Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease

Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive...
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Gespeichert in:

Autor*in:	Seravalle, Gino [verfasserIn] Mancia, Giuseppe Grassi, Guido

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2014

Schlagwörter:	Sympathetic nervous system Cardiovascular disease Target organ damage Antiadrenergic therapy Hypertension Congestive heart failure Obesity Metabolic syndrome Renal failure Renal denervation Carotid baroreceptor stimulation

Anmerkung:	© Springer International Publishing Switzerland 2014

Übergeordnetes Werk:	Enthalten in: High blood pressure & cardiovascular prevention - [S.l.] : Springer International, 2003, 21(2014), 2 vom: 01. Mai, Seite 89-105
Übergeordnetes Werk:	volume:21 ; year:2014 ; number:2 ; day:01 ; month:05 ; pages:89-105

Links:	Volltext

DOI / URN:	10.1007/s40292-014-0056-1

Katalog-ID:	SPR036381942

Internformat


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520			\|a Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects.
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650		4	\|a Renal failure \|7 (dpeaa)DE-He213
650		4	\|a Renal denervation \|7 (dpeaa)DE-He213
650		4	\|a Carotid baroreceptor stimulation \|7 (dpeaa)DE-He213
700	1		\|a Mancia, Giuseppe \|4 aut
700	1		\|a Grassi, Guido \|4 aut
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allfields	10.1007/s40292-014-0056-1 doi (DE-627)SPR036381942 (SPR)s40292-014-0056-1-e DE-627 ger DE-627 rakwb eng Seravalle, Gino verfasserin aut Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing Switzerland 2014 Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213 Mancia, Giuseppe aut Grassi, Guido aut Enthalten in High blood pressure & cardiovascular prevention [S.l.] : Springer International, 2003 21(2014), 2 vom: 01. Mai, Seite 89-105 (DE-627)490226264 (DE-600)2192466-1 1179-1985 nnns volume:21 year:2014 number:2 day:01 month:05 pages:89-105 https://dx.doi.org/10.1007/s40292-014-0056-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 21 2014 2 01 05 89-105
spelling	10.1007/s40292-014-0056-1 doi (DE-627)SPR036381942 (SPR)s40292-014-0056-1-e DE-627 ger DE-627 rakwb eng Seravalle, Gino verfasserin aut Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing Switzerland 2014 Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213 Mancia, Giuseppe aut Grassi, Guido aut Enthalten in High blood pressure & cardiovascular prevention [S.l.] : Springer International, 2003 21(2014), 2 vom: 01. Mai, Seite 89-105 (DE-627)490226264 (DE-600)2192466-1 1179-1985 nnns volume:21 year:2014 number:2 day:01 month:05 pages:89-105 https://dx.doi.org/10.1007/s40292-014-0056-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 21 2014 2 01 05 89-105
allfields_unstemmed	10.1007/s40292-014-0056-1 doi (DE-627)SPR036381942 (SPR)s40292-014-0056-1-e DE-627 ger DE-627 rakwb eng Seravalle, Gino verfasserin aut Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing Switzerland 2014 Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213 Mancia, Giuseppe aut Grassi, Guido aut Enthalten in High blood pressure & cardiovascular prevention [S.l.] : Springer International, 2003 21(2014), 2 vom: 01. Mai, Seite 89-105 (DE-627)490226264 (DE-600)2192466-1 1179-1985 nnns volume:21 year:2014 number:2 day:01 month:05 pages:89-105 https://dx.doi.org/10.1007/s40292-014-0056-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 21 2014 2 01 05 89-105
allfieldsGer	10.1007/s40292-014-0056-1 doi (DE-627)SPR036381942 (SPR)s40292-014-0056-1-e DE-627 ger DE-627 rakwb eng Seravalle, Gino verfasserin aut Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing Switzerland 2014 Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213 Mancia, Giuseppe aut Grassi, Guido aut Enthalten in High blood pressure & cardiovascular prevention [S.l.] : Springer International, 2003 21(2014), 2 vom: 01. Mai, Seite 89-105 (DE-627)490226264 (DE-600)2192466-1 1179-1985 nnns volume:21 year:2014 number:2 day:01 month:05 pages:89-105 https://dx.doi.org/10.1007/s40292-014-0056-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 21 2014 2 01 05 89-105
allfieldsSound	10.1007/s40292-014-0056-1 doi (DE-627)SPR036381942 (SPR)s40292-014-0056-1-e DE-627 ger DE-627 rakwb eng Seravalle, Gino verfasserin aut Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing Switzerland 2014 Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213 Mancia, Giuseppe aut Grassi, Guido aut Enthalten in High blood pressure & cardiovascular prevention [S.l.] : Springer International, 2003 21(2014), 2 vom: 01. Mai, Seite 89-105 (DE-627)490226264 (DE-600)2192466-1 1179-1985 nnns volume:21 year:2014 number:2 day:01 month:05 pages:89-105 https://dx.doi.org/10.1007/s40292-014-0056-1 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_206 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 21 2014 2 01 05 89-105
language	English
source	Enthalten in High blood pressure & cardiovascular prevention 21(2014), 2 vom: 01. Mai, Seite 89-105 volume:21 year:2014 number:2 day:01 month:05 pages:89-105
sourceStr	Enthalten in High blood pressure & cardiovascular prevention 21(2014), 2 vom: 01. Mai, Seite 89-105 volume:21 year:2014 number:2 day:01 month:05 pages:89-105
