The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap

Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Matsui, Yoshio [verfasserIn] Kadoya, Yoshinori [verfasserIn] Horibe, Shuji [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2012

Schlagwörter:	Medial Collateral Ligament Lateral Median Normal Knee Medial Median Injured Knee

Übergeordnetes Werk:	Enthalten in: Clinical orthopaedics and related research - Philadelphia, PA : Wolters Kluwer Health, 1963, 471(2012), 4 vom: 21. Nov., Seite 1299-1304
Übergeordnetes Werk:	volume:471 ; year:2012 ; number:4 ; day:21 ; month:11 ; pages:1299-1304

Links:	Volltext

DOI / URN:	10.1007/s11999-012-2707-7

Katalog-ID:	SPR023694963

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520			\|a Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.
650		4	\|a Medial Collateral Ligament \|7 (dpeaa)DE-He213
650		4	\|a Lateral Median \|7 (dpeaa)DE-He213
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700	1		\|a Kadoya, Yoshinori \|e verfasserin \|4 aut
700	1		\|a Horibe, Shuji \|e verfasserin \|4 aut
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allfields	10.1007/s11999-012-2707-7 doi (DE-627)SPR023694963 (SPR)s11999-012-2707-7-e DE-627 ger DE-627 rakwb eng 610 ASE 44.83 bkl Matsui, Yoshio verfasserin aut The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap 2012 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap. Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213 Kadoya, Yoshinori verfasserin aut Horibe, Shuji verfasserin aut Enthalten in Clinical orthopaedics and related research Philadelphia, PA : Wolters Kluwer Health, 1963 471(2012), 4 vom: 21. Nov., Seite 1299-1304 (DE-627)316019062 (DE-600)2018318-5 1528-1132 nnns volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304 https://dx.doi.org/10.1007/s11999-012-2707-7 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 44.83 ASE AR 471 2012 4 21 11 1299-1304
spelling	10.1007/s11999-012-2707-7 doi (DE-627)SPR023694963 (SPR)s11999-012-2707-7-e DE-627 ger DE-627 rakwb eng 610 ASE 44.83 bkl Matsui, Yoshio verfasserin aut The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap 2012 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap. Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213 Kadoya, Yoshinori verfasserin aut Horibe, Shuji verfasserin aut Enthalten in Clinical orthopaedics and related research Philadelphia, PA : Wolters Kluwer Health, 1963 471(2012), 4 vom: 21. Nov., Seite 1299-1304 (DE-627)316019062 (DE-600)2018318-5 1528-1132 nnns volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304 https://dx.doi.org/10.1007/s11999-012-2707-7 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 44.83 ASE AR 471 2012 4 21 11 1299-1304
allfields_unstemmed	10.1007/s11999-012-2707-7 doi (DE-627)SPR023694963 (SPR)s11999-012-2707-7-e DE-627 ger DE-627 rakwb eng 610 ASE 44.83 bkl Matsui, Yoshio verfasserin aut The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap 2012 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap. Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213 Kadoya, Yoshinori verfasserin aut Horibe, Shuji verfasserin aut Enthalten in Clinical orthopaedics and related research Philadelphia, PA : Wolters Kluwer Health, 1963 471(2012), 4 vom: 21. Nov., Seite 1299-1304 (DE-627)316019062 (DE-600)2018318-5 1528-1132 nnns volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304 https://dx.doi.org/10.1007/s11999-012-2707-7 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 44.83 ASE AR 471 2012 4 21 11 1299-1304
allfieldsGer	10.1007/s11999-012-2707-7 doi (DE-627)SPR023694963 (SPR)s11999-012-2707-7-e DE-627 ger DE-627 rakwb eng 610 ASE 44.83 bkl Matsui, Yoshio verfasserin aut The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap 2012 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap. Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213 Kadoya, Yoshinori verfasserin aut Horibe, Shuji verfasserin aut Enthalten in Clinical orthopaedics and related research Philadelphia, PA : Wolters Kluwer Health, 1963 471(2012), 4 vom: 21. Nov., Seite 1299-1304 (DE-627)316019062 (DE-600)2018318-5 1528-1132 nnns volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304 https://dx.doi.org/10.1007/s11999-012-2707-7 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 44.83 ASE AR 471 2012 4 21 11 1299-1304
allfieldsSound	10.1007/s11999-012-2707-7 doi (DE-627)SPR023694963 (SPR)s11999-012-2707-7-e DE-627 ger DE-627 rakwb eng 610 ASE 44.83 bkl Matsui, Yoshio verfasserin aut The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap 2012 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap. Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213 Kadoya, Yoshinori verfasserin aut Horibe, Shuji verfasserin aut Enthalten in Clinical orthopaedics and related research Philadelphia, PA : Wolters Kluwer Health, 1963 471(2012), 4 vom: 21. Nov., Seite 1299-1304 (DE-627)316019062 (DE-600)2018318-5 1528-1132 nnns volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304 https://dx.doi.org/10.1007/s11999-012-2707-7 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 44.83 ASE AR 471 2012 4 21 11 1299-1304
language	English
source	Enthalten in Clinical orthopaedics and related research 471(2012), 4 vom: 21. Nov., Seite 1299-1304 volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304
sourceStr	Enthalten in Clinical orthopaedics and related research 471(2012), 4 vom: 21. Nov., Seite 1299-1304 volume:471 year:2012 number:4 day:21 month:11 pages:1299-1304
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	Medial Collateral Ligament Lateral Median Normal Knee Medial Median Injured Knee
dewey-raw	610
isfreeaccess_bool	false
container_title	Clinical orthopaedics and related research
authorswithroles_txt_mv	Matsui, Yoshio @@aut@@ Kadoya, Yoshinori @@aut@@ Horibe, Shuji @@aut@@
publishDateDaySort_date	2012-11-21T00:00:00Z
hierarchy_top_id	316019062
dewey-sort	3610
id	SPR023694963
language_de	englisch
