Putative mechanisms of the relationship between obesity and migraine progression
Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, in...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Bigal, Marcelo E. [verfasserIn] Lipton, Richard B. [verfasserIn] |
|---|
| Format: |
E-Artikel |
|---|---|
| Sprache: |
Englisch |
| Erschienen: |
2008 |
|---|
| Schlagwörter: |
|---|
| Übergeordnetes Werk: |
Enthalten in: Current Review of Pain - Current Medicine Group, 1998, 12(2008), 3 vom: Juni, Seite 207-212 |
|---|---|
| Übergeordnetes Werk: |
volume:12 ; year:2008 ; number:3 ; month:06 ; pages:207-212 |
| Links: |
|---|
| DOI / URN: |
10.1007/s11916-008-0036-z |
|---|
| Katalog-ID: |
SPR023005289 |
|---|
| LEADER | 01000caa a22002652 4500 | ||
|---|---|---|---|
| 001 | SPR023005289 | ||
| 003 | DE-627 | ||
| 005 | 20201125053631.0 | ||
| 007 | cr uuu---uuuuu | ||
| 008 | 201006s2008 xx |||||o 00| ||eng c | ||
| 024 | 7 | |a 10.1007/s11916-008-0036-z |2 doi | |
| 035 | |a (DE-627)SPR023005289 | ||
| 035 | |a (SPR)s11916-008-0036-z-e | ||
| 040 | |a DE-627 |b ger |c DE-627 |e rakwb | ||
| 041 | |a eng | ||
| 100 | 1 | |a Bigal, Marcelo E. |e verfasserin |4 aut | |
| 245 | 1 | 0 | |a Putative mechanisms of the relationship between obesity and migraine progression |
| 264 | 1 | |c 2008 | |
| 336 | |a Text |b txt |2 rdacontent | ||
| 337 | |a Computermedien |b c |2 rdamedia | ||
| 338 | |a Online-Ressource |b cr |2 rdacarrier | ||
| 520 | |a Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. | ||
| 650 | 4 | |a Obesity |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Migraine |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Chronic Migraine |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Migraine With Aura |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Chronic Daily Headache |7 (dpeaa)DE-He213 | |
| 700 | 1 | |a Lipton, Richard B. |e verfasserin |4 aut | |
| 773 | 0 | 8 | |i Enthalten in |t Current Review of Pain |d Current Medicine Group, 1998 |g 12(2008), 3 vom: Juni, Seite 207-212 |w (DE-627)SPR023001666 |7 nnns |
| 773 | 1 | 8 | |g volume:12 |g year:2008 |g number:3 |g month:06 |g pages:207-212 |
| 856 | 4 | 0 | |u https://dx.doi.org/10.1007/s11916-008-0036-z |z lizenzpflichtig |3 Volltext |
| 912 | |a GBV_USEFLAG_A | ||
| 912 | |a SYSFLAG_A | ||
| 912 | |a GBV_SPRINGER | ||
| 912 | |a GBV_ILN_73 | ||
| 912 | |a GBV_ILN_252 | ||
| 951 | |a AR | ||
| 952 | |d 12 |j 2008 |e 3 |c 06 |h 207-212 | ||
| author_variant |
m e b me meb r b l rb rbl |
|---|---|
| matchkey_str |
bigalmarceloeliptonrichardb:2008----:uaieehnssfhrltosibtenbsta |
| hierarchy_sort_str |
2008 |
| publishDate |
2008 |
| allfields |
10.1007/s11916-008-0036-z doi (DE-627)SPR023005289 (SPR)s11916-008-0036-z-e DE-627 ger DE-627 rakwb eng Bigal, Marcelo E. verfasserin aut Putative mechanisms of the relationship between obesity and migraine progression 2008 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 Lipton, Richard B. verfasserin aut Enthalten in Current Review of Pain Current Medicine Group, 1998 12(2008), 3 vom: Juni, Seite 207-212 (DE-627)SPR023001666 nnns volume:12 year:2008 number:3 month:06 pages:207-212 https://dx.doi.org/10.1007/s11916-008-0036-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 AR 12 2008 3 06 207-212 |
| spelling |
10.1007/s11916-008-0036-z doi (DE-627)SPR023005289 (SPR)s11916-008-0036-z-e DE-627 ger DE-627 rakwb eng Bigal, Marcelo E. verfasserin aut Putative mechanisms of the relationship between obesity and migraine progression 2008 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 Lipton, Richard B. verfasserin aut Enthalten in Current Review of Pain Current Medicine Group, 1998 12(2008), 3 vom: Juni, Seite 207-212 (DE-627)SPR023001666 nnns volume:12 year:2008 number:3 month:06 pages:207-212 https://dx.doi.org/10.1007/s11916-008-0036-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 AR 12 2008 3 06 207-212 |
| allfields_unstemmed |
10.1007/s11916-008-0036-z doi (DE-627)SPR023005289 (SPR)s11916-008-0036-z-e DE-627 ger DE-627 rakwb eng Bigal, Marcelo E. verfasserin aut Putative mechanisms of the relationship between obesity and migraine progression 2008 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 Lipton, Richard B. verfasserin aut Enthalten in Current Review of Pain Current Medicine Group, 1998 12(2008), 3 vom: Juni, Seite 207-212 (DE-627)SPR023001666 nnns volume:12 year:2008 number:3 month:06 pages:207-212 https://dx.doi.org/10.1007/s11916-008-0036-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 AR 12 2008 3 06 207-212 |
| allfieldsGer |
10.1007/s11916-008-0036-z doi (DE-627)SPR023005289 (SPR)s11916-008-0036-z-e DE-627 ger DE-627 rakwb eng Bigal, Marcelo E. verfasserin aut Putative mechanisms of the relationship between obesity and migraine progression 2008 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 Lipton, Richard B. verfasserin aut Enthalten in Current Review of Pain Current Medicine Group, 1998 12(2008), 3 vom: Juni, Seite 207-212 (DE-627)SPR023001666 nnns volume:12 year:2008 number:3 month:06 pages:207-212 https://dx.doi.org/10.1007/s11916-008-0036-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 AR 12 2008 3 06 207-212 |
