Septic arthritis: immunopathogenesis, experimental models and therapy
Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destr...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Colavite, Priscila Maria [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
2014 |
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| Schlagwörter: |
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| Anmerkung: |
© Colavite and Sartori; licensee BioMed Central Ltd. 2014 |
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| Übergeordnetes Werk: |
Enthalten in: The journal of venomous animals and toxins including tropical diseases - Botucatu : CEVAP, 1995, 20(2014), 1 vom: 06. Mai |
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| Übergeordnetes Werk: |
volume:20 ; year:2014 ; number:1 ; day:06 ; month:05 |
| Links: |
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| DOI / URN: |
10.1186/1678-9199-20-19 |
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| Katalog-ID: |
SPR036398292 |
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10.1186/1678-9199-20-19 doi (DE-627)SPR036398292 (SPR)1678-9199-20-19-e DE-627 ger DE-627 rakwb eng Colavite, Priscila Maria verfasserin aut Septic arthritis: immunopathogenesis, experimental models and therapy 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Colavite and Sartori; licensee BioMed Central Ltd. 2014 Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. Septic arthritis (dpeaa)DE-He213 Mice (dpeaa)DE-He213 Sartori, Alexandrina aut Enthalten in The journal of venomous animals and toxins including tropical diseases Botucatu : CEVAP, 1995 20(2014), 1 vom: 06. Mai (DE-627)324824459 (DE-600)2031021-3 1678-9199 nnns volume:20 year:2014 number:1 day:06 month:05 https://dx.doi.org/10.1186/1678-9199-20-19 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2014 1 06 05 |
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10.1186/1678-9199-20-19 doi (DE-627)SPR036398292 (SPR)1678-9199-20-19-e DE-627 ger DE-627 rakwb eng Colavite, Priscila Maria verfasserin aut Septic arthritis: immunopathogenesis, experimental models and therapy 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Colavite and Sartori; licensee BioMed Central Ltd. 2014 Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. Septic arthritis (dpeaa)DE-He213 Mice (dpeaa)DE-He213 Sartori, Alexandrina aut Enthalten in The journal of venomous animals and toxins including tropical diseases Botucatu : CEVAP, 1995 20(2014), 1 vom: 06. Mai (DE-627)324824459 (DE-600)2031021-3 1678-9199 nnns volume:20 year:2014 number:1 day:06 month:05 https://dx.doi.org/10.1186/1678-9199-20-19 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2014 1 06 05 |
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10.1186/1678-9199-20-19 doi (DE-627)SPR036398292 (SPR)1678-9199-20-19-e DE-627 ger DE-627 rakwb eng Colavite, Priscila Maria verfasserin aut Septic arthritis: immunopathogenesis, experimental models and therapy 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Colavite and Sartori; licensee BioMed Central Ltd. 2014 Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. Septic arthritis (dpeaa)DE-He213 Mice (dpeaa)DE-He213 Sartori, Alexandrina aut Enthalten in The journal of venomous animals and toxins including tropical diseases Botucatu : CEVAP, 1995 20(2014), 1 vom: 06. Mai (DE-627)324824459 (DE-600)2031021-3 1678-9199 nnns volume:20 year:2014 number:1 day:06 month:05 https://dx.doi.org/10.1186/1678-9199-20-19 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2014 1 06 05 |
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10.1186/1678-9199-20-19 doi (DE-627)SPR036398292 (SPR)1678-9199-20-19-e DE-627 ger DE-627 rakwb eng Colavite, Priscila Maria verfasserin aut Septic arthritis: immunopathogenesis, experimental models and therapy 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Colavite and Sartori; licensee BioMed Central Ltd. 2014 Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. Septic arthritis (dpeaa)DE-He213 Mice (dpeaa)DE-He213 Sartori, Alexandrina aut Enthalten in The journal of venomous animals and toxins including tropical diseases Botucatu : CEVAP, 1995 20(2014), 1 vom: 06. Mai (DE-627)324824459 (DE-600)2031021-3 1678-9199 nnns volume:20 year:2014 number:1 day:06 month:05 https://dx.doi.org/10.1186/1678-9199-20-19 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2014 1 06 05 |
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10.1186/1678-9199-20-19 doi (DE-627)SPR036398292 (SPR)1678-9199-20-19-e DE-627 ger DE-627 rakwb eng Colavite, Priscila Maria verfasserin aut Septic arthritis: immunopathogenesis, experimental models and therapy 2014 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Colavite and Sartori; licensee BioMed Central Ltd. 2014 Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. Septic arthritis (dpeaa)DE-He213 Mice (dpeaa)DE-He213 Sartori, Alexandrina aut Enthalten in The journal of venomous animals and toxins including tropical diseases Botucatu : CEVAP, 1995 20(2014), 1 vom: 06. Mai (DE-627)324824459 (DE-600)2031021-3 1678-9199 nnns volume:20 year:2014 number:1 day:06 month:05 https://dx.doi.org/10.1186/1678-9199-20-19 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2014 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2014 1 06 05 |
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Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. © Colavite and Sartori; licensee BioMed Central Ltd. 2014 |
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Abstract Septic arthritis is an inflammatory disease of the joints that is started by an infection whose most common agent is Staphylococcus aureus. In this review we discuss some of the most arthritogenic bacterial factors and the contribution of innate and specific immune mechanisms to joint destruction. Special emphasis is given to the induction of experimental arthritis by S. aureus in mice. The improvement of therapy by association of antibiotics with down-modulation of immunity is also included. © Colavite and Sartori; licensee BioMed Central Ltd. 2014 |
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