Prostatitis and serum prostate-specific antigen
Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various propose...
Ausführliche Beschreibung
Autor*in: |
Sindhwani, Puneet [verfasserIn] Wilson, Christopher M. [verfasserIn] |
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Format: |
E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2005 |
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Schlagwörter: |
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Übergeordnetes Werk: |
Enthalten in: Current prostate reports - Philadelphia, Pa. : Current Science, 2003, 3(2005), 2 vom: 01. Juni, Seite 81-86 |
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Übergeordnetes Werk: |
volume:3 ; year:2005 ; number:2 ; day:01 ; month:06 ; pages:81-86 |
Links: |
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DOI / URN: |
10.1007/s11918-005-0020-6 |
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SPR023014776 |
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10.1007/s11918-005-0020-6 doi (DE-627)SPR023014776 (SPR)s11918-005-0020-6-e DE-627 ger DE-627 rakwb eng 610 ASE Sindhwani, Puneet verfasserin aut Prostatitis and serum prostate-specific antigen 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. Prostatitis (dpeaa)DE-He213 Chronic Prostatitis (dpeaa)DE-He213 Benign Prostatic Hypertrophy (dpeaa)DE-He213 Chronic Pelvic Pain Syndrome (dpeaa)DE-He213 Express Prostatic Secretion (dpeaa)DE-He213 Wilson, Christopher M. verfasserin aut Enthalten in Current prostate reports Philadelphia, Pa. : Current Science, 2003 3(2005), 2 vom: 01. Juni, Seite 81-86 (DE-627)51163806X (DE-600)2233535-3 1544-225X nnns volume:3 year:2005 number:2 day:01 month:06 pages:81-86 https://dx.doi.org/10.1007/s11918-005-0020-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_40 GBV_ILN_702 GBV_ILN_2190 AR 3 2005 2 01 06 81-86 |
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10.1007/s11918-005-0020-6 doi (DE-627)SPR023014776 (SPR)s11918-005-0020-6-e DE-627 ger DE-627 rakwb eng 610 ASE Sindhwani, Puneet verfasserin aut Prostatitis and serum prostate-specific antigen 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. Prostatitis (dpeaa)DE-He213 Chronic Prostatitis (dpeaa)DE-He213 Benign Prostatic Hypertrophy (dpeaa)DE-He213 Chronic Pelvic Pain Syndrome (dpeaa)DE-He213 Express Prostatic Secretion (dpeaa)DE-He213 Wilson, Christopher M. verfasserin aut Enthalten in Current prostate reports Philadelphia, Pa. : Current Science, 2003 3(2005), 2 vom: 01. Juni, Seite 81-86 (DE-627)51163806X (DE-600)2233535-3 1544-225X nnns volume:3 year:2005 number:2 day:01 month:06 pages:81-86 https://dx.doi.org/10.1007/s11918-005-0020-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_40 GBV_ILN_702 GBV_ILN_2190 AR 3 2005 2 01 06 81-86 |
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10.1007/s11918-005-0020-6 doi (DE-627)SPR023014776 (SPR)s11918-005-0020-6-e DE-627 ger DE-627 rakwb eng 610 ASE Sindhwani, Puneet verfasserin aut Prostatitis and serum prostate-specific antigen 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. Prostatitis (dpeaa)DE-He213 Chronic Prostatitis (dpeaa)DE-He213 Benign Prostatic Hypertrophy (dpeaa)DE-He213 Chronic Pelvic Pain Syndrome (dpeaa)DE-He213 Express Prostatic Secretion (dpeaa)DE-He213 Wilson, Christopher M. verfasserin aut Enthalten in Current prostate reports Philadelphia, Pa. : Current Science, 2003 3(2005), 2 vom: 01. Juni, Seite 81-86 (DE-627)51163806X (DE-600)2233535-3 1544-225X nnns volume:3 year:2005 number:2 day:01 month:06 pages:81-86 https://dx.doi.org/10.1007/s11918-005-0020-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_40 GBV_ILN_702 GBV_ILN_2190 AR 3 2005 2 01 06 81-86 |
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10.1007/s11918-005-0020-6 doi (DE-627)SPR023014776 (SPR)s11918-005-0020-6-e DE-627 ger DE-627 rakwb eng 610 ASE Sindhwani, Puneet verfasserin aut Prostatitis and serum prostate-specific antigen 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. Prostatitis (dpeaa)DE-He213 Chronic Prostatitis (dpeaa)DE-He213 Benign Prostatic Hypertrophy (dpeaa)DE-He213 Chronic Pelvic Pain Syndrome (dpeaa)DE-He213 Express Prostatic Secretion (dpeaa)DE-He213 Wilson, Christopher M. verfasserin aut Enthalten in Current prostate reports Philadelphia, Pa. : Current Science, 2003 3(2005), 2 vom: 01. Juni, Seite 81-86 (DE-627)51163806X (DE-600)2233535-3 1544-225X nnns volume:3 year:2005 number:2 day:01 month:06 pages:81-86 https://dx.doi.org/10.1007/s11918-005-0020-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_40 GBV_ILN_702 GBV_ILN_2190 AR 3 2005 2 01 06 81-86 |
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10.1007/s11918-005-0020-6 doi (DE-627)SPR023014776 (SPR)s11918-005-0020-6-e DE-627 ger DE-627 rakwb eng 610 ASE Sindhwani, Puneet verfasserin aut Prostatitis and serum prostate-specific antigen 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. Prostatitis (dpeaa)DE-He213 Chronic Prostatitis (dpeaa)DE-He213 Benign Prostatic Hypertrophy (dpeaa)DE-He213 Chronic Pelvic Pain Syndrome (dpeaa)DE-He213 Express Prostatic Secretion (dpeaa)DE-He213 Wilson, Christopher M. verfasserin aut Enthalten in Current prostate reports Philadelphia, Pa. : Current Science, 2003 3(2005), 2 vom: 01. Juni, Seite 81-86 (DE-627)51163806X (DE-600)2233535-3 1544-225X nnns volume:3 year:2005 number:2 day:01 month:06 pages:81-86 https://dx.doi.org/10.1007/s11918-005-0020-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_40 GBV_ILN_702 GBV_ILN_2190 AR 3 2005 2 01 06 81-86 |
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Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. |
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Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. |
abstract_unstemmed |
Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. Inflammation has been proposed to be a precursor of prostate adenocarcinoma. |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR023014776</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230520013016.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2005 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11918-005-0020-6</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR023014776</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11918-005-0020-6-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="q">ASE</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Sindhwani, Puneet</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Prostatitis and serum prostate-specific antigen</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2005</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Abstract Prostatitis is an inflammatory condition of the prostate and has been divided into four categories according to the National Institutes of Health classification. This article reviews the various types of prostatitis and their effect on serum prostate-specific antigen levels. Various proposed mechanisms of this elevation include leakage of prostatespecific antigen (PSA) into the blood stream, hypervascularity, and altered vascular permeability secondary to inflammation. Acute prostatitis can lead to an increase in PSA, which usually returns to normal levels with appropriate antibiotics within 1 to 3 months. Patients with chronic prostatitis have a less well-defined decrease in PSA after an antibiotic course. Whether a course of antibiotics prior to biopsy increases the yield has not been well established. Asymptomatic inflammation of the prostate has been recognized to be an important confounding factor in patients with an elevated PSA. 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