Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken
Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intrac...
Ausführliche Beschreibung
Autor*in: |
Zendehdel, Morteza [verfasserIn] Hassanpour, Shahin [verfasserIn] Babapour, Vahab [verfasserIn] Charkhkar, Saeed [verfasserIn] Mahdavi, Mahshid [verfasserIn] |
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E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2015 |
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Schlagwörter: |
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Übergeordnetes Werk: |
Enthalten in: Letters in peptide science - Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994, 21(2015), 3 vom: 27. Feb., Seite 289-297 |
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Übergeordnetes Werk: |
volume:21 ; year:2015 ; number:3 ; day:27 ; month:02 ; pages:289-297 |
Links: |
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DOI / URN: |
10.1007/s10989-015-9457-9 |
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Katalog-ID: |
SPR015391140 |
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520 | |a Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. | ||
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10.1007/s10989-015-9457-9 doi (DE-627)SPR015391140 (SPR)s10989-015-9457-9-e DE-627 ger DE-627 rakwb eng 540 ASE 35.76 bkl Zendehdel, Morteza verfasserin aut Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 Hassanpour, Shahin verfasserin aut Babapour, Vahab verfasserin aut Charkhkar, Saeed verfasserin aut Mahdavi, Mahshid verfasserin aut Enthalten in Letters in peptide science Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994 21(2015), 3 vom: 27. Feb., Seite 289-297 (DE-627)311005055 (DE-600)2007589-3 1573-496X nnns volume:21 year:2015 number:3 day:27 month:02 pages:289-297 https://dx.doi.org/10.1007/s10989-015-9457-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_152 GBV_ILN_161 GBV_ILN_171 GBV_ILN_187 GBV_ILN_224 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 35.76 ASE AR 21 2015 3 27 02 289-297 |
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10.1007/s10989-015-9457-9 doi (DE-627)SPR015391140 (SPR)s10989-015-9457-9-e DE-627 ger DE-627 rakwb eng 540 ASE 35.76 bkl Zendehdel, Morteza verfasserin aut Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 Hassanpour, Shahin verfasserin aut Babapour, Vahab verfasserin aut Charkhkar, Saeed verfasserin aut Mahdavi, Mahshid verfasserin aut Enthalten in Letters in peptide science Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994 21(2015), 3 vom: 27. Feb., Seite 289-297 (DE-627)311005055 (DE-600)2007589-3 1573-496X nnns volume:21 year:2015 number:3 day:27 month:02 pages:289-297 https://dx.doi.org/10.1007/s10989-015-9457-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_152 GBV_ILN_161 GBV_ILN_171 GBV_ILN_187 GBV_ILN_224 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 35.76 ASE AR 21 2015 3 27 02 289-297 |
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10.1007/s10989-015-9457-9 doi (DE-627)SPR015391140 (SPR)s10989-015-9457-9-e DE-627 ger DE-627 rakwb eng 540 ASE 35.76 bkl Zendehdel, Morteza verfasserin aut Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 Hassanpour, Shahin verfasserin aut Babapour, Vahab verfasserin aut Charkhkar, Saeed verfasserin aut Mahdavi, Mahshid verfasserin aut Enthalten in Letters in peptide science Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994 21(2015), 3 vom: 27. Feb., Seite 289-297 (DE-627)311005055 (DE-600)2007589-3 1573-496X nnns volume:21 year:2015 number:3 day:27 month:02 pages:289-297 https://dx.doi.org/10.1007/s10989-015-9457-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_152 GBV_ILN_161 GBV_ILN_171 GBV_ILN_187 GBV_ILN_224 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 35.76 ASE AR 21 2015 3 27 02 289-297 |
allfieldsGer |
10.1007/s10989-015-9457-9 doi (DE-627)SPR015391140 (SPR)s10989-015-9457-9-e DE-627 ger DE-627 rakwb eng 540 ASE 35.76 bkl Zendehdel, Morteza verfasserin aut Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 Hassanpour, Shahin verfasserin aut Babapour, Vahab verfasserin aut Charkhkar, Saeed verfasserin aut Mahdavi, Mahshid verfasserin aut Enthalten in Letters in peptide science Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994 21(2015), 3 vom: 27. Feb., Seite 289-297 (DE-627)311005055 (DE-600)2007589-3 1573-496X nnns volume:21 year:2015 number:3 day:27 month:02 pages:289-297 https://dx.doi.org/10.1007/s10989-015-9457-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_152 GBV_ILN_161 GBV_ILN_171 GBV_ILN_187 GBV_ILN_224 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 35.76 ASE AR 21 2015 3 27 02 289-297 |
