBV_ILN_2065
912			\|a GBV_ILN_2068
912			\|a GBV_ILN_2086
912			\|a GBV_ILN_2088
912			\|a GBV_ILN_2093
912			\|a GBV_ILN_2106
912			\|a GBV_ILN_2107
912			\|a GBV_ILN_2108
912			\|a GBV_ILN_2110
912			\|a GBV_ILN_2111
912			\|a GBV_ILN_2112
912			\|a GBV_ILN_2113
912			\|a GBV_ILN_2116
912			\|a GBV_ILN_2118
912			\|a GBV_ILN_2119
912			\|a GBV_ILN_2122
912			\|a GBV_ILN_2129
912			\|a GBV_ILN_2143
912			\|a GBV_ILN_2144
912			\|a GBV_ILN_2147
912			\|a GBV_ILN_2148
912			\|a GBV_ILN_2152
912			\|a GBV_ILN_2153
912			\|a GBV_ILN_2188
912			\|a GBV_ILN_2190
912			\|a GBV_ILN_2232
912			\|a GBV_ILN_2336
912			\|a GBV_ILN_2339
912			\|a GBV_ILN_2414
912			\|a GBV_ILN_2424
912			\|a GBV_ILN_2446
912			\|a GBV_ILN_2470
912			\|a GBV_ILN_2472
912			\|a GBV_ILN_2507
912			\|a GBV_ILN_2522
912			\|a GBV_ILN_2548
912			\|a GBV_ILN_4012
912			\|a GBV_ILN_4035
912			\|a GBV_ILN_4037
912			\|a GBV_ILN_4046
912			\|a GBV_ILN_4112
912			\|a GBV_ILN_4125
912			\|a GBV_ILN_4126
912			\|a GBV_ILN_4242
912			\|a GBV_ILN_4246
912			\|a GBV_ILN_4249
912			\|a GBV_ILN_4251
912			\|a GBV_ILN_4305
912			\|a GBV_ILN_4306
912			\|a GBV_ILN_4307
912			\|a GBV_ILN_4313
912			\|a GBV_ILN_4322
912			\|a GBV_ILN_4323
912			\|a GBV_ILN_4324
912			\|a GBV_ILN_4325
912			\|a GBV_ILN_4326
912			\|a GBV_ILN_4328
912			\|a GBV_ILN_4333
912			\|a GBV_ILN_4334
912			\|a GBV_ILN_4335
912			\|a GBV_ILN_4336
912			\|a GBV_ILN_4338
912			\|a GBV_ILN_4393
912			\|a GBV_ILN_4700
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951			\|a AR
952			\|d 16 \|j 2009 \|e 1 \|b 23 \|c 12 \|h 48-54

Indexfelder

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publishDate	2009
allfields	10.1007/s00761-009-1765-2 doi (DE-627)SPR007780826 (SPR)s00761-009-1765-2-e DE-627 ger DE-627 rakwb ger 610 ASE 44.81 bkl Böll, B. verfasserin aut Experimentelle Therapie des Hodgkin-Lymphoms 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen. Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213 Borchmann, P. verfasserin aut Engert, A. verfasserin aut Enthalten in Der Onkologe Berlin : Springer, 1996 16(2009), 1 vom: 23. Dez., Seite 48-54 (DE-627)254638546 (DE-600)1462966-5 1433-0415 nnns volume:16 year:2009 number:1 day:23 month:12 pages:48-54 https://dx.doi.org/10.1007/s00761-009-1765-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2339 GBV_ILN_2414 GBV_ILN_2424 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.81 ASE AR 16 2009 1 23 12 48-54
spelling	10.1007/s00761-009-1765-2 doi (DE-627)SPR007780826 (SPR)s00761-009-1765-2-e DE-627 ger DE-627 rakwb ger 610 ASE 44.81 bkl Böll, B. verfasserin aut Experimentelle Therapie des Hodgkin-Lymphoms 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen. Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213 Borchmann, P. verfasserin aut Engert, A. verfasserin aut Enthalten in Der Onkologe Berlin : Springer, 1996 16(2009), 1 vom: 23. Dez., Seite 48-54 (DE-627)254638546 (DE-600)1462966-5 1433-0415 nnns volume:16 year:2009 number:1 day:23 month:12 pages:48-54 https://dx.doi.org/10.1007/s00761-009-1765-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2339 GBV_ILN_2414 GBV_ILN_2424 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.81 ASE AR 16 2009 1 23 12 48-54
allfields_unstemmed	10.1007/s00761-009-1765-2 doi (DE-627)SPR007780826 (SPR)s00761-009-1765-2-e DE-627 ger DE-627 rakwb ger 610 ASE 44.81 bkl Böll, B. verfasserin aut Experimentelle Therapie des Hodgkin-Lymphoms 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen. Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213 Borchmann, P. verfasserin aut Engert, A. verfasserin aut Enthalten in Der Onkologe Berlin : Springer, 1996 16(2009), 1 vom: 23. Dez., Seite 48-54 (DE-627)254638546 (DE-600)1462966-5 1433-0415 nnns volume:16 year:2009 number:1 day:23 month:12 pages:48-54 https://dx.doi.org/10.1007/s00761-009-1765-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2339 GBV_ILN_2414 GBV_ILN_2424 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.81 ASE AR 16 2009 1 23 12 48-54
