Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD

Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not sign...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Arcos-Burgos, Mauricio [verfasserIn] Muenke, Maximilian [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2010

Schlagwörter:	ADHD Complex trait Gene Genetics LPHN3 Latrophilin

Übergeordnetes Werk:	Enthalten in: ADHD Attention Deficit and Hyperactivity Disorders - Wien [u.a.] : Springer, 2009, 2(2010), 3 vom: 16. Okt., Seite 139-147
Übergeordnetes Werk:	volume:2 ; year:2010 ; number:3 ; day:16 ; month:10 ; pages:139-147

Links:	Volltext

DOI / URN:	10.1007/s12402-010-0030-2

Katalog-ID:	SPR025874284

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allfields	10.1007/s12402-010-0030-2 doi (DE-627)SPR025874284 (SPR)s12402-010-0030-2-e DE-627 ger DE-627 rakwb eng 610 ASE 44.91 bkl Arcos-Burgos, Mauricio verfasserin aut Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD. ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213 Muenke, Maximilian verfasserin aut Enthalten in ADHD Attention Deficit and Hyperactivity Disorders Wien [u.a.] : Springer, 2009 2(2010), 3 vom: 16. Okt., Seite 139-147 (DE-627)595709958 (DE-600)2486747-0 1866-6647 nnns volume:2 year:2010 number:3 day:16 month:10 pages:139-147 https://dx.doi.org/10.1007/s12402-010-0030-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_60 GBV_ILN_120 GBV_ILN_266 GBV_ILN_281 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4306 44.91 ASE AR 2 2010 3 16 10 139-147
spelling	10.1007/s12402-010-0030-2 doi (DE-627)SPR025874284 (SPR)s12402-010-0030-2-e DE-627 ger DE-627 rakwb eng 610 ASE 44.91 bkl Arcos-Burgos, Mauricio verfasserin aut Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD. ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213 Muenke, Maximilian verfasserin aut Enthalten in ADHD Attention Deficit and Hyperactivity Disorders Wien [u.a.] : Springer, 2009 2(2010), 3 vom: 16. Okt., Seite 139-147 (DE-627)595709958 (DE-600)2486747-0 1866-6647 nnns volume:2 year:2010 number:3 day:16 month:10 pages:139-147 https://dx.doi.org/10.1007/s12402-010-0030-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_60 GBV_ILN_120 GBV_ILN_266 GBV_ILN_281 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4306 44.91 ASE AR 2 2010 3 16 10 139-147
allfields_unstemmed	10.1007/s12402-010-0030-2 doi (DE-627)SPR025874284 (SPR)s12402-010-0030-2-e DE-627 ger DE-627 rakwb eng 610 ASE 44.91 bkl Arcos-Burgos, Mauricio verfasserin aut Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD. ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213 Muenke, Maximilian verfasserin aut Enthalten in ADHD Attention Deficit and Hyperactivity Disorders Wien [u.a.] : Springer, 2009 2(2010), 3 vom: 16. Okt., Seite 139-147 (DE-627)595709958 (DE-600)2486747-0 1866-6647 nnns volume:2 year:2010 number:3 day:16 month:10 pages:139-147 https://dx.doi.org/10.1007/s12402-010-0030-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_60 GBV_ILN_120 GBV_ILN_266 GBV_ILN_281 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4306 44.91 ASE AR 2 2010 3 16 10 139-147
allfieldsGer	10.1007/s12402-010-0030-2 doi (DE-627)SPR025874284 (SPR)s12402-010-0030-2-e DE-627 ger DE-627 rakwb eng 610 ASE 44.91 bkl Arcos-Burgos, Mauricio verfasserin aut Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD. ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213 Muenke, Maximilian verfasserin aut Enthalten in ADHD Attention Deficit and Hyperactivity Disorders Wien [u.a.] : Springer, 2009 2(2010), 3 vom: 16. Okt., Seite 139-147 (DE-627)595709958 (DE-600)2486747-0 1866-6647 nnns volume:2 year:2010 number:3 day:16 month:10 pages:139-147 https://dx.doi.org/10.1007/s12402-010-0030-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_60 GBV_ILN_120 GBV_ILN_266 GBV_ILN_281 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4306 44.91 ASE AR 2 2010 3 16 10 139-147
allfieldsSound	10.1007/s12402-010-0030-2 doi (DE-627)SPR025874284 (SPR)s12402-010-0030-2-e DE-627 ger DE-627 rakwb eng 610 ASE 44.91 bkl Arcos-Burgos, Mauricio verfasserin aut Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD. ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213 Muenke, Maximilian verfasserin aut Enthalten in ADHD Attention Deficit and Hyperactivity Disorders Wien [u.a.] : Springer, 2009 2(2010), 3 vom: 16. Okt., Seite 139-147 (DE-627)595709958 (DE-600)2486747-0 1866-6647 nnns volume:2 year:2010 number:3 day:16 month:10 pages:139-147 https://dx.doi.org/10.1007/s12402-010-0030-2 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_60 GBV_ILN_120 GBV_ILN_266 GBV_ILN_281 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2007 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2055 GBV_ILN_2059 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2106 GBV_ILN_2108 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4306 44.91 ASE AR 2 2010 3 16 10 139-147
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source	Enthalten in ADHD Attention Deficit and Hyperactivity Disorders 2(2010), 3 vom: 16. Okt., Seite 139-147 volume:2 year:2010 number:3 day:16 month:10 pages:139-147
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author	Arcos-Burgos, Mauricio
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topic_title	610 ASE 44.91 bkl Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD ADHD (dpeaa)DE-He213 Complex trait (dpeaa)DE-He213 Gene (dpeaa)DE-He213 Genetics (dpeaa)DE-He213 LPHN3 (dpeaa)DE-He213 Latrophilin (dpeaa)DE-He213
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title_full	Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD
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title_sort	toward a better understanding of adhd: lphn3 gene variants and the susceptibility to develop adhd
title_auth	Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD
abstract	Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD.
abstractGer	Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD.
abstract_unstemmed	Abstract During the past 15 years, an impressive amount of genetic information has become available in the research field of psychiatry, particularly as it relates to attention-deficit/hyperactivity disorder (ADHD). However, the classical clinical approach to ADHD has minimally affected and not significantly been improved by this genetic revolution. It is difficult to predict how long it will take for genetic findings to alter the way clinicians treat patients with ADHD. New medications or treatment protocols may take years to become routine clinical practice. However, when taken together, recent successes in genomics, pharmacogenomics, and genetic epidemiology have the potential (1) to prevent comorbid consequences of ADHD, (2) to individualize therapies for patients with ADHD, and (3) to define new epidemiological policies to aid with the impact of ADHD on society. Here, we present an overview of how genetic research may affect and improve the quality of life of patients with ADHD: as an example, we use the discovery of LPHN3, a new gene in which variants have recently been shown to be associated with ADHD.
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title_short	Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD
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Toward a better understanding of ADHD: LPHN3 gene variants and the susceptibility to develop ADHD

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