Chromatin, gene silencing and HIV latency
Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Mok, Hoi-Ping [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2007 |
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| Schlagwörter: |
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| Anmerkung: |
© BioMed Central Ltd 2007 |
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| Übergeordnetes Werk: |
Enthalten in: Genome biology - London : BioMed Central, 2000, 8(2007), 11 vom: 23. Nov. |
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| Übergeordnetes Werk: |
volume:8 ; year:2007 ; number:11 ; day:23 ; month:11 |
| Links: |
|---|
| DOI / URN: |
10.1186/gb-2007-8-11-228 |
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SPR029997690 |
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10.1186/gb-2007-8-11-228 doi (DE-627)SPR029997690 (SPR)gb-2007-8-11-228-e DE-627 ger DE-627 rakwb eng Mok, Hoi-Ping verfasserin aut Chromatin, gene silencing and HIV latency 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2007 Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. Murine Leukemia Virus (dpeaa)DE-He213 Bovine Leukemia Virus (dpeaa)DE-He213 Human Immunodeficiency Virus (dpeaa)DE-He213 Human Immunodeficiency Virus Infection (dpeaa)DE-He213 Proviral Expression (dpeaa)DE-He213 Lever, Andrew ML aut Enthalten in Genome biology London : BioMed Central, 2000 8(2007), 11 vom: 23. Nov. (DE-627)326173617 (DE-600)2040529-7 1474-760X nnns volume:8 year:2007 number:11 day:23 month:11 https://dx.doi.org/10.1186/gb-2007-8-11-228 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 8 2007 11 23 11 |
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10.1186/gb-2007-8-11-228 doi (DE-627)SPR029997690 (SPR)gb-2007-8-11-228-e DE-627 ger DE-627 rakwb eng Mok, Hoi-Ping verfasserin aut Chromatin, gene silencing and HIV latency 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2007 Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. Murine Leukemia Virus (dpeaa)DE-He213 Bovine Leukemia Virus (dpeaa)DE-He213 Human Immunodeficiency Virus (dpeaa)DE-He213 Human Immunodeficiency Virus Infection (dpeaa)DE-He213 Proviral Expression (dpeaa)DE-He213 Lever, Andrew ML aut Enthalten in Genome biology London : BioMed Central, 2000 8(2007), 11 vom: 23. Nov. (DE-627)326173617 (DE-600)2040529-7 1474-760X nnns volume:8 year:2007 number:11 day:23 month:11 https://dx.doi.org/10.1186/gb-2007-8-11-228 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 8 2007 11 23 11 |
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10.1186/gb-2007-8-11-228 doi (DE-627)SPR029997690 (SPR)gb-2007-8-11-228-e DE-627 ger DE-627 rakwb eng Mok, Hoi-Ping verfasserin aut Chromatin, gene silencing and HIV latency 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2007 Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. Murine Leukemia Virus (dpeaa)DE-He213 Bovine Leukemia Virus (dpeaa)DE-He213 Human Immunodeficiency Virus (dpeaa)DE-He213 Human Immunodeficiency Virus Infection (dpeaa)DE-He213 Proviral Expression (dpeaa)DE-He213 Lever, Andrew ML aut Enthalten in Genome biology London : BioMed Central, 2000 8(2007), 11 vom: 23. Nov. (DE-627)326173617 (DE-600)2040529-7 1474-760X nnns volume:8 year:2007 number:11 day:23 month:11 https://dx.doi.org/10.1186/gb-2007-8-11-228 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 8 2007 11 23 11 |
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10.1186/gb-2007-8-11-228 doi (DE-627)SPR029997690 (SPR)gb-2007-8-11-228-e DE-627 ger DE-627 rakwb eng Mok, Hoi-Ping verfasserin aut Chromatin, gene silencing and HIV latency 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2007 Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. Murine Leukemia Virus (dpeaa)DE-He213 Bovine Leukemia Virus (dpeaa)DE-He213 Human Immunodeficiency Virus (dpeaa)DE-He213 Human Immunodeficiency Virus Infection (dpeaa)DE-He213 Proviral Expression (dpeaa)DE-He213 Lever, Andrew ML aut Enthalten in Genome biology London : BioMed Central, 2000 8(2007), 11 vom: 23. Nov. (DE-627)326173617 (DE-600)2040529-7 1474-760X nnns volume:8 year:2007 number:11 day:23 month:11 https://dx.doi.org/10.1186/gb-2007-8-11-228 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 8 2007 11 23 11 |
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10.1186/gb-2007-8-11-228 doi (DE-627)SPR029997690 (SPR)gb-2007-8-11-228-e DE-627 ger DE-627 rakwb eng Mok, Hoi-Ping verfasserin aut Chromatin, gene silencing and HIV latency 2007 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2007 Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. Murine Leukemia Virus (dpeaa)DE-He213 Bovine Leukemia Virus (dpeaa)DE-He213 Human Immunodeficiency Virus (dpeaa)DE-He213 Human Immunodeficiency Virus Infection (dpeaa)DE-He213 Proviral Expression (dpeaa)DE-He213 Lever, Andrew ML aut Enthalten in Genome biology London : BioMed Central, 2000 8(2007), 11 vom: 23. Nov. (DE-627)326173617 (DE-600)2040529-7 1474-760X nnns volume:8 year:2007 number:11 day:23 month:11 https://dx.doi.org/10.1186/gb-2007-8-11-228 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 8 2007 11 23 11 |
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Enthalten in Genome biology 8(2007), 11 vom: 23. Nov. volume:8 year:2007 number:11 day:23 month:11 |
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Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. © BioMed Central Ltd 2007 |
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Abstract One of the cellular defenses against virus infection is the silencing of viral gene expression. There is evidence that at least two gene-silencing mechanisms are used against the human immuno-deficiency virus (HIV). Paradoxically, this cellular defense mechanism contributes to viral latency and persistence, and we review here the relationship of viral latency to gene-silencing mechanisms. © BioMed Central Ltd 2007 |
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