allfieldsSound |
10.1007/s10989-015-9457-9 doi (DE-627)SPR015391140 (SPR)s10989-015-9457-9-e DE-627 ger DE-627 rakwb eng 540 ASE 35.76 bkl Zendehdel, Morteza verfasserin aut Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 Hassanpour, Shahin verfasserin aut Babapour, Vahab verfasserin aut Charkhkar, Saeed verfasserin aut Mahdavi, Mahshid verfasserin aut Enthalten in Letters in peptide science Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994 21(2015), 3 vom: 27. Feb., Seite 289-297 (DE-627)311005055 (DE-600)2007589-3 1573-496X nnns volume:21 year:2015 number:3 day:27 month:02 pages:289-297 https://dx.doi.org/10.1007/s10989-015-9457-9 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_152 GBV_ILN_161 GBV_ILN_171 GBV_ILN_187 GBV_ILN_224 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 35.76 ASE AR 21 2015 3 27 02 289-297 |
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540 ASE 35.76 bkl Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken Cannabinoidergic (dpeaa)DE-He213 Opioidergic system (dpeaa)DE-He213 Food intake (dpeaa)DE-He213 Chicken (dpeaa)DE-He213 |
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interaction between endocannabinoid and opioidergic systems regulates food intake in neonatal chicken |
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Interaction Between Endocannabinoid and Opioidergic Systems Regulates Food Intake in Neonatal Chicken |
abstract |
Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. |
abstractGer |
Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. |
abstract_unstemmed |
Abstract Endocannabinoids and opiates have regulatory role in some physiological functions in mammals but their interaction(s) have not been studied in avian. This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken. |
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This survey is designed to investigate interaction of these systems on feeding behavior in neonatal chickens. In experiment 1, chicken intracerebroventricular (ICV) injected with saline, DAMGO (µ-opioid receptors agonist, 125 pmol), SR141716A ($ CB_{1} $ receptors antagonist, 6.25 µg) and SR141716A + DAMGO. In experiment 2, saline, DAMGO, AM630 ($ CB_{2} $ receptors antagonist, 1.25 µg) and DAMGO + AM630. Experiments 3–6 followed the procedure similar to experiments 1 and 2, except DPDPE (δ-opioid receptors agonist, 40 pmol) and U-50488H (κ-opioid receptors agonist, 30 nmol) instead of DAMGO were used. In experiment 7, saline, Naloxone (opioid receptors antagonist, 5 µg), 2-AG ($ CB_{1} $ receptors agonist, 2 µg), Naloxone + 2-AG were used. Experiment 8 was similar to experiment 7, except CB65 ($ CB_{2} $ receptors agonist, 1.25 µg) used instead of 2-AG. Cumulative food intake was recorded until 120 min post injection. Data provided that, ICV injection of DAMGO decreased food intake and its effect amplified by $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P < 0.001). DPDPE increased food intake and $ CB_{2} $ receptors antagonist blocked DPDPE-induced hyperphagia (P < 0.001). U-50488H-induced feeding but its effect did not alter via $ CB_{1} $ and $ CB_{2} $ receptors antagonist (P > 0.05). Hyperphagia-induced by $ CB_{1} $ and $ CB_{2} $ receptors agonist amplified by naloxone (P < 0.001). Perhaps there is interaction between endocannabinoid and opioidergic systems on appetite regulation in chicken.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Cannabinoidergic</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Opioidergic system</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Food intake</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Chicken</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Hassanpour, Shahin</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Babapour, Vahab</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Charkhkar, Saeed</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Mahdavi, Mahshid</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Letters in peptide science</subfield><subfield code="d">Dordrecht [u.a.] : Springer Science + Business Media B.V, 1994</subfield><subfield code="g">21(2015), 3 vom: 27. 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