Functional roles of lncRNAs and its potential mechanisms in neuropathic pain

Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progre...
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Autor*in:	Tang, Simin [verfasserIn] Zhou, Jun Jing, Huan Liao, Meijuan Lin, Sen Huang, Zhenxing Huang, Teng Zhong, Jiying

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2019

Schlagwörter:	lncRNAs Neuropathic pain Spinal cord injury Peripheral nerve injury Central nerve injury

Anmerkung:	© The Author(s). 2019

Übergeordnetes Werk:	Enthalten in: Clinical epigenetics - [S.l.] : BioMed Central, 2010, 11(2019), 1 vom: 15. Mai
Übergeordnetes Werk:	volume:11 ; year:2019 ; number:1 ; day:15 ; month:05

Links:	Volltext

DOI / URN:	10.1186/s13148-019-0671-8

Katalog-ID:	SPR030678439

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520			\|a Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future.
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allfields	10.1186/s13148-019-0671-8 doi (DE-627)SPR030678439 (SPR)s13148-019-0671-8-e DE-627 ger DE-627 rakwb eng Tang, Simin verfasserin aut Functional roles of lncRNAs and its potential mechanisms in neuropathic pain 2019 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2019 Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213 Zhou, Jun aut Jing, Huan aut Liao, Meijuan aut Lin, Sen aut Huang, Zhenxing aut Huang, Teng aut Zhong, Jiying aut Enthalten in Clinical epigenetics [S.l.] : BioMed Central, 2010 11(2019), 1 vom: 15. Mai (DE-627)626459028 (DE-600)2553921-8 1868-7083 nnns volume:11 year:2019 number:1 day:15 month:05 https://dx.doi.org/10.1186/s13148-019-0671-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2019 1 15 05
spelling	10.1186/s13148-019-0671-8 doi (DE-627)SPR030678439 (SPR)s13148-019-0671-8-e DE-627 ger DE-627 rakwb eng Tang, Simin verfasserin aut Functional roles of lncRNAs and its potential mechanisms in neuropathic pain 2019 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2019 Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213 Zhou, Jun aut Jing, Huan aut Liao, Meijuan aut Lin, Sen aut Huang, Zhenxing aut Huang, Teng aut Zhong, Jiying aut Enthalten in Clinical epigenetics [S.l.] : BioMed Central, 2010 11(2019), 1 vom: 15. Mai (DE-627)626459028 (DE-600)2553921-8 1868-7083 nnns volume:11 year:2019 number:1 day:15 month:05 https://dx.doi.org/10.1186/s13148-019-0671-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2019 1 15 05
allfields_unstemmed	10.1186/s13148-019-0671-8 doi (DE-627)SPR030678439 (SPR)s13148-019-0671-8-e DE-627 ger DE-627 rakwb eng Tang, Simin verfasserin aut Functional roles of lncRNAs and its potential mechanisms in neuropathic pain 2019 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2019 Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213 Zhou, Jun aut Jing, Huan aut Liao, Meijuan aut Lin, Sen aut Huang, Zhenxing aut Huang, Teng aut Zhong, Jiying aut Enthalten in Clinical epigenetics [S.l.] : BioMed Central, 2010 11(2019), 1 vom: 15. Mai (DE-627)626459028 (DE-600)2553921-8 1868-7083 nnns volume:11 year:2019 number:1 day:15 month:05 https://dx.doi.org/10.1186/s13148-019-0671-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2019 1 15 05
allfieldsGer	10.1186/s13148-019-0671-8 doi (DE-627)SPR030678439 (SPR)s13148-019-0671-8-e DE-627 ger DE-627 rakwb eng Tang, Simin verfasserin aut Functional roles of lncRNAs and its potential mechanisms in neuropathic pain 2019 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2019 Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213 Zhou, Jun aut Jing, Huan aut Liao, Meijuan aut Lin, Sen aut Huang, Zhenxing aut Huang, Teng aut Zhong, Jiying aut Enthalten in Clinical epigenetics [S.l.] : BioMed Central, 2010 11(2019), 1 vom: 15. Mai (DE-627)626459028 (DE-600)2553921-8 1868-7083 nnns volume:11 year:2019 number:1 day:15 month:05 https://dx.doi.org/10.1186/s13148-019-0671-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2019 1 15 05
allfieldsSound	10.1186/s13148-019-0671-8 doi (DE-627)SPR030678439 (SPR)s13148-019-0671-8-e DE-627 ger DE-627 rakwb eng Tang, Simin verfasserin aut Functional roles of lncRNAs and its potential mechanisms in neuropathic pain 2019 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s). 2019 Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213 Zhou, Jun aut Jing, Huan aut Liao, Meijuan aut Lin, Sen aut Huang, Zhenxing aut Huang, Teng aut Zhong, Jiying aut Enthalten in Clinical epigenetics [S.l.] : BioMed Central, 2010 11(2019), 1 vom: 15. Mai (DE-627)626459028 (DE-600)2553921-8 1868-7083 nnns volume:11 year:2019 number:1 day:15 month:05 https://dx.doi.org/10.1186/s13148-019-0671-8 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 11 2019 1 15 05