fullrecord	<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR023694963</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230519183450.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2012 xx \|\|\|\|\|o 00\| \|\|eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11999-012-2707-7</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR023694963</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11999-012-2707-7-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="q">ASE</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">44.83</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Matsui, Yoshio</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="4"><subfield code="a">The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2012</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Medial Collateral Ligament</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Lateral Median</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Normal Knee</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Medial Median</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Injured Knee</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Kadoya, Yoshinori</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Horibe, Shuji</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Clinical orthopaedics and related research</subfield><subfield code="d">Philadelphia, PA : Wolters Kluwer Health, 1963</subfield><subfield code="g">471(2012), 4 vom: 21. 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author	Matsui, Yoshio
spellingShingle	Matsui, Yoshio ddc 610 bkl 44.83 misc Medial Collateral Ligament misc Lateral Median misc Normal Knee misc Medial Median misc Injured Knee The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap
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topic_title	610 ASE 44.83 bkl The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap Medial Collateral Ligament (dpeaa)DE-He213 Lateral Median (dpeaa)DE-He213 Normal Knee (dpeaa)DE-He213 Medial Median (dpeaa)DE-He213 Injured Knee (dpeaa)DE-He213
topic	ddc 610 bkl 44.83 misc Medial Collateral Ligament misc Lateral Median misc Normal Knee misc Medial Median misc Injured Knee
topic_unstemmed	ddc 610 bkl 44.83 misc Medial Collateral Ligament misc Lateral Median misc Normal Knee misc Medial Median misc Injured Knee
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title	The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap
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title_full	The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap
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journal	Clinical orthopaedics and related research
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author_browse	Matsui, Yoshio Kadoya, Yoshinori Horibe, Shuji
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title_sort	intact posterior cruciate ligament not only controls posterior displacement but also maintains the flexion gap
title_auth	The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap
abstract	Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.
abstractGer	Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.
abstract_unstemmed	Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.
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title_short	The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap
url	https://dx.doi.org/10.1007/s11999-012-2707-7
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up_date	2024-07-03T20:42:02.930Z
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fullrecord_marcxml	<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR023694963</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230519183450.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2012 xx \|\|\|\|\|o 00\| \|\|eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11999-012-2707-7</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR023694963</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11999-012-2707-7-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="082" ind1="0" ind2="4"><subfield code="a">610</subfield><subfield code="q">ASE</subfield></datafield><datafield tag="084" ind1=" " ind2=" "><subfield code="a">44.83</subfield><subfield code="2">bkl</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Matsui, Yoshio</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="4"><subfield code="a">The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2012</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Background The PCL is a strong stabilizer of the knee and provides posterior stability to the tibia. However, sagittal alignment of the PCL with the knee at 90° flexion suggests the PCL might play a role not only in posterior stabilization but also in maintaining the flexion gap. Questions/purposes We determined whether the intact PCL helps maintain the flexion gap. Methods We examined axial radiographs and gravity sag views of 17 patients with chronic isolated unilateral PCL injury. The flexion gap was defined as the mean value of the medial and lateral distances between the femoral and tibial bones on the axial radiograph. Increase in the flexion gap and posterior laxity were determined by comparing the patients’ injured and contralateral uninjured knees. Results The flexion gap of PCL injured knees (median, 7.5 mm; range, 5.3–11.5 mm; medial median, 6.2 mm; medial range, 3.7–8.3 mm; lateral median, 7.9 mm; lateral range, 5.3–11.5 mm) was larger than that seen in uninjured knees (median, 5.0 mm; range, 4.0–7.6 mm; medial median, 4.6 mm; medial range 3.4–7.1 mm; lateral median, 5.6; lateral range, 4.5–11.2 mm). The increment in the medial distance was similar to that in the lateral distance. Posterior laxity of injured knees was 9.1 (median); 5.4 to 15.2 (range) mm greater than that of uninjured knees. We found no correlation between posterior laxity and the flexion gap increment. Conclusions Our data suggest the intact PCL controls posterior displacement and maintains the flexion gap.</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Medial Collateral Ligament</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Lateral Median</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Normal Knee</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Medial Median</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Injured Knee</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Kadoya, Yoshinori</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Horibe, Shuji</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Clinical orthopaedics and related research</subfield><subfield code="d">Philadelphia, PA : Wolters Kluwer Health, 1963</subfield><subfield code="g">471(2012), 4 vom: 21. 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The Intact Posterior Cruciate Ligament Not Only Controls Posterior Displacement but Also Maintains the Flexion Gap

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