Impact of Genetics on the Diagnosis and Treatment of Renal Cancer
Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary pap...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Singer, Eric A. [verfasserIn] Bratslavsky, Gennady [verfasserIn] Middelton, Lindsay [verfasserIn] Srinivasan, Ramaprasad [verfasserIn] Linehan, W. Marston [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2010 |
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| Schlagwörter: |
Hereditary papillary renal carcinoma |
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| Übergeordnetes Werk: |
Enthalten in: Current urology reports - Philadelphia, Pa. : Current Science Inc., 2000, 12(2010), 1 vom: 03. Dez., Seite 47-55 |
|---|---|
| Übergeordnetes Werk: |
volume:12 ; year:2010 ; number:1 ; day:03 ; month:12 ; pages:47-55 |
| Links: |
|---|
| DOI / URN: |
10.1007/s11934-010-0156-y |
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| Katalog-ID: |
SPR023048662 |
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| 520 | |a Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. | ||
| 650 | 4 | |a Kidney cancer |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Genetics |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a von Hippel-Lindau |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a VHL |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Hereditary papillary renal carcinoma |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a HPRC |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Hereditary leiomyomatosis and renal cell carcinoma |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a HLRCC |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Birt-Hogg-Dube’ |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a BHD |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Clear cell |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Papillary |7 (dpeaa)DE-He213 | |
| 650 | 4 | |a Chromophobe |7 (dpeaa)DE-He213 | |
| 700 | 1 | |a Bratslavsky, Gennady |e verfasserin |4 aut | |
| 700 | 1 | |a Middelton, Lindsay |e verfasserin |4 aut | |
| 700 | 1 | |a Srinivasan, Ramaprasad |e verfasserin |4 aut | |
| 700 | 1 | |a Linehan, W. Marston |e verfasserin |4 aut | |
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10.1007/s11934-010-0156-y doi (DE-627)SPR023048662 (SPR)s11934-010-0156-y-e DE-627 ger DE-627 rakwb eng 610 ASE 44.88 bkl Singer, Eric A. verfasserin aut Impact of Genetics on the Diagnosis and Treatment of Renal Cancer 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 Bratslavsky, Gennady verfasserin aut Middelton, Lindsay verfasserin aut Srinivasan, Ramaprasad verfasserin aut Linehan, W. Marston verfasserin aut Enthalten in Current urology reports Philadelphia, Pa. : Current Science Inc., 2000 12(2010), 1 vom: 03. Dez., Seite 47-55 (DE-627)357168496 (DE-600)2094190-0 1534-6285 nnns volume:12 year:2010 number:1 day:03 month:12 pages:47-55 https://dx.doi.org/10.1007/s11934-010-0156-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.88 ASE AR 12 2010 1 03 12 47-55 |
| spelling |
10.1007/s11934-010-0156-y doi (DE-627)SPR023048662 (SPR)s11934-010-0156-y-e DE-627 ger DE-627 rakwb eng 610 ASE 44.88 bkl Singer, Eric A. verfasserin aut Impact of Genetics on the Diagnosis and Treatment of Renal Cancer 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 Bratslavsky, Gennady verfasserin aut Middelton, Lindsay verfasserin aut Srinivasan, Ramaprasad verfasserin aut Linehan, W. Marston verfasserin aut Enthalten in Current urology reports Philadelphia, Pa. : Current Science Inc., 2000 12(2010), 1 vom: 03. Dez., Seite 47-55 (DE-627)357168496 (DE-600)2094190-0 1534-6285 nnns volume:12 year:2010 number:1 day:03 month:12 pages:47-55 https://dx.doi.org/10.1007/s11934-010-0156-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.88 ASE AR 12 2010 1 03 12 47-55 |
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10.1007/s11934-010-0156-y doi (DE-627)SPR023048662 (SPR)s11934-010-0156-y-e DE-627 ger DE-627 rakwb eng 610 ASE 44.88 bkl Singer, Eric A. verfasserin aut Impact of Genetics on the Diagnosis and Treatment of Renal Cancer 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 Bratslavsky, Gennady verfasserin aut Middelton, Lindsay verfasserin aut Srinivasan, Ramaprasad verfasserin aut Linehan, W. Marston verfasserin aut Enthalten in Current urology reports Philadelphia, Pa. : Current Science Inc., 2000 12(2010), 1 vom: 03. Dez., Seite 47-55 (DE-627)357168496 (DE-600)2094190-0 1534-6285 nnns volume:12 year:2010 number:1 day:03 month:12 pages:47-55 https://dx.doi.org/10.1007/s11934-010-0156-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.88 ASE AR 12 2010 1 03 12 47-55 |
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10.1007/s11934-010-0156-y doi (DE-627)SPR023048662 (SPR)s11934-010-0156-y-e DE-627 ger DE-627 rakwb eng 610 ASE 44.88 bkl Singer, Eric A. verfasserin aut Impact of Genetics on the Diagnosis and Treatment of Renal Cancer 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 Bratslavsky, Gennady verfasserin aut Middelton, Lindsay verfasserin aut Srinivasan, Ramaprasad verfasserin aut Linehan, W. Marston verfasserin aut Enthalten in Current urology reports Philadelphia, Pa. : Current Science Inc., 2000 12(2010), 1 vom: 03. Dez., Seite 47-55 (DE-627)357168496 (DE-600)2094190-0 1534-6285 nnns volume:12 year:2010 number:1 day:03 month:12 pages:47-55 https://dx.doi.org/10.1007/s11934-010-0156-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.88 ASE AR 12 2010 1 03 12 47-55 |
