Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer
Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and...
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Gespeichert in:
| Autor*in: |
Jaffrey, Samie R. [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2017 |
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| Schlagwörter: |
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| Anmerkung: |
© The Author(s). 2017 |
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| Übergeordnetes Werk: |
Enthalten in: Genome medicine - London : BioMed Central, 2009, 9(2017), 1 vom: 12. Jan. |
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| Übergeordnetes Werk: |
volume:9 ; year:2017 ; number:1 ; day:12 ; month:01 |
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| DOI / URN: |
10.1186/s13073-016-0395-8 |
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10.1186/s13073-016-0395-8 doi (DE-627)SPR030650747 (SPR)s13073-016-0395-8-e DE-627 ger DE-627 rakwb eng Jaffrey, Samie R. verfasserin aut Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2017 Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 Kharas, Michael G. aut Enthalten in Genome medicine London : BioMed Central, 2009 9(2017), 1 vom: 12. Jan. (DE-627)594424275 (DE-600)2484394-5 1756-994X nnns volume:9 year:2017 number:1 day:12 month:01 https://dx.doi.org/10.1186/s13073-016-0395-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2017 1 12 01 |
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10.1186/s13073-016-0395-8 doi (DE-627)SPR030650747 (SPR)s13073-016-0395-8-e DE-627 ger DE-627 rakwb eng Jaffrey, Samie R. verfasserin aut Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2017 Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 Kharas, Michael G. aut Enthalten in Genome medicine London : BioMed Central, 2009 9(2017), 1 vom: 12. Jan. (DE-627)594424275 (DE-600)2484394-5 1756-994X nnns volume:9 year:2017 number:1 day:12 month:01 https://dx.doi.org/10.1186/s13073-016-0395-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2017 1 12 01 |
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10.1186/s13073-016-0395-8 doi (DE-627)SPR030650747 (SPR)s13073-016-0395-8-e DE-627 ger DE-627 rakwb eng Jaffrey, Samie R. verfasserin aut Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2017 Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 Kharas, Michael G. aut Enthalten in Genome medicine London : BioMed Central, 2009 9(2017), 1 vom: 12. Jan. (DE-627)594424275 (DE-600)2484394-5 1756-994X nnns volume:9 year:2017 number:1 day:12 month:01 https://dx.doi.org/10.1186/s13073-016-0395-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2017 1 12 01 |
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10.1186/s13073-016-0395-8 doi (DE-627)SPR030650747 (SPR)s13073-016-0395-8-e DE-627 ger DE-627 rakwb eng Jaffrey, Samie R. verfasserin aut Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2017 Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 Kharas, Michael G. aut Enthalten in Genome medicine London : BioMed Central, 2009 9(2017), 1 vom: 12. Jan. (DE-627)594424275 (DE-600)2484394-5 1756-994X nnns volume:9 year:2017 number:1 day:12 month:01 https://dx.doi.org/10.1186/s13073-016-0395-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2017 1 12 01 |
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10.1186/s13073-016-0395-8 doi (DE-627)SPR030650747 (SPR)s13073-016-0395-8-e DE-627 ger DE-627 rakwb eng Jaffrey, Samie R. verfasserin aut Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2017 Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 Kharas, Michael G. aut Enthalten in Genome medicine London : BioMed Central, 2009 9(2017), 1 vom: 12. Jan. (DE-627)594424275 (DE-600)2484394-5 1756-994X nnns volume:9 year:2017 number:1 day:12 month:01 https://dx.doi.org/10.1186/s13073-016-0395-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 9 2017 1 12 01 |
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Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer Myeloid Leukemia (dpeaa)DE-He213 Cancer Stem Cell (dpeaa)DE-He213 Breast Cancer Stem Cell (dpeaa)DE-He213 Cancer Stem Cell Phenotype (dpeaa)DE-He213 Methylation Complex (dpeaa)DE-He213 |
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emerging links between $ m^{6} $a and misregulated mrna methylation in cancer |
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Emerging links between $ m^{6} $A and misregulated mRNA methylation in cancer |
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Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. © The Author(s). 2017 |
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Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. © The Author(s). 2017 |
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Editorial summary N6-methyladenosine ($ m^{6} $A) in mRNA has emerged as a crucial epitranscriptomic modification that controls cellular differentiation and pluripotency. Recent studies are pointing to a role for the RNA methylation program in cancer self-renewal and cell fate, making this a new and promising therapeutic avenue for investigation. © The Author(s). 2017 |
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