| allfieldsSound |
10.1007/s11916-008-0036-z doi (DE-627)SPR023005289 (SPR)s11916-008-0036-z-e DE-627 ger DE-627 rakwb eng Bigal, Marcelo E. verfasserin aut Putative mechanisms of the relationship between obesity and migraine progression 2008 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 Lipton, Richard B. verfasserin aut Enthalten in Current Review of Pain Current Medicine Group, 1998 12(2008), 3 vom: Juni, Seite 207-212 (DE-627)SPR023001666 nnns volume:12 year:2008 number:3 month:06 pages:207-212 https://dx.doi.org/10.1007/s11916-008-0036-z lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 AR 12 2008 3 06 207-212 |
| language |
English |
| source |
Enthalten in Current Review of Pain 12(2008), 3 vom: Juni, Seite 207-212 volume:12 year:2008 number:3 month:06 pages:207-212 |
| sourceStr |
Enthalten in Current Review of Pain 12(2008), 3 vom: Juni, Seite 207-212 volume:12 year:2008 number:3 month:06 pages:207-212 |
| format_phy_str_mv |
Article |
| institution |
findex.gbv.de |
| topic_facet |
Obesity Migraine Chronic Migraine Migraine With Aura Chronic Daily Headache |
| isfreeaccess_bool |
false |
| container_title |
Current Review of Pain |
| authorswithroles_txt_mv |
Bigal, Marcelo E. @@aut@@ Lipton, Richard B. @@aut@@ |
| publishDateDaySort_date |
2008-06-01T00:00:00Z |
| hierarchy_top_id |
SPR023001666 |
| id |
SPR023005289 |
| language_de |
englisch |
| fullrecord |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR023005289</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20201125053631.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2008 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11916-008-0036-z</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR023005289</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11916-008-0036-z-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Bigal, Marcelo E.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Putative mechanisms of the relationship between obesity and migraine progression</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2008</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Obesity</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Migraine</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Chronic Migraine</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Migraine With Aura</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Chronic Daily Headache</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Lipton, Richard B.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Current Review of Pain</subfield><subfield code="d">Current Medicine Group, 1998</subfield><subfield code="g">12(2008), 3 vom: Juni, Seite 207-212</subfield><subfield code="w">(DE-627)SPR023001666</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:12</subfield><subfield code="g">year:2008</subfield><subfield code="g">number:3</subfield><subfield code="g">month:06</subfield><subfield code="g">pages:207-212</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://dx.doi.org/10.1007/s11916-008-0036-z</subfield><subfield code="z">lizenzpflichtig</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_SPRINGER</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_73</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_252</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">12</subfield><subfield code="j">2008</subfield><subfield code="e">3</subfield><subfield code="c">06</subfield><subfield code="h">207-212</subfield></datafield></record></collection>
|
| author |
Bigal, Marcelo E. |
| spellingShingle |
Bigal, Marcelo E. misc Obesity misc Migraine misc Chronic Migraine misc Migraine With Aura misc Chronic Daily Headache Putative mechanisms of the relationship between obesity and migraine progression |
| authorStr |
Bigal, Marcelo E. |
| ppnlink_with_tag_str_mv |
@@773@@(DE-627)SPR023001666 |
| format |
electronic Article |
| delete_txt_mv |
keep |
| author_role |
aut aut |
| collection |
springer |
| remote_str |
true |
| illustrated |
Not Illustrated |
| topic_title |
Putative mechanisms of the relationship between obesity and migraine progression Obesity (dpeaa)DE-He213 Migraine (dpeaa)DE-He213 Chronic Migraine (dpeaa)DE-He213 Migraine With Aura (dpeaa)DE-He213 Chronic Daily Headache (dpeaa)DE-He213 |
| topic |
misc Obesity misc Migraine misc Chronic Migraine misc Migraine With Aura misc Chronic Daily Headache |
| topic_unstemmed |
misc Obesity misc Migraine misc Chronic Migraine misc Migraine With Aura misc Chronic Daily Headache |
| topic_browse |
misc Obesity misc Migraine misc Chronic Migraine misc Migraine With Aura misc Chronic Daily Headache |
| format_facet |
Elektronische Aufsätze Aufsätze Elektronische Ressource |
| format_main_str_mv |