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10.1007/s11934-010-0156-y doi (DE-627)SPR023048662 (SPR)s11934-010-0156-y-e DE-627 ger DE-627 rakwb eng 610 ASE 44.88 bkl Singer, Eric A. verfasserin aut Impact of Genetics on the Diagnosis and Treatment of Renal Cancer 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 Bratslavsky, Gennady verfasserin aut Middelton, Lindsay verfasserin aut Srinivasan, Ramaprasad verfasserin aut Linehan, W. Marston verfasserin aut Enthalten in Current urology reports Philadelphia, Pa. : Current Science Inc., 2000 12(2010), 1 vom: 03. Dez., Seite 47-55 (DE-627)357168496 (DE-600)2094190-0 1534-6285 nnns volume:12 year:2010 number:1 day:03 month:12 pages:47-55 https://dx.doi.org/10.1007/s11934-010-0156-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 44.88 ASE AR 12 2010 1 03 12 47-55 |
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Enthalten in Current urology reports 12(2010), 1 vom: 03. Dez., Seite 47-55 volume:12 year:2010 number:1 day:03 month:12 pages:47-55 |
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Kidney cancer Genetics von Hippel-Lindau VHL Hereditary papillary renal carcinoma HPRC Hereditary leiomyomatosis and renal cell carcinoma HLRCC Birt-Hogg-Dube’ BHD Clear cell Papillary Chromophobe |
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Singer, Eric A. @@aut@@ Bratslavsky, Gennady @@aut@@ Middelton, Lindsay @@aut@@ Srinivasan, Ramaprasad @@aut@@ Linehan, W. Marston @@aut@@ |
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Singer, Eric A. |
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Singer, Eric A. ddc 610 bkl 44.88 misc Kidney cancer misc Genetics misc von Hippel-Lindau misc VHL misc Hereditary papillary renal carcinoma misc HPRC misc Hereditary leiomyomatosis and renal cell carcinoma misc HLRCC misc Birt-Hogg-Dube’ misc BHD misc Clear cell misc Papillary misc Chromophobe Impact of Genetics on the Diagnosis and Treatment of Renal Cancer |
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610 ASE 44.88 bkl Impact of Genetics on the Diagnosis and Treatment of Renal Cancer Kidney cancer (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 von Hippel-Lindau (dpeaa)DE-He213 VHL (dpeaa)DE-He213 Hereditary papillary renal carcinoma (dpeaa)DE-He213 HPRC (dpeaa)DE-He213 Hereditary leiomyomatosis and renal cell carcinoma (dpeaa)DE-He213 HLRCC (dpeaa)DE-He213 Birt-Hogg-Dube’ (dpeaa)DE-He213 BHD (dpeaa)DE-He213 Clear cell (dpeaa)DE-He213 Papillary (dpeaa)DE-He213 Chromophobe (dpeaa)DE-He213 |
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ddc 610 bkl 44.88 misc Kidney cancer misc Genetics misc von Hippel-Lindau misc VHL misc Hereditary papillary renal carcinoma misc HPRC misc Hereditary leiomyomatosis and renal cell carcinoma misc HLRCC misc Birt-Hogg-Dube’ misc BHD misc Clear cell misc Papillary misc Chromophobe |
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ddc 610 bkl 44.88 misc Kidney cancer misc Genetics misc von Hippel-Lindau misc VHL misc Hereditary papillary renal carcinoma misc HPRC misc Hereditary leiomyomatosis and renal cell carcinoma misc HLRCC misc Birt-Hogg-Dube’ misc BHD misc Clear cell misc Papillary misc Chromophobe |
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ddc 610 bkl 44.88 misc Kidney cancer misc Genetics misc von Hippel-Lindau misc VHL misc Hereditary papillary renal carcinoma misc HPRC misc Hereditary leiomyomatosis and renal cell carcinoma misc HLRCC misc Birt-Hogg-Dube’ misc BHD misc Clear cell misc Papillary misc Chromophobe |
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Impact of Genetics on the Diagnosis and Treatment of Renal Cancer |
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impact of genetics on the diagnosis and treatment of renal cancer |
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Impact of Genetics on the Diagnosis and Treatment of Renal Cancer |
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Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. |
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Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. |
| abstract_unstemmed |
Abstract Kidney cancer is a heterogeneous disease comprised of a number of histologic subtypes, each associated with unique genetic mutations, clinical features, and sensitivity to treatment. By examining families affected with the hereditary kidney cancer syndromes von Hippel-Lindau, hereditary papillary renal cell carcinoma, hereditary leiomyomatosis and renal cell carcinoma, and Birt-Hogg-Dubé, researchers have been able to identify the genes responsible for these syndromes. This work has revealed that kidney cancer is fundamentally a metabolic disorder, and as such, novel targeted therapies specific to their molecular biology have been developed and employed in both the hereditary and sporadic forms of renal cell carcinoma. |
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| score |
7.3974047 |