Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence
Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in de...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Smilowitz, Nathaniel R. [verfasserIn] Naoulou, Becky [verfasserIn] Sedlis, Steven P. [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2015 |
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| Schlagwörter: |
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| Übergeordnetes Werk: |
Enthalten in: Current atherosclerosis reports - Heidelberg [u.a.] : Springer, 1999, 17(2015), 2 vom: 27. Jan. |
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| Übergeordnetes Werk: |
volume:17 ; year:2015 ; number:2 ; day:27 ; month:01 |
| Links: |
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| DOI / URN: |
10.1007/s11883-014-0478-1 |
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| Katalog-ID: |
SPR022861580 |
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| 100 | 1 | |a Smilowitz, Nathaniel R. |e verfasserin |4 aut | |
| 245 | 1 | 0 | |a Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence |
| 264 | 1 | |c 2015 | |
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| 520 | |a Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. | ||
| 650 | 4 | |a Type 2 |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Myocardial infarction |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Myocardial necrosis |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Supply-demand mismatch |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Perioperative MI |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Endothelial dysfunction |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Vasospasm |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Anemia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Respiratory failure |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Hypoxia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Tachyarrhythmia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Bradyarrhythmia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Hypotension |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Hypertension |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Shock |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Sepsis |7 (dpeaa)DE-He213 | |
| 700 | 1 | |a Naoulou, Becky |e verfasserin |4 aut | |
| 700 | 1 | |a Sedlis, Steven P. |e verfasserin |4 aut | |
| 773 | 0 | 8 | |i Enthalten in |t Current atherosclerosis reports |d Heidelberg [u.a.] : Springer, 1999 |g 17(2015), 2 vom: 27. Jan. |w (DE-627)357168178 |w (DE-600)2094154-7 |x 1534-6242 |7 nnns |
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| 856 | 4 | 0 | |u https://dx.doi.org/10.1007/s11883-014-0478-1 |z lizenzpflichtig |3 Volltext |
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10.1007/s11883-014-0478-1 doi (DE-627)SPR022861580 (SPR)s11883-014-0478-1-e DE-627 ger DE-627 rakwb eng 610 ASE 44.85 bkl Smilowitz, Nathaniel R. verfasserin aut Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 Naoulou, Becky verfasserin aut Sedlis, Steven P. verfasserin aut Enthalten in Current atherosclerosis reports Heidelberg [u.a.] : Springer, 1999 17(2015), 2 vom: 27. Jan. (DE-627)357168178 (DE-600)2094154-7 1534-6242 nnns volume:17 year:2015 number:2 day:27 month:01 https://dx.doi.org/10.1007/s11883-014-0478-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.85 ASE AR 17 2015 2 27 01 |
| spelling |
10.1007/s11883-014-0478-1 doi (DE-627)SPR022861580 (SPR)s11883-014-0478-1-e DE-627 ger DE-627 rakwb eng 610 ASE 44.85 bkl Smilowitz, Nathaniel R. verfasserin aut Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 Naoulou, Becky verfasserin aut Sedlis, Steven P. verfasserin aut Enthalten in Current atherosclerosis reports Heidelberg [u.a.] : Springer, 1999 17(2015), 2 vom: 27. Jan. (DE-627)357168178 (DE-600)2094154-7 1534-6242 nnns volume:17 year:2015 number:2 day:27 month:01 https://dx.doi.org/10.1007/s11883-014-0478-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.85 ASE AR 17 2015 2 27 01 |
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10.1007/s11883-014-0478-1 doi (DE-627)SPR022861580 (SPR)s11883-014-0478-1-e DE-627 ger DE-627 rakwb eng 610 ASE 44.85 bkl Smilowitz, Nathaniel R. verfasserin aut Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 Naoulou, Becky verfasserin aut Sedlis, Steven P. verfasserin aut Enthalten in Current atherosclerosis reports Heidelberg [u.a.] : Springer, 1999 17(2015), 2 vom: 27. Jan. (DE-627)357168178 (DE-600)2094154-7 1534-6242 nnns volume:17 year:2015 number:2 day:27 month:01 https://dx.doi.org/10.1007/s11883-014-0478-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.85 ASE AR 17 2015 2 27 01 |
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10.1007/s11883-014-0478-1 doi (DE-627)SPR022861580 (SPR)s11883-014-0478-1-e DE-627 ger DE-627 rakwb eng 610 ASE 44.85 bkl Smilowitz, Nathaniel R. verfasserin aut Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 Naoulou, Becky verfasserin aut Sedlis, Steven P. verfasserin aut Enthalten in Current atherosclerosis reports Heidelberg [u.a.] : Springer, 1999 17(2015), 2 vom: 27. Jan. (DE-627)357168178 (DE-600)2094154-7 1534-6242 nnns volume:17 year:2015 number:2 day:27 month:01 https://dx.doi.org/10.1007/s11883-014-0478-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.85 ASE AR 17 2015 2 27 01 |
