Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease
Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no conf...
Ausführliche Beschreibung
Autor*in: |
Ercin, Cemal Nuri [verfasserIn] Dogru, Teoman [verfasserIn] Tapan, Serkan [verfasserIn] Karslioglu, Yıldırım [verfasserIn] Haymana, Cem [verfasserIn] Kilic, Selim [verfasserIn] Sonmez, Alper [verfasserIn] Yesilova, Zeki [verfasserIn] Uygun, Ahmet [verfasserIn] Gulsen, Mustafa [verfasserIn] Bagci, Sait [verfasserIn] Kemal Erbil, M. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2009 |
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Schlagwörter: |
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Übergeordnetes Werk: |
Enthalten in: Digestive diseases and sciences - Dordrecht : Springer Science + Business Media B.V., 1934, 55(2009), 4 vom: 14. Mai, Seite 1128-1134 |
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Übergeordnetes Werk: |
volume:55 ; year:2009 ; number:4 ; day:14 ; month:05 ; pages:1128-1134 |
Links: |
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DOI / URN: |
10.1007/s10620-009-0817-1 |
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Katalog-ID: |
SPR011854030 |
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520 | |a Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. | ||
650 | 4 | |a Soluble CD40 ligand |7 (dpeaa)DE-He213 | |
650 | 4 | |a Soluble P-selectin |7 (dpeaa)DE-He213 | |
650 | 4 | |a NAFLD |7 (dpeaa)DE-He213 | |
650 | 4 | |a Atherosclerosis |7 (dpeaa)DE-He213 | |
700 | 1 | |a Dogru, Teoman |e verfasserin |4 aut | |
700 | 1 | |a Tapan, Serkan |e verfasserin |4 aut | |
700 | 1 | |a Karslioglu, Yıldırım |e verfasserin |4 aut | |
700 | 1 | |a Haymana, Cem |e verfasserin |4 aut | |
700 | 1 | |a Kilic, Selim |e verfasserin |4 aut | |
700 | 1 | |a Sonmez, Alper |e verfasserin |4 aut | |
700 | 1 | |a Yesilova, Zeki |e verfasserin |4 aut | |
700 | 1 | |a Uygun, Ahmet |e verfasserin |4 aut | |
700 | 1 | |a Gulsen, Mustafa |e verfasserin |4 aut | |
700 | 1 | |a Bagci, Sait |e verfasserin |4 aut | |
700 | 1 | |a Kemal Erbil, M. |e verfasserin |4 aut | |
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2009 |
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10.1007/s10620-009-0817-1 doi (DE-627)SPR011854030 (SPR)s10620-009-0817-1-e DE-627 ger DE-627 rakwb eng 610 ASE 610 ASE 44.87 bkl Ercin, Cemal Nuri verfasserin aut Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 Dogru, Teoman verfasserin aut Tapan, Serkan verfasserin aut Karslioglu, Yıldırım verfasserin aut Haymana, Cem verfasserin aut Kilic, Selim verfasserin aut Sonmez, Alper verfasserin aut Yesilova, Zeki verfasserin aut Uygun, Ahmet verfasserin aut Gulsen, Mustafa verfasserin aut Bagci, Sait verfasserin aut Kemal Erbil, M. verfasserin aut Enthalten in Digestive diseases and sciences Dordrecht : Springer Science + Business Media B.V., 1934 55(2009), 4 vom: 14. Mai, Seite 1128-1134 (DE-627)320525384 (DE-600)2015102-0 1573-2568 nnns volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 https://dx.doi.org/10.1007/s10620-009-0817-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.87 ASE AR 55 2009 4 14 05 1128-1134 |
spelling |
10.1007/s10620-009-0817-1 doi (DE-627)SPR011854030 (SPR)s10620-009-0817-1-e DE-627 ger DE-627 rakwb eng 610 ASE 610 ASE 44.87 bkl Ercin, Cemal Nuri verfasserin aut Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 Dogru, Teoman verfasserin aut Tapan, Serkan verfasserin aut Karslioglu, Yıldırım verfasserin aut Haymana, Cem verfasserin aut Kilic, Selim verfasserin aut Sonmez, Alper verfasserin aut Yesilova, Zeki verfasserin aut Uygun, Ahmet verfasserin aut Gulsen, Mustafa verfasserin aut Bagci, Sait verfasserin aut Kemal Erbil, M. verfasserin aut Enthalten in Digestive diseases and sciences Dordrecht : Springer Science + Business Media B.V., 1934 55(2009), 4 vom: 14. Mai, Seite 1128-1134 (DE-627)320525384 (DE-600)2015102-0 1573-2568 nnns volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 https://dx.doi.org/10.1007/s10620-009-0817-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.87 ASE AR 55 2009 4 14 05 1128-1134 |
