Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?

Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Gagliardi, Christian [verfasserIn] Tabacchi, Elena Bonfiglioli, Rachele Diodato, Stefania Nanni, Cristina Guidalotti, Pierluigi Lorenzini, Massimiliano Lodi, Filippo Milandri, Agnese Rapezzi, Claudio Fanti, Stefano

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2016

Schlagwörter:	[18F]-NaF PET/CT cardiac amyloidosis cardiomyopathy PET/CT imaging

Anmerkung:	© American Society of Nuclear Cardiology 2016

Übergeordnetes Werk:	Enthalten in: Journal of nuclear cardiology - New York, NY : Springer, 1994, 24(2016), 2 vom: 14. März, Seite 746-749
Übergeordnetes Werk:	volume:24 ; year:2016 ; number:2 ; day:14 ; month:03 ; pages:746-749

Links:	Volltext

DOI / URN:	10.1007/s12350-016-0457-8

Katalog-ID:	SPR024982911

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520			\|a Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA.
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912			\|a GBV_ILN_213
912			\|a GBV_ILN_224
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allfields	10.1007/s12350-016-0457-8 doi (DE-627)SPR024982911 (SPR)s12350-016-0457-8-e DE-627 ger DE-627 rakwb eng Gagliardi, Christian verfasserin aut Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © American Society of Nuclear Cardiology 2016 Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213 Tabacchi, Elena aut Bonfiglioli, Rachele aut Diodato, Stefania aut Nanni, Cristina aut Guidalotti, Pierluigi aut Lorenzini, Massimiliano aut Lodi, Filippo aut Milandri, Agnese aut Rapezzi, Claudio aut Fanti, Stefano aut Enthalten in Journal of nuclear cardiology New York, NY : Springer, 1994 24(2016), 2 vom: 14. März, Seite 746-749 (DE-627)329395173 (DE-600)2048325-9 1532-6551 nnns volume:24 year:2016 number:2 day:14 month:03 pages:746-749 https://dx.doi.org/10.1007/s12350-016-0457-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 24 2016 2 14 03 746-749
spelling	10.1007/s12350-016-0457-8 doi (DE-627)SPR024982911 (SPR)s12350-016-0457-8-e DE-627 ger DE-627 rakwb eng Gagliardi, Christian verfasserin aut Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © American Society of Nuclear Cardiology 2016 Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213 Tabacchi, Elena aut Bonfiglioli, Rachele aut Diodato, Stefania aut Nanni, Cristina aut Guidalotti, Pierluigi aut Lorenzini, Massimiliano aut Lodi, Filippo aut Milandri, Agnese aut Rapezzi, Claudio aut Fanti, Stefano aut Enthalten in Journal of nuclear cardiology New York, NY : Springer, 1994 24(2016), 2 vom: 14. März, Seite 746-749 (DE-627)329395173 (DE-600)2048325-9 1532-6551 nnns volume:24 year:2016 number:2 day:14 month:03 pages:746-749 https://dx.doi.org/10.1007/s12350-016-0457-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 24 2016 2 14 03 746-749
allfields_unstemmed	10.1007/s12350-016-0457-8 doi (DE-627)SPR024982911 (SPR)s12350-016-0457-8-e DE-627 ger DE-627 rakwb eng Gagliardi, Christian verfasserin aut Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © American Society of Nuclear Cardiology 2016 Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213 Tabacchi, Elena aut Bonfiglioli, Rachele aut Diodato, Stefania aut Nanni, Cristina aut Guidalotti, Pierluigi aut Lorenzini, Massimiliano aut Lodi, Filippo aut Milandri, Agnese aut Rapezzi, Claudio aut Fanti, Stefano aut Enthalten in Journal of nuclear cardiology New York, NY : Springer, 1994 24(2016), 2 vom: 14. März, Seite 746-749 (DE-627)329395173 (DE-600)2048325-9 1532-6551 nnns volume:24 year:2016 number:2 day:14 month:03 pages:746-749 https://dx.doi.org/10.1007/s12350-016-0457-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 24 2016 2 14 03 746-749
allfieldsGer	10.1007/s12350-016-0457-8 doi (DE-627)SPR024982911 (SPR)s12350-016-0457-8-e DE-627 ger DE-627 rakwb eng Gagliardi, Christian verfasserin aut Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © American Society of Nuclear Cardiology 2016 Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213 Tabacchi, Elena aut Bonfiglioli, Rachele aut Diodato, Stefania aut Nanni, Cristina aut Guidalotti, Pierluigi aut Lorenzini, Massimiliano aut Lodi, Filippo aut Milandri, Agnese aut Rapezzi, Claudio aut Fanti, Stefano aut Enthalten in Journal of nuclear cardiology New York, NY : Springer, 1994 24(2016), 2 vom: 14. März, Seite 746-749 (DE-627)329395173 (DE-600)2048325-9 1532-6551 nnns volume:24 year:2016 number:2 day:14 month:03 pages:746-749 https://dx.doi.org/10.1007/s12350-016-0457-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 24 2016 2 14 03 746-749