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	Sympathetic nervous system Cardiovascular disease Target organ damage Antiadrenergic therapy Hypertension Congestive heart failure Obesity Metabolic syndrome Renal failure Renal denervation Carotid baroreceptor stimulation
isfreeaccess_bool	false
container_title	High blood pressure & cardiovascular prevention
authorswithroles_txt_mv	Seravalle, Gino @@aut@@ Mancia, Giuseppe @@aut@@ Grassi, Guido @@aut@@
publishDateDaySort_date	2014-05-01T00:00:00Z
hierarchy_top_id	490226264
id	SPR036381942
language_de	englisch
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This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. 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author	Seravalle, Gino
spellingShingle	Seravalle, Gino misc Sympathetic nervous system misc Cardiovascular disease misc Target organ damage misc Antiadrenergic therapy misc Hypertension misc Congestive heart failure misc Obesity misc Metabolic syndrome misc Renal failure misc Renal denervation misc Carotid baroreceptor stimulation Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease
authorStr	Seravalle, Gino
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topic_title	Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease Sympathetic nervous system (dpeaa)DE-He213 Cardiovascular disease (dpeaa)DE-He213 Target organ damage (dpeaa)DE-He213 Antiadrenergic therapy (dpeaa)DE-He213 Hypertension (dpeaa)DE-He213 Congestive heart failure (dpeaa)DE-He213 Obesity (dpeaa)DE-He213 Metabolic syndrome (dpeaa)DE-He213 Renal failure (dpeaa)DE-He213 Renal denervation (dpeaa)DE-He213 Carotid baroreceptor stimulation (dpeaa)DE-He213
topic	misc Sympathetic nervous system misc Cardiovascular disease misc Target organ damage misc Antiadrenergic therapy misc Hypertension misc Congestive heart failure misc Obesity misc Metabolic syndrome misc Renal failure misc Renal denervation misc Carotid baroreceptor stimulation
topic_unstemmed	misc Sympathetic nervous system misc Cardiovascular disease misc Target organ damage misc Antiadrenergic therapy misc Hypertension misc Congestive heart failure misc Obesity misc Metabolic syndrome misc Renal failure misc Renal denervation misc Carotid baroreceptor stimulation
topic_browse	misc Sympathetic nervous system misc Cardiovascular disease misc Target organ damage misc Antiadrenergic therapy misc Hypertension misc Congestive heart failure misc Obesity misc Metabolic syndrome misc Renal failure misc Renal denervation misc Carotid baroreceptor stimulation
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title	Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease
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title_full	Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease
author_sort	Seravalle, Gino
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author_browse	Seravalle, Gino Mancia, Giuseppe Grassi, Guido
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doi_str_mv	10.1007/s40292-014-0056-1
title_sort	role of the sympathetic nervous system in hypertension and hypertension-related cardiovascular disease
title_auth	Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease
abstract	Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. © Springer International Publishing Switzerland 2014
abstractGer	Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. © Springer International Publishing Switzerland 2014
abstract_unstemmed	Abstract A number of cardiovascular disease have been shown to be characterized by a marked increase in sympathetic drive to the heart and the peripheral circulation. This is the case for essential hypertension, congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome, obstructive sleep apnea, and chronic renal disease. This review focuses on the most recent findings documenting the role of sympathetic neural factors in the development and progression of the hypertensive state as well as in the pathogenesis of hypertension-related target organ damage. It also reviews the role of sympathetic neural factors in the development of cardiovascular diseases not necessarily strictly related to the hypertensive state, such as congestive heart failure, cardiac arrhythmias, obesity, metabolic syndrome and renal failure. The paper will finally review the pharmacological and non-pharmacological interventions acting on the sympathetic drive. Emphasis will be given to the new approaches, such as renal nerves ablation and carotid baroreceptor stimulation, which have been shown to exert sympathoinhibitory effects. © Springer International Publishing Switzerland 2014
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title_short	Role of the Sympathetic Nervous System in Hypertension and Hypertension-Related Cardiovascular Disease
url	https://dx.doi.org/10.1007/s40292-014-0056-1
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author2	Mancia, Giuseppe Grassi, Guido
author2Str	Mancia, Giuseppe Grassi, Guido
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doi_str	10.1007/s40292-014-0056-1
up_date	2024-07-03T17:17:12.598Z
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