allfields_unstemmed |
10.1007/s10620-009-0817-1 doi (DE-627)SPR011854030 (SPR)s10620-009-0817-1-e DE-627 ger DE-627 rakwb eng 610 ASE 610 ASE 44.87 bkl Ercin, Cemal Nuri verfasserin aut Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 Dogru, Teoman verfasserin aut Tapan, Serkan verfasserin aut Karslioglu, Yıldırım verfasserin aut Haymana, Cem verfasserin aut Kilic, Selim verfasserin aut Sonmez, Alper verfasserin aut Yesilova, Zeki verfasserin aut Uygun, Ahmet verfasserin aut Gulsen, Mustafa verfasserin aut Bagci, Sait verfasserin aut Kemal Erbil, M. verfasserin aut Enthalten in Digestive diseases and sciences Dordrecht : Springer Science + Business Media B.V., 1934 55(2009), 4 vom: 14. Mai, Seite 1128-1134 (DE-627)320525384 (DE-600)2015102-0 1573-2568 nnns volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 https://dx.doi.org/10.1007/s10620-009-0817-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.87 ASE AR 55 2009 4 14 05 1128-1134 |
allfieldsGer |
10.1007/s10620-009-0817-1 doi (DE-627)SPR011854030 (SPR)s10620-009-0817-1-e DE-627 ger DE-627 rakwb eng 610 ASE 610 ASE 44.87 bkl Ercin, Cemal Nuri verfasserin aut Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 Dogru, Teoman verfasserin aut Tapan, Serkan verfasserin aut Karslioglu, Yıldırım verfasserin aut Haymana, Cem verfasserin aut Kilic, Selim verfasserin aut Sonmez, Alper verfasserin aut Yesilova, Zeki verfasserin aut Uygun, Ahmet verfasserin aut Gulsen, Mustafa verfasserin aut Bagci, Sait verfasserin aut Kemal Erbil, M. verfasserin aut Enthalten in Digestive diseases and sciences Dordrecht : Springer Science + Business Media B.V., 1934 55(2009), 4 vom: 14. Mai, Seite 1128-1134 (DE-627)320525384 (DE-600)2015102-0 1573-2568 nnns volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 https://dx.doi.org/10.1007/s10620-009-0817-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.87 ASE AR 55 2009 4 14 05 1128-1134 |
allfieldsSound |
10.1007/s10620-009-0817-1 doi (DE-627)SPR011854030 (SPR)s10620-009-0817-1-e DE-627 ger DE-627 rakwb eng 610 ASE 610 ASE 44.87 bkl Ercin, Cemal Nuri verfasserin aut Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 Dogru, Teoman verfasserin aut Tapan, Serkan verfasserin aut Karslioglu, Yıldırım verfasserin aut Haymana, Cem verfasserin aut Kilic, Selim verfasserin aut Sonmez, Alper verfasserin aut Yesilova, Zeki verfasserin aut Uygun, Ahmet verfasserin aut Gulsen, Mustafa verfasserin aut Bagci, Sait verfasserin aut Kemal Erbil, M. verfasserin aut Enthalten in Digestive diseases and sciences Dordrecht : Springer Science + Business Media B.V., 1934 55(2009), 4 vom: 14. Mai, Seite 1128-1134 (DE-627)320525384 (DE-600)2015102-0 1573-2568 nnns volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 https://dx.doi.org/10.1007/s10620-009-0817-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_267 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_711 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.87 ASE AR 55 2009 4 14 05 1128-1134 |
language |
English |
source |
Enthalten in Digestive diseases and sciences 55(2009), 4 vom: 14. Mai, Seite 1128-1134 volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 |
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Enthalten in Digestive diseases and sciences 55(2009), 4 vom: 14. Mai, Seite 1128-1134 volume:55 year:2009 number:4 day:14 month:05 pages:1128-1134 |
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Soluble CD40 ligand Soluble P-selectin NAFLD Atherosclerosis |
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Digestive diseases and sciences |
authorswithroles_txt_mv |
Ercin, Cemal Nuri @@aut@@ Dogru, Teoman @@aut@@ Tapan, Serkan @@aut@@ Karslioglu, Yıldırım @@aut@@ Haymana, Cem @@aut@@ Kilic, Selim @@aut@@ Sonmez, Alper @@aut@@ Yesilova, Zeki @@aut@@ Uygun, Ahmet @@aut@@ Gulsen, Mustafa @@aut@@ Bagci, Sait @@aut@@ Kemal Erbil, M. @@aut@@ |
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2009-05-14T00:00:00Z |