allfieldsGer	10.1007/s00761-009-1765-2 doi (DE-627)SPR007780826 (SPR)s00761-009-1765-2-e DE-627 ger DE-627 rakwb ger 610 ASE 44.81 bkl Böll, B. verfasserin aut Experimentelle Therapie des Hodgkin-Lymphoms 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen. Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213 Borchmann, P. verfasserin aut Engert, A. verfasserin aut Enthalten in Der Onkologe Berlin : Springer, 1996 16(2009), 1 vom: 23. Dez., Seite 48-54 (DE-627)254638546 (DE-600)1462966-5 1433-0415 nnns volume:16 year:2009 number:1 day:23 month:12 pages:48-54 https://dx.doi.org/10.1007/s00761-009-1765-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2339 GBV_ILN_2414 GBV_ILN_2424 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.81 ASE AR 16 2009 1 23 12 48-54
allfieldsSound	10.1007/s00761-009-1765-2 doi (DE-627)SPR007780826 (SPR)s00761-009-1765-2-e DE-627 ger DE-627 rakwb ger 610 ASE 44.81 bkl Böll, B. verfasserin aut Experimentelle Therapie des Hodgkin-Lymphoms 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen. Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213 Borchmann, P. verfasserin aut Engert, A. verfasserin aut Enthalten in Der Onkologe Berlin : Springer, 1996 16(2009), 1 vom: 23. Dez., Seite 48-54 (DE-627)254638546 (DE-600)1462966-5 1433-0415 nnns volume:16 year:2009 number:1 day:23 month:12 pages:48-54 https://dx.doi.org/10.1007/s00761-009-1765-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2339 GBV_ILN_2414 GBV_ILN_2424 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.81 ASE AR 16 2009 1 23 12 48-54
language	German
source	Enthalten in Der Onkologe 16(2009), 1 vom: 23. Dez., Seite 48-54 volume:16 year:2009 number:1 day:23 month:12 pages:48-54
sourceStr	Enthalten in Der Onkologe 16(2009), 1 vom: 23. Dez., Seite 48-54 volume:16 year:2009 number:1 day:23 month:12 pages:48-54
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	Immunotherapy Small molecular substances Targeted therapy H-RS cell Relapsed Hodgkin‘s lymphoma
dewey-raw	610
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container_title	Der Onkologe
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publishDateDaySort_date	2009-12-23T00:00:00Z
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author	Böll, B.
spellingShingle	Böll, B. ddc 610 bkl 44.81 misc Immunotherapy misc Small molecular substances misc Targeted therapy misc H-RS cell misc Relapsed Hodgkin‘s lymphoma Experimentelle Therapie des Hodgkin-Lymphoms
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topic_title	610 ASE 44.81 bkl Experimentelle Therapie des Hodgkin-Lymphoms Immunotherapy (dpeaa)DE-He213 Small molecular substances (dpeaa)DE-He213 Targeted therapy (dpeaa)DE-He213 H-RS cell (dpeaa)DE-He213 Relapsed Hodgkin‘s lymphoma (dpeaa)DE-He213
topic	ddc 610 bkl 44.81 misc Immunotherapy misc Small molecular substances misc Targeted therapy misc H-RS cell misc Relapsed Hodgkin‘s lymphoma
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title_sort	experimentelle therapie des hodgkin-lymphoms
title_auth	Experimentelle Therapie des Hodgkin-Lymphoms
abstract	Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen.
abstractGer	Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen.
abstract_unstemmed	Zusammenfassung Experimentelle Therapiestrategien werden derzeit bei Patienten im Rezidiv eines Hodgkin-Lymphoms nach Hochdosischemotherapie angewendet. Neben monoklonalen Antikörpern, Radioimmunkonjugaten, Immuntoxinen und zellulären Therapeutika haben insbesondere immunmodulatorische Substanzen wie Lenalidomid sowie Deacetylaseinhibitoren und mTOR-Inhibitoren eine klinisch relevante Wirksamkeit gezeigt. Derzeit laufende klinische Studien sollen helfen, neue Erkenntnisse über die Biologie der Erkrankung zu gewinnen und eine effektivere und weniger toxische Therapie des Hodgkin-Lymphoms in allen Stadien zu ermöglichen.
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container_issue	1
title_short	Experimentelle Therapie des Hodgkin-Lymphoms
url	https://dx.doi.org/10.1007/s00761-009-1765-2
remote_bool	true
author2	Borchmann, P. Engert, A.
author2Str	Borchmann, P. Engert, A.
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isOA_txt	false
hochschulschrift_bool	false
doi_str	10.1007/s00761-009-1765-2
up_date	2024-07-03T15:12:22.430Z
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