Anton's syndrome due to cerebrovascular disease: a case report

Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, oth...
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Gespeichert in:

Autor*in:	Maddula, Mohana [verfasserIn] Lutton, Stuart Keegan, Breffni

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2009

Schlagwörter:	Occipital Lobe Occipital Cortex Hypertensive Encephalopathy Agnosia Cortical Blindness

Anmerkung:	© Maddula et al.; licensee Cases Network Ltd. 2009

Übergeordnetes Werk:	Enthalten in: Journal of medical case reports - London : BioMed Central, 2007, 3(2009), 1 vom: 09. Sept.
Übergeordnetes Werk:	volume:3 ; year:2009 ; number:1 ; day:09 ; month:09

Links:	Volltext

DOI / URN:	10.4076/1752-1947-3-9028

Katalog-ID:	SPR031031315

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520			\|a Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation.
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allfields	10.4076/1752-1947-3-9028 doi (DE-627)SPR031031315 (SPR)1752-1947-3-9028-e DE-627 ger DE-627 rakwb eng Maddula, Mohana verfasserin aut Anton's syndrome due to cerebrovascular disease: a case report 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Maddula et al.; licensee Cases Network Ltd. 2009 Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213 Lutton, Stuart aut Keegan, Breffni aut Enthalten in Journal of medical case reports London : BioMed Central, 2007 3(2009), 1 vom: 09. Sept. (DE-627)524231389 (DE-600)2269805-X 1752-1947 nnns volume:3 year:2009 number:1 day:09 month:09 https://dx.doi.org/10.4076/1752-1947-3-9028 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009 1 09 09
spelling	10.4076/1752-1947-3-9028 doi (DE-627)SPR031031315 (SPR)1752-1947-3-9028-e DE-627 ger DE-627 rakwb eng Maddula, Mohana verfasserin aut Anton's syndrome due to cerebrovascular disease: a case report 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Maddula et al.; licensee Cases Network Ltd. 2009 Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213 Lutton, Stuart aut Keegan, Breffni aut Enthalten in Journal of medical case reports London : BioMed Central, 2007 3(2009), 1 vom: 09. Sept. (DE-627)524231389 (DE-600)2269805-X 1752-1947 nnns volume:3 year:2009 number:1 day:09 month:09 https://dx.doi.org/10.4076/1752-1947-3-9028 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009 1 09 09
allfields_unstemmed	10.4076/1752-1947-3-9028 doi (DE-627)SPR031031315 (SPR)1752-1947-3-9028-e DE-627 ger DE-627 rakwb eng Maddula, Mohana verfasserin aut Anton's syndrome due to cerebrovascular disease: a case report 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Maddula et al.; licensee Cases Network Ltd. 2009 Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213 Lutton, Stuart aut Keegan, Breffni aut Enthalten in Journal of medical case reports London : BioMed Central, 2007 3(2009), 1 vom: 09. Sept. (DE-627)524231389 (DE-600)2269805-X 1752-1947 nnns volume:3 year:2009 number:1 day:09 month:09 https://dx.doi.org/10.4076/1752-1947-3-9028 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009 1 09 09
allfieldsGer	10.4076/1752-1947-3-9028 doi (DE-627)SPR031031315 (SPR)1752-1947-3-9028-e DE-627 ger DE-627 rakwb eng Maddula, Mohana verfasserin aut Anton's syndrome due to cerebrovascular disease: a case report 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Maddula et al.; licensee Cases Network Ltd. 2009 Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213 Lutton, Stuart aut Keegan, Breffni aut Enthalten in Journal of medical case reports London : BioMed Central, 2007 3(2009), 1 vom: 09. Sept. (DE-627)524231389 (DE-600)2269805-X 1752-1947 nnns volume:3 year:2009 number:1 day:09 month:09 https://dx.doi.org/10.4076/1752-1947-3-9028 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009 1 09 09
allfieldsSound	10.4076/1752-1947-3-9028 doi (DE-627)SPR031031315 (SPR)1752-1947-3-9028-e DE-627 ger DE-627 rakwb eng Maddula, Mohana verfasserin aut Anton's syndrome due to cerebrovascular disease: a case report 2009 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Maddula et al.; licensee Cases Network Ltd. 2009 Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213 Lutton, Stuart aut Keegan, Breffni aut Enthalten in Journal of medical case reports London : BioMed Central, 2007 3(2009), 1 vom: 09. Sept. (DE-627)524231389 (DE-600)2269805-X 1752-1947 nnns volume:3 year:2009 number:1 day:09 month:09 https://dx.doi.org/10.4076/1752-1947-3-9028 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_2522 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 3 2009 1 09 09
language	English
source	Enthalten in Journal of medical case reports 3(2009), 1 vom: 09. Sept. volume:3 year:2009 number:1 day:09 month:09
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topic_facet	Occipital Lobe Occipital Cortex Hypertensive Encephalopathy Agnosia Cortical Blindness
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authorswithroles_txt_mv	Maddula, Mohana @@aut@@ Lutton, Stuart @@aut@@ Keegan, Breffni @@aut@@
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It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. 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author	Maddula, Mohana
spellingShingle	Maddula, Mohana misc Occipital Lobe misc Occipital Cortex misc Hypertensive Encephalopathy misc Agnosia misc Cortical Blindness Anton's syndrome due to cerebrovascular disease: a case report
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topic_title	Anton's syndrome due to cerebrovascular disease: a case report Occipital Lobe (dpeaa)DE-He213 Occipital Cortex (dpeaa)DE-He213 Hypertensive Encephalopathy (dpeaa)DE-He213 Agnosia (dpeaa)DE-He213 Cortical Blindness (dpeaa)DE-He213
topic	misc Occipital Lobe misc Occipital Cortex misc Hypertensive Encephalopathy misc Agnosia misc Cortical Blindness
topic_unstemmed	misc Occipital Lobe misc Occipital Cortex misc Hypertensive Encephalopathy misc Agnosia misc Cortical Blindness
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title	Anton's syndrome due to cerebrovascular disease: a case report
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title_sort	anton's syndrome due to cerebrovascular disease: a case report
title_auth	Anton's syndrome due to cerebrovascular disease: a case report
abstract	Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. © Maddula et al.; licensee Cases Network Ltd. 2009
abstractGer	Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. © Maddula et al.; licensee Cases Network Ltd. 2009
abstract_unstemmed	Introduction Anton's syndrome describes the condition in which patients deny their blindness despite objective evidence of visual loss, and moreover confabulate to support their stance. It is a rare extension of cortical blindness in which, in addition to the injury to the occipital cortex, other cortical centres are also affected, with patients typically behaving as if they were sighted. Case presentation We present a case report of an 83-year-old white woman with cortical blindness as a result of bilateral occipital lobe infarcts. Despite her obvious blindness, illustrated by her walking into objects, the patient expressed denial of visual loss and demonstrated confabulation in her accounts of her surroundings, consistent with a diagnosis of Anton's syndrome. Conclusions A suspicion of cortical blindness and Anton's syndrome should be considered in patients with atypical visual loss and evidence of occipital lobe injury. Cerebrovascular disease is the most common cause of Anton's syndrome, as in our patient. However, any condition that may result in cortical blindness can potentially lead to Anton's syndrome. Recovery of visual function will depend on the underlying aetiology, with cases due to occipital lobe infarction after cerebrovascular events being less likely to result in complete recovery. Management in these circumstances should accordingly focus on secondary prevention and rehabilitation. © Maddula et al.; licensee Cases Network Ltd. 2009
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title_short	Anton's syndrome due to cerebrovascular disease: a case report
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