Frontotemporal Dementia and Primary Progressive Aphasia: An Update
Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe an...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Kirshner, Howard S. [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2010 |
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| Schlagwörter: |
Frontotemporal lobar degeneration (FTLG) Primary progressive aphasia (PPA) Logopenic or logopenic/phonological progressive aphasia |
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| Übergeordnetes Werk: |
Enthalten in: Current neurology and neuroscience reports - Philadelphia, Pa. : Current Science Inc., 2001, 10(2010), 6 vom: 18. Aug., Seite 504-511 |
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| Übergeordnetes Werk: |
volume:10 ; year:2010 ; number:6 ; day:18 ; month:08 ; pages:504-511 |
| Links: |
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| DOI / URN: |
10.1007/s11910-010-0145-z |
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SPR02297296X |
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| 520 | |a Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. | ||
| 650 | 4 | |a Frontotemporal dementia (FTD) |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Frontotemporal lobar degeneration (FTLG) |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Primary progressive aphasia (PPA) |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Progressive nonfluent aphasia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Semantic dementia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Logopenic or logopenic/phonological progressive aphasia |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Tauopathy |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Ubiquitinopathy |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Progranulin mutation |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a FTD/MND |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a MAP |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a TAR-DNA binding protein (TDP-43) |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Corticobasal degeneration |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Progressive supranuclear palsy |7 (dpeaa)DE-He213 | |
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10.1007/s11910-010-0145-z doi (DE-627)SPR02297296X (SPR)s11910-010-0145-z-e DE-627 ger DE-627 rakwb eng 610 ASE 44.90 bkl Kirshner, Howard S. verfasserin aut Frontotemporal Dementia and Primary Progressive Aphasia: An Update 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 Enthalten in Current neurology and neuroscience reports Philadelphia, Pa. : Current Science Inc., 2001 10(2010), 6 vom: 18. Aug., Seite 504-511 (DE-627)35716833X (DE-600)2094171-7 1534-6293 nnns volume:10 year:2010 number:6 day:18 month:08 pages:504-511 https://dx.doi.org/10.1007/s11910-010-0145-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.90 ASE AR 10 2010 6 18 08 504-511 |
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10.1007/s11910-010-0145-z doi (DE-627)SPR02297296X (SPR)s11910-010-0145-z-e DE-627 ger DE-627 rakwb eng 610 ASE 44.90 bkl Kirshner, Howard S. verfasserin aut Frontotemporal Dementia and Primary Progressive Aphasia: An Update 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 Enthalten in Current neurology and neuroscience reports Philadelphia, Pa. : Current Science Inc., 2001 10(2010), 6 vom: 18. Aug., Seite 504-511 (DE-627)35716833X (DE-600)2094171-7 1534-6293 nnns volume:10 year:2010 number:6 day:18 month:08 pages:504-511 https://dx.doi.org/10.1007/s11910-010-0145-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.90 ASE AR 10 2010 6 18 08 504-511 |
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10.1007/s11910-010-0145-z doi (DE-627)SPR02297296X (SPR)s11910-010-0145-z-e DE-627 ger DE-627 rakwb eng 610 ASE 44.90 bkl Kirshner, Howard S. verfasserin aut Frontotemporal Dementia and Primary Progressive Aphasia: An Update 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 Enthalten in Current neurology and neuroscience reports Philadelphia, Pa. : Current Science Inc., 2001 10(2010), 6 vom: 18. Aug., Seite 504-511 (DE-627)35716833X (DE-600)2094171-7 1534-6293 nnns volume:10 year:2010 number:6 day:18 month:08 pages:504-511 https://dx.doi.org/10.1007/s11910-010-0145-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.90 ASE AR 10 2010 6 18 08 504-511 |
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10.1007/s11910-010-0145-z doi (DE-627)SPR02297296X (SPR)s11910-010-0145-z-e DE-627 ger DE-627 rakwb eng 610 ASE 44.90 bkl Kirshner, Howard S. verfasserin aut Frontotemporal Dementia and Primary Progressive Aphasia: An Update 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 Enthalten in Current neurology and neuroscience reports Philadelphia, Pa. : Current Science Inc., 2001 10(2010), 6 vom: 18. Aug., Seite 504-511 (DE-627)35716833X (DE-600)2094171-7 1534-6293 nnns volume:10 year:2010 number:6 day:18 month:08 pages:504-511 https://dx.doi.org/10.1007/s11910-010-0145-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.90 ASE AR 10 2010 6 18 08 504-511 |
