B cells in rheumatoid synovitis

Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, rec...
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Gespeichert in:

Autor*in:	Weyand, Cornelia M [verfasserIn] Seyler, Thorsten M Goronzy, Jörg J

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2005

Schlagwörter:	Rheumatoid Arthritis Patient Synovial Tissue Germinal Center Germinal Center Formation Rheumatoid Synovitis

Anmerkung:	© BioMed Central Ltd 2005

Übergeordnetes Werk:	Enthalten in: Arthritis Research & Therapy - London : BioMed Central, 1999, 7(2005), Suppl 3 vom: 18. Mai
Übergeordnetes Werk:	volume:7 ; year:2005 ; number:Suppl 3 ; day:18 ; month:05

Links:	Volltext

DOI / URN:	10.1186/ar1737

Katalog-ID:	SPR030821835

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allfields	10.1186/ar1737 doi (DE-627)SPR030821835 (SPR)ar1737-e DE-627 ger DE-627 rakwb eng Weyand, Cornelia M verfasserin aut B cells in rheumatoid synovitis 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2005 Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213 Seyler, Thorsten M aut Goronzy, Jörg J aut Enthalten in Arthritis Research & Therapy London : BioMed Central, 1999 7(2005), Suppl 3 vom: 18. Mai (DE-627)326646418 (DE-600)2041668-4 1478-6354 nnns volume:7 year:2005 number:Suppl 3 day:18 month:05 https://dx.doi.org/10.1186/ar1737 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 7 2005 Suppl 3 18 05
spelling	10.1186/ar1737 doi (DE-627)SPR030821835 (SPR)ar1737-e DE-627 ger DE-627 rakwb eng Weyand, Cornelia M verfasserin aut B cells in rheumatoid synovitis 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2005 Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213 Seyler, Thorsten M aut Goronzy, Jörg J aut Enthalten in Arthritis Research & Therapy London : BioMed Central, 1999 7(2005), Suppl 3 vom: 18. Mai (DE-627)326646418 (DE-600)2041668-4 1478-6354 nnns volume:7 year:2005 number:Suppl 3 day:18 month:05 https://dx.doi.org/10.1186/ar1737 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 7 2005 Suppl 3 18 05
allfields_unstemmed	10.1186/ar1737 doi (DE-627)SPR030821835 (SPR)ar1737-e DE-627 ger DE-627 rakwb eng Weyand, Cornelia M verfasserin aut B cells in rheumatoid synovitis 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2005 Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213 Seyler, Thorsten M aut Goronzy, Jörg J aut Enthalten in Arthritis Research & Therapy London : BioMed Central, 1999 7(2005), Suppl 3 vom: 18. Mai (DE-627)326646418 (DE-600)2041668-4 1478-6354 nnns volume:7 year:2005 number:Suppl 3 day:18 month:05 https://dx.doi.org/10.1186/ar1737 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 7 2005 Suppl 3 18 05
allfieldsGer	10.1186/ar1737 doi (DE-627)SPR030821835 (SPR)ar1737-e DE-627 ger DE-627 rakwb eng Weyand, Cornelia M verfasserin aut B cells in rheumatoid synovitis 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2005 Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213 Seyler, Thorsten M aut Goronzy, Jörg J aut Enthalten in Arthritis Research & Therapy London : BioMed Central, 1999 7(2005), Suppl 3 vom: 18. Mai (DE-627)326646418 (DE-600)2041668-4 1478-6354 nnns volume:7 year:2005 number:Suppl 3 day:18 month:05 https://dx.doi.org/10.1186/ar1737 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 7 2005 Suppl 3 18 05
allfieldsSound	10.1186/ar1737 doi (DE-627)SPR030821835 (SPR)ar1737-e DE-627 ger DE-627 rakwb eng Weyand, Cornelia M verfasserin aut B cells in rheumatoid synovitis 2005 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © BioMed Central Ltd 2005 Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213 Seyler, Thorsten M aut Goronzy, Jörg J aut Enthalten in Arthritis Research & Therapy London : BioMed Central, 1999 7(2005), Suppl 3 vom: 18. Mai (DE-627)326646418 (DE-600)2041668-4 1478-6354 nnns volume:7 year:2005 number:Suppl 3 day:18 month:05 https://dx.doi.org/10.1186/ar1737 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 7 2005 Suppl 3 18 05
language	English
source	Enthalten in Arthritis Research & Therapy 7(2005), Suppl 3 vom: 18. Mai volume:7 year:2005 number:Suppl 3 day:18 month:05
sourceStr	Enthalten in Arthritis Research & Therapy 7(2005), Suppl 3 vom: 18. Mai volume:7 year:2005 number:Suppl 3 day:18 month:05
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topic_facet	Rheumatoid Arthritis Patient Synovial Tissue Germinal Center Germinal Center Formation Rheumatoid Synovitis
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container_title	Arthritis Research & Therapy
authorswithroles_txt_mv	Weyand, Cornelia M @@aut@@ Seyler, Thorsten M @@aut@@ Goronzy, Jörg J @@aut@@
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author	Weyand, Cornelia M
spellingShingle	Weyand, Cornelia M misc Rheumatoid Arthritis Patient misc Synovial Tissue misc Germinal Center misc Germinal Center Formation misc Rheumatoid Synovitis B cells in rheumatoid synovitis
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topic_title	B cells in rheumatoid synovitis Rheumatoid Arthritis Patient (dpeaa)DE-He213 Synovial Tissue (dpeaa)DE-He213 Germinal Center (dpeaa)DE-He213 Germinal Center Formation (dpeaa)DE-He213 Rheumatoid Synovitis (dpeaa)DE-He213
topic	misc Rheumatoid Arthritis Patient misc Synovial Tissue misc Germinal Center misc Germinal Center Formation misc Rheumatoid Synovitis
topic_unstemmed	misc Rheumatoid Arthritis Patient misc Synovial Tissue misc Germinal Center misc Germinal Center Formation misc Rheumatoid Synovitis
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title	B cells in rheumatoid synovitis
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title_sort	b cells in rheumatoid synovitis
title_auth	B cells in rheumatoid synovitis
abstract	Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. © BioMed Central Ltd 2005
abstractGer	Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. © BioMed Central Ltd 2005
abstract_unstemmed	Abstract In rheumatoid arthritis, T cells, B cells, macrophages, and dendritic cells invade the synovial membranes, establishing complex microstructures that promote inflammatory/tissue destructive lesions. B cell involvement has been considered to be limited to autoantibody production. However, recent studies suggest that B cells support rheumatoid disease through other mechanisms. A critical element of rheumatoid synovitis is the process of ectopic lymphoid neogenesis, with highly efficient lymphoid architectures established in a nonlymphoid tissue site. Rheumatoid synovitis recapitulates the pathways of lymph node formation, and B cells play a key role in this process. Furthermore, studies of rheumatoid lesions implanted in immunodeficient mice suggest that T cell activation in synovitis is B cell dependent, indicating the role played by B cells in presenting antigens and providing survival signals. © BioMed Central Ltd 2005
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title_short	B cells in rheumatoid synovitis
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