allfieldsSound	10.1007/s12350-016-0457-8 doi (DE-627)SPR024982911 (SPR)s12350-016-0457-8-e DE-627 ger DE-627 rakwb eng Gagliardi, Christian verfasserin aut Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? 2016 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © American Society of Nuclear Cardiology 2016 Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213 Tabacchi, Elena aut Bonfiglioli, Rachele aut Diodato, Stefania aut Nanni, Cristina aut Guidalotti, Pierluigi aut Lorenzini, Massimiliano aut Lodi, Filippo aut Milandri, Agnese aut Rapezzi, Claudio aut Fanti, Stefano aut Enthalten in Journal of nuclear cardiology New York, NY : Springer, 1994 24(2016), 2 vom: 14. März, Seite 746-749 (DE-627)329395173 (DE-600)2048325-9 1532-6551 nnns volume:24 year:2016 number:2 day:14 month:03 pages:746-749 https://dx.doi.org/10.1007/s12350-016-0457-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 24 2016 2 14 03 746-749
language	English
source	Enthalten in Journal of nuclear cardiology 24(2016), 2 vom: 14. März, Seite 746-749 volume:24 year:2016 number:2 day:14 month:03 pages:746-749
sourceStr	Enthalten in Journal of nuclear cardiology 24(2016), 2 vom: 14. März, Seite 746-749 volume:24 year:2016 number:2 day:14 month:03 pages:746-749
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	[18F]-NaF PET/CT cardiac amyloidosis cardiomyopathy PET/CT imaging
isfreeaccess_bool	false
container_title	Journal of nuclear cardiology
authorswithroles_txt_mv	Gagliardi, Christian @@aut@@ Tabacchi, Elena @@aut@@ Bonfiglioli, Rachele @@aut@@ Diodato, Stefania @@aut@@ Nanni, Cristina @@aut@@ Guidalotti, Pierluigi @@aut@@ Lorenzini, Massimiliano @@aut@@ Lodi, Filippo @@aut@@ Milandri, Agnese @@aut@@ Rapezzi, Claudio @@aut@@ Fanti, Stefano @@aut@@
publishDateDaySort_date	2016-03-14T00:00:00Z
hierarchy_top_id	329395173
id	SPR024982911
language_de	englisch
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author	Gagliardi, Christian
spellingShingle	Gagliardi, Christian misc [18F]-NaF PET/CT misc cardiac amyloidosis misc cardiomyopathy misc PET/CT imaging Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?
authorStr	Gagliardi, Christian
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topic_title	Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT? [18F]-NaF PET/CT (dpeaa)DE-He213 cardiac amyloidosis (dpeaa)DE-He213 cardiomyopathy (dpeaa)DE-He213 PET/CT imaging (dpeaa)DE-He213
topic	misc [18F]-NaF PET/CT misc cardiac amyloidosis misc cardiomyopathy misc PET/CT imaging
topic_unstemmed	misc [18F]-NaF PET/CT misc cardiac amyloidosis misc cardiomyopathy misc PET/CT imaging
topic_browse	misc [18F]-NaF PET/CT misc cardiac amyloidosis misc cardiomyopathy misc PET/CT imaging
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title	Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?
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title_full	Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?
author_sort	Gagliardi, Christian
journal	Journal of nuclear cardiology
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author_browse	Gagliardi, Christian Tabacchi, Elena Bonfiglioli, Rachele Diodato, Stefania Nanni, Cristina Guidalotti, Pierluigi Lorenzini, Massimiliano Lodi, Filippo Milandri, Agnese Rapezzi, Claudio Fanti, Stefano
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author-letter	Gagliardi, Christian
doi_str_mv	10.1007/s12350-016-0457-8
title_sort	does the etiology of cardiac amyloidosis determine the myocardial uptake of [18f]-naf pet/ct?
title_auth	Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?
abstract	Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. © American Society of Nuclear Cardiology 2016
abstractGer	Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. © American Society of Nuclear Cardiology 2016
abstract_unstemmed	Abstract Cardiac amyloidosis (CA) leads to variable degrees of myocardial infiltration with a final echocardiographic phenotype of “hypertrophy.” Although many non-invasive imaging techniques (MRI, CT, scintigraphy, PET) are useful, the definitive diagnosis is still based on myocardial histology. We explored the possible role of [18F]-NaF PET/CT in the diagnosis of this disease in two cases with wild-type (ATTRwt) or mutant (ATTRm) Ile68Leu transthyretin (TTR)-related CA. © American Society of Nuclear Cardiology 2016
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container_issue	2
title_short	Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?
url	https://dx.doi.org/10.1007/s12350-016-0457-8
remote_bool	true
author2	Tabacchi, Elena Bonfiglioli, Rachele Diodato, Stefania Nanni, Cristina Guidalotti, Pierluigi Lorenzini, Massimiliano Lodi, Filippo Milandri, Agnese Rapezzi, Claudio Fanti, Stefano
author2Str	Tabacchi, Elena Bonfiglioli, Rachele Diodato, Stefania Nanni, Cristina Guidalotti, Pierluigi Lorenzini, Massimiliano Lodi, Filippo Milandri, Agnese Rapezzi, Claudio Fanti, Stefano
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doi_str	10.1007/s12350-016-0457-8
up_date	2024-07-04T03:04:20.687Z
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Does the etiology of cardiac amyloidosis determine the myocardial uptake of [18F]-NaF PET/CT?

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