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10.1007/s11883-014-0478-1 doi (DE-627)SPR022861580 (SPR)s11883-014-0478-1-e DE-627 ger DE-627 rakwb eng 610 ASE 44.85 bkl Smilowitz, Nathaniel R. verfasserin aut Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence 2015 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 Naoulou, Becky verfasserin aut Sedlis, Steven P. verfasserin aut Enthalten in Current atherosclerosis reports Heidelberg [u.a.] : Springer, 1999 17(2015), 2 vom: 27. Jan. (DE-627)357168178 (DE-600)2094154-7 1534-6242 nnns volume:17 year:2015 number:2 day:27 month:01 https://dx.doi.org/10.1007/s11883-014-0478-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.85 ASE AR 17 2015 2 27 01 |
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Enthalten in Current atherosclerosis reports 17(2015), 2 vom: 27. Jan. volume:17 year:2015 number:2 day:27 month:01 |
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Type 2 Myocardial infarction Myocardial necrosis Supply-demand mismatch Perioperative MI Endothelial dysfunction Vasospasm Anemia Respiratory failure Hypoxia Tachyarrhythmia Bradyarrhythmia Hypotension Hypertension Shock Sepsis |
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Smilowitz, Nathaniel R. @@aut@@ Naoulou, Becky @@aut@@ Sedlis, Steven P. @@aut@@ |
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Smilowitz, Nathaniel R. |
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Smilowitz, Nathaniel R. ddc 610 bkl 44.85 misc Type 2 misc Myocardial infarction misc Myocardial necrosis misc Supply-demand mismatch misc Perioperative MI misc Endothelial dysfunction misc Vasospasm misc Anemia misc Respiratory failure misc Hypoxia misc Tachyarrhythmia misc Bradyarrhythmia misc Hypotension misc Hypertension misc Shock misc Sepsis Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence |
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610 ASE 44.85 bkl Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence Type 2 (dpeaa)DE-He213 Myocardial infarction (dpeaa)DE-He213 Myocardial necrosis (dpeaa)DE-He213 Supply-demand mismatch (dpeaa)DE-He213 Perioperative MI (dpeaa)DE-He213 Endothelial dysfunction (dpeaa)DE-He213 Vasospasm (dpeaa)DE-He213 Anemia (dpeaa)DE-He213 Respiratory failure (dpeaa)DE-He213 Hypoxia (dpeaa)DE-He213 Tachyarrhythmia (dpeaa)DE-He213 Bradyarrhythmia (dpeaa)DE-He213 Hypotension (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Shock (dpeaa)DE-He213 Sepsis (dpeaa)DE-He213 |
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ddc 610 bkl 44.85 misc Type 2 misc Myocardial infarction misc Myocardial necrosis misc Supply-demand mismatch misc Perioperative MI misc Endothelial dysfunction misc Vasospasm misc Anemia misc Respiratory failure misc Hypoxia misc Tachyarrhythmia misc Bradyarrhythmia misc Hypotension misc Hypertension misc Shock misc Sepsis |
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ddc 610 bkl 44.85 misc Type 2 misc Myocardial infarction misc Myocardial necrosis misc Supply-demand mismatch misc Perioperative MI misc Endothelial dysfunction misc Vasospasm misc Anemia misc Respiratory failure misc Hypoxia misc Tachyarrhythmia misc Bradyarrhythmia misc Hypotension misc Hypertension misc Shock misc Sepsis |
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ddc 610 bkl 44.85 misc Type 2 misc Myocardial infarction misc Myocardial necrosis misc Supply-demand mismatch misc Perioperative MI misc Endothelial dysfunction misc Vasospasm misc Anemia misc Respiratory failure misc Hypoxia misc Tachyarrhythmia misc Bradyarrhythmia misc Hypotension misc Hypertension misc Shock misc Sepsis |
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Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence |
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Smilowitz, Nathaniel R. |
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Current atherosclerosis reports |
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Smilowitz, Nathaniel R. |
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diagnosis and management of type ii myocardial infarction: increased demand for a limited supply of evidence |
| title_auth |
Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence |
| abstract |
Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. |
| abstractGer |
Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. |
| abstract_unstemmed |
Abstract Type 2 myocardial infarction (type 2 MI) is defined as myocardial necrosis that results from an imbalance of myocardial oxygen supply and demand. Although type 2 MI is highly prevalent and strongly associated with mortality, the pathophysiology remains poorly understood. Discrepancies in definitions, frequency of screening, diagnostic approaches, and methods of adjudication lead to confusion and misclassification. To date, there is no consensus on the diagnostic criteria for type 2 MI. No guidelines exist for the optimal management of this condition, and further investigation is urgently needed. This review explores the existing evidence on the pathophysiology, diagnosis, prognosis, and management of type 2 MI. |
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Diagnosis and Management of Type II Myocardial Infarction: Increased Demand for a Limited Supply of Evidence |
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| score |
7.4001474 |