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320525384 |
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3610 |
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|
author |
Ercin, Cemal Nuri |
spellingShingle |
Ercin, Cemal Nuri ddc 610 bkl 44.87 misc Soluble CD40 ligand misc Soluble P-selectin misc NAFLD misc Atherosclerosis Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease |
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Ercin, Cemal Nuri |
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1573-2568 |
topic_title |
610 ASE 44.87 bkl Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease Soluble CD40 ligand (dpeaa)DE-He213 Soluble P-selectin (dpeaa)DE-He213 NAFLD (dpeaa)DE-He213 Atherosclerosis (dpeaa)DE-He213 |
topic |
ddc 610 bkl 44.87 misc Soluble CD40 ligand misc Soluble P-selectin misc NAFLD misc Atherosclerosis |
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ddc 610 bkl 44.87 misc Soluble CD40 ligand misc Soluble P-selectin misc NAFLD misc Atherosclerosis |
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ddc 610 bkl 44.87 misc Soluble CD40 ligand misc Soluble P-selectin misc NAFLD misc Atherosclerosis |
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Elektronische Aufsätze Aufsätze Elektronische Ressource |
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(DE-627)320525384 (DE-600)2015102-0 |
title |
Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease |
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(DE-627)SPR011854030 (SPR)s10620-009-0817-1-e |
title_full |
Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease |
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Ercin, Cemal Nuri |
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Ercin, Cemal Nuri Dogru, Teoman Tapan, Serkan Karslioglu, Yıldırım Haymana, Cem Kilic, Selim Sonmez, Alper Yesilova, Zeki Uygun, Ahmet Gulsen, Mustafa Bagci, Sait Kemal Erbil, M. |
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author-letter |
Ercin, Cemal Nuri |
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title_sort |
levels of soluble cd40 ligand and p-selectin in nonalcoholic fatty liver disease |
title_auth |
Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease |
abstract |
Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. |
abstractGer |
Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. |
abstract_unstemmed |
Abstract Nonalcoholic fatty liver disease (NAFLD) is linked to an increased risk of cardiovascular disease. We aimed to research whether the levels of soluble P-selectin (sP-selectin) and soluble CD40 ligand (sCD40L), markers of endothelial function, are altered in subjects with NAFLD having no confounding factors for atherosclerosis. sCD40L, sP-selectin, and high-sensitivity C-reactive protein (hsCRP) levels, and homeostasis model assessment of insulin resistance (HOMA-IR) indexes were measured in 50 NAFLD subjects and 30 healthy controls. sCD40L, sP-selectin, and hsCRP levels were not significantly different between two groups (P = 0.48, 0.51, and 0.34, respectively). Body mass index, waist circumference, and insulin levels and HOMA indexes were significantly higher in subjects with NAFLD (all P < 0.001). The present data show that sCD40L and sP-selectin may not contribute to the accelerated atherogenesis associated with this clinically relevant condition. |
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title_short |
Levels of Soluble CD40 Ligand and P-Selectin in Nonalcoholic Fatty Liver Disease |
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Dogru, Teoman Tapan, Serkan Karslioglu, Yıldırım Haymana, Cem Kilic, Selim Sonmez, Alper Yesilova, Zeki Uygun, Ahmet Gulsen, Mustafa Bagci, Sait Kemal Erbil, M. |
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Dogru, Teoman Tapan, Serkan Karslioglu, Yıldırım Haymana, Cem Kilic, Selim Sonmez, Alper Yesilova, Zeki Uygun, Ahmet Gulsen, Mustafa Bagci, Sait Kemal Erbil, M. |
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|
score |
7.401908 |