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10.1007/s11910-010-0145-z doi (DE-627)SPR02297296X (SPR)s11910-010-0145-z-e DE-627 ger DE-627 rakwb eng 610 ASE 44.90 bkl Kirshner, Howard S. verfasserin aut Frontotemporal Dementia and Primary Progressive Aphasia: An Update 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 Enthalten in Current neurology and neuroscience reports Philadelphia, Pa. : Current Science Inc., 2001 10(2010), 6 vom: 18. Aug., Seite 504-511 (DE-627)35716833X (DE-600)2094171-7 1534-6293 nnns volume:10 year:2010 number:6 day:18 month:08 pages:504-511 https://dx.doi.org/10.1007/s11910-010-0145-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.90 ASE AR 10 2010 6 18 08 504-511 |
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Enthalten in Current neurology and neuroscience reports 10(2010), 6 vom: 18. Aug., Seite 504-511 volume:10 year:2010 number:6 day:18 month:08 pages:504-511 |
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Frontotemporal dementia (FTD) Frontotemporal lobar degeneration (FTLG) Primary progressive aphasia (PPA) Progressive nonfluent aphasia Semantic dementia Logopenic or logopenic/phonological progressive aphasia Tauopathy Ubiquitinopathy Progranulin mutation FTD/MND MAP TAR-DNA binding protein (TDP-43) Corticobasal degeneration Progressive supranuclear palsy |
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Kirshner, Howard S. |
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Kirshner, Howard S. ddc 610 bkl 44.90 misc Frontotemporal dementia (FTD) misc Frontotemporal lobar degeneration (FTLG) misc Primary progressive aphasia (PPA) misc Progressive nonfluent aphasia misc Semantic dementia misc Logopenic or logopenic/phonological progressive aphasia misc Tauopathy misc Ubiquitinopathy misc Progranulin mutation misc FTD/MND misc MAP misc TAR-DNA binding protein (TDP-43) misc Corticobasal degeneration misc Progressive supranuclear palsy Frontotemporal Dementia and Primary Progressive Aphasia: An Update |
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610 ASE 44.90 bkl Frontotemporal Dementia and Primary Progressive Aphasia: An Update Frontotemporal dementia (FTD) (dpeaa)DE-He213 Frontotemporal lobar degeneration (FTLG) (dpeaa)DE-He213 Primary progressive aphasia (PPA) (dpeaa)DE-He213 Progressive nonfluent aphasia (dpeaa)DE-He213 Semantic dementia (dpeaa)DE-He213 Logopenic or logopenic/phonological progressive aphasia (dpeaa)DE-He213 Tauopathy (dpeaa)DE-He213 Ubiquitinopathy (dpeaa)DE-He213 Progranulin mutation (dpeaa)DE-He213 FTD/MND (dpeaa)DE-He213 MAP (dpeaa)DE-He213 TAR-DNA binding protein (TDP-43) (dpeaa)DE-He213 Corticobasal degeneration (dpeaa)DE-He213 Progressive supranuclear palsy (dpeaa)DE-He213 |
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ddc 610 bkl 44.90 misc Frontotemporal dementia (FTD) misc Frontotemporal lobar degeneration (FTLG) misc Primary progressive aphasia (PPA) misc Progressive nonfluent aphasia misc Semantic dementia misc Logopenic or logopenic/phonological progressive aphasia misc Tauopathy misc Ubiquitinopathy misc Progranulin mutation misc FTD/MND misc MAP misc TAR-DNA binding protein (TDP-43) misc Corticobasal degeneration misc Progressive supranuclear palsy |
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ddc 610 bkl 44.90 misc Frontotemporal dementia (FTD) misc Frontotemporal lobar degeneration (FTLG) misc Primary progressive aphasia (PPA) misc Progressive nonfluent aphasia misc Semantic dementia misc Logopenic or logopenic/phonological progressive aphasia misc Tauopathy misc Ubiquitinopathy misc Progranulin mutation misc FTD/MND misc MAP misc TAR-DNA binding protein (TDP-43) misc Corticobasal degeneration misc Progressive supranuclear palsy |
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frontotemporal dementia and primary progressive aphasia: an update |
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Frontotemporal Dementia and Primary Progressive Aphasia: An Update |
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Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. |
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Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. |
| abstract_unstemmed |
Abstract Frontotemporal dementias are syndromes of progressive dysfunction of the frontal and/or temporal lobes, either unilaterally or bilaterally. These syndromes were described clinically under the terms “primary progressive aphasia” in the United States and “frontotemporal dementia” in Europe and the United Kingdom. They are diagnosed by the clinical features of a frontal lobe neurobehavioral syndrome, or a language and cognitive deterioration. In recent years, molecular genetic findings in these syndromes, especially the tau and progranulin mutations on chromosome 17, have provided a molecular and genetic foundation for the understanding of frontotemporal dementia. These disorders are distinct from Alzheimer’s disease but have some overlap with the syndrome of corticobasal degeneration, and with motor neuron disease. Treatments remain very limited, mainly involving therapy for the mood and behavioral symptoms, but advances in the molecular and genetic understanding of these conditions will hopefully lead to more specific therapies in the future. |
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| score |
7.401963 |