language	English
source	Enthalten in Clinical epigenetics 11(2019), 1 vom: 15. Mai volume:11 year:2019 number:1 day:15 month:05
sourceStr	Enthalten in Clinical epigenetics 11(2019), 1 vom: 15. Mai volume:11 year:2019 number:1 day:15 month:05
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	lncRNAs Neuropathic pain Spinal cord injury Peripheral nerve injury Central nerve injury
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container_title	Clinical epigenetics
authorswithroles_txt_mv	Tang, Simin @@aut@@ Zhou, Jun @@aut@@ Jing, Huan @@aut@@ Liao, Meijuan @@aut@@ Lin, Sen @@aut@@ Huang, Zhenxing @@aut@@ Huang, Teng @@aut@@ Zhong, Jiying @@aut@@
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author	Tang, Simin
spellingShingle	Tang, Simin misc lncRNAs misc Neuropathic pain misc Spinal cord injury misc Peripheral nerve injury misc Central nerve injury Functional roles of lncRNAs and its potential mechanisms in neuropathic pain
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topic_title	Functional roles of lncRNAs and its potential mechanisms in neuropathic pain lncRNAs (dpeaa)DE-He213 Neuropathic pain (dpeaa)DE-He213 Spinal cord injury (dpeaa)DE-He213 Peripheral nerve injury (dpeaa)DE-He213 Central nerve injury (dpeaa)DE-He213
topic	misc lncRNAs misc Neuropathic pain misc Spinal cord injury misc Peripheral nerve injury misc Central nerve injury
topic_unstemmed	misc lncRNAs misc Neuropathic pain misc Spinal cord injury misc Peripheral nerve injury misc Central nerve injury
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title	Functional roles of lncRNAs and its potential mechanisms in neuropathic pain
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title_full	Functional roles of lncRNAs and its potential mechanisms in neuropathic pain
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title_sort	functional roles of lncrnas and its potential mechanisms in neuropathic pain
title_auth	Functional roles of lncRNAs and its potential mechanisms in neuropathic pain
abstract	Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. © The Author(s). 2019
abstractGer	Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. © The Author(s). 2019
abstract_unstemmed	Abstract Neuropathic pain (NP) is ranked as one of the major forms of chronic pain and emerges as a direct consequence of a lesion or disease affecting the somatosensory nervous system. Despite great advances into the mechanisms of NP, clinical practice is still not satisfactory. Fortunately, progress in elucidating unique features and multiple molecular mechanisms of long non-coding RNAs (lncRNAs) in NP has emerged in the past 10 years, suggesting that novel therapeutic strategies for pain treatment may be proposed. In this review, we will concentrate on recent studies associated with lncRNAs in NP. First, we will describe the alterations of lncRNA expression after spinal cord injury (SCI) and peripheral nerve injury (PNI), and then we illustrate the role of some specific lncRNAs in detail, which may offer new insights into our understanding of the etiology and pathophysiology of NP. Finally, we put special emphasis on the altered expression of lncRNAs in the diverse biological process of NP. Recent advances we summarized above in the development of NP may facilitate translation of these findings from bench to bedside in the future. © The Author(s). 2019
collection_details	GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700
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title_short	Functional roles of lncRNAs and its potential mechanisms in neuropathic pain
url	https://dx.doi.org/10.1186/s13148-019-0671-8
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author2	Zhou, Jun Jing, Huan Liao, Meijuan Lin, Sen Huang, Zhenxing Huang, Teng Zhong, Jiying
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Functional roles of lncRNAs and its potential mechanisms in neuropathic pain

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