Text Zeitschrift/Artikel |
| carriertype_str_mv |
cr |
| hierarchy_parent_title |
Current Review of Pain |
| hierarchy_parent_id |
SPR023001666 |
| hierarchy_top_title |
Current Review of Pain |
| isfreeaccess_txt |
false |
| familylinks_str_mv |
(DE-627)SPR023001666 |
| title |
Putative mechanisms of the relationship between obesity and migraine progression |
| ctrlnum |
(DE-627)SPR023005289 (SPR)s11916-008-0036-z-e |
| title_full |
Putative mechanisms of the relationship between obesity and migraine progression |
| author_sort |
Bigal, Marcelo E. |
| journal |
Current Review of Pain |
| journalStr |
Current Review of Pain |
| lang_code |
eng |
| isOA_bool |
false |
| recordtype |
marc |
| publishDateSort |
2008 |
| contenttype_str_mv |
txt |
| container_start_page |
207 |
| author_browse |
Bigal, Marcelo E. Lipton, Richard B. |
| container_volume |
12 |
| format_se |
Elektronische Aufsätze |
| author-letter |
Bigal, Marcelo E. |
| doi_str_mv |
10.1007/s11916-008-0036-z |
| author2-role |
verfasserin |
| title_sort |
putative mechanisms of the relationship between obesity and migraine progression |
| title_auth |
Putative mechanisms of the relationship between obesity and migraine progression |
| abstract |
Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. |
| abstractGer |
Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. |
| abstract_unstemmed |
Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship. |
| collection_details |
GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_73 GBV_ILN_252 |
| container_issue |
3 |
| title_short |
Putative mechanisms of the relationship between obesity and migraine progression |
| url |
https://dx.doi.org/10.1007/s11916-008-0036-z |
| remote_bool |
true |
| author2 |
Lipton, Richard B. |
| author2Str |
Lipton, Richard B. |
| ppnlink |
SPR023001666 |
| mediatype_str_mv |
c |
| isOA_txt |
false |
| hochschulschrift_bool |
false |
| doi_str |
10.1007/s11916-008-0036-z |
| up_date |
2024-07-03T16:15:57.309Z |
| _version_ |
1803575211913641984 |
| fullrecord_marcxml |
<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR023005289</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20201125053631.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2008 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11916-008-0036-z</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR023005289</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11916-008-0036-z-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Bigal, Marcelo E.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Putative mechanisms of the relationship between obesity and migraine progression</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2008</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Abstract Studies suggest that obesity is associated with migraine progression from an episodic into a chronic form. We discuss putative mechanisms to justify this relationship. Several of the inflammatory mediators that are increased in obese individuals are important in migraine pathophysiology, including interleukins and calcitonin gene-related peptide. Both migraine and obesity are prothrombotic states. Substances that are important in metabolic control are nociceptive at certain levels. Hypothalamic dysfunction in the orexin pathways seems to be a risk factor for both conditions. In addition, we discuss the importance of metabolic syndrome and autonomic dysfunction in modulating the obesity/migraine progression relationship.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Obesity</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Migraine</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Chronic Migraine</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Migraine With Aura</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Chronic Daily Headache</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Lipton, Richard B.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Current Review of Pain</subfield><subfield code="d">Current Medicine Group, 1998</subfield><subfield code="g">12(2008), 3 vom: Juni, Seite 207-212</subfield><subfield code="w">(DE-627)SPR023001666</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:12</subfield><subfield code="g">year:2008</subfield><subfield code="g">number:3</subfield><subfield code="g">month:06</subfield><subfield code="g">pages:207-212</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://dx.doi.org/10.1007/s11916-008-0036-z</subfield><subfield code="z">lizenzpflichtig</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_SPRINGER</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_73</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_ILN_252</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">12</subfield><subfield code="j">2008</subfield><subfield code="e">3</subfield><subfield code="c">06</subfield><subfield code="h">207-212</subfield></datafield></record></collection>
|
| score |
7.400216 |