Blood pressure variability and reflex control in the elderly
Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic contro...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
Mancia, Giuseppe [verfasserIn] |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1995 |
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| Schlagwörter: |
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| Anmerkung: |
© Springer Internal Publishing Switzerland 1995 |
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| Übergeordnetes Werk: |
Enthalten in: Aging clinical and experimental research - Berlin : Heidelberg : Springer, 2002, 7(1995), 1 vom: Feb., Seite 3-9 |
|---|---|
| Übergeordnetes Werk: |
volume:7 ; year:1995 ; number:1 ; month:02 ; pages:3-9 |
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| DOI / URN: |
10.1007/BF03324282 |
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| 520 | |a Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) | ||
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10.1007/BF03324282 doi (DE-627)SPR036588229 (SPR)BF03324282-e DE-627 ger DE-627 rakwb eng Mancia, Giuseppe verfasserin aut Blood pressure variability and reflex control in the elderly 1995 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer Internal Publishing Switzerland 1995 Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) Aging (dpeaa)DE-He213 arterial baroreceptors (dpeaa)DE-He213 blood pressure variability (dpeaa)DE-He213 cardiopulmonary receptors (dpeaa)DE-He213 sympathetic activity (dpeaa)DE-He213 Di Rienzo, M. aut Grassi, G. aut Parati, G. aut Enthalten in Aging clinical and experimental research Berlin : Heidelberg : Springer, 2002 7(1995), 1 vom: Feb., Seite 3-9 (DE-627)369554930 (DE-600)2119282-0 1720-8319 nnns volume:7 year:1995 number:1 month:02 pages:3-9 https://dx.doi.org/10.1007/BF03324282 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA AR 7 1995 1 02 3-9 |
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10.1007/BF03324282 doi (DE-627)SPR036588229 (SPR)BF03324282-e DE-627 ger DE-627 rakwb eng Mancia, Giuseppe verfasserin aut Blood pressure variability and reflex control in the elderly 1995 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer Internal Publishing Switzerland 1995 Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) Aging (dpeaa)DE-He213 arterial baroreceptors (dpeaa)DE-He213 blood pressure variability (dpeaa)DE-He213 cardiopulmonary receptors (dpeaa)DE-He213 sympathetic activity (dpeaa)DE-He213 Di Rienzo, M. aut Grassi, G. aut Parati, G. aut Enthalten in Aging clinical and experimental research Berlin : Heidelberg : Springer, 2002 7(1995), 1 vom: Feb., Seite 3-9 (DE-627)369554930 (DE-600)2119282-0 1720-8319 nnns volume:7 year:1995 number:1 month:02 pages:3-9 https://dx.doi.org/10.1007/BF03324282 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA AR 7 1995 1 02 3-9 |
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10.1007/BF03324282 doi (DE-627)SPR036588229 (SPR)BF03324282-e DE-627 ger DE-627 rakwb eng Mancia, Giuseppe verfasserin aut Blood pressure variability and reflex control in the elderly 1995 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer Internal Publishing Switzerland 1995 Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) Aging (dpeaa)DE-He213 arterial baroreceptors (dpeaa)DE-He213 blood pressure variability (dpeaa)DE-He213 cardiopulmonary receptors (dpeaa)DE-He213 sympathetic activity (dpeaa)DE-He213 Di Rienzo, M. aut Grassi, G. aut Parati, G. aut Enthalten in Aging clinical and experimental research Berlin : Heidelberg : Springer, 2002 7(1995), 1 vom: Feb., Seite 3-9 (DE-627)369554930 (DE-600)2119282-0 1720-8319 nnns volume:7 year:1995 number:1 month:02 pages:3-9 https://dx.doi.org/10.1007/BF03324282 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA AR 7 1995 1 02 3-9 |
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10.1007/BF03324282 doi (DE-627)SPR036588229 (SPR)BF03324282-e DE-627 ger DE-627 rakwb eng Mancia, Giuseppe verfasserin aut Blood pressure variability and reflex control in the elderly 1995 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer Internal Publishing Switzerland 1995 Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) Aging (dpeaa)DE-He213 arterial baroreceptors (dpeaa)DE-He213 blood pressure variability (dpeaa)DE-He213 cardiopulmonary receptors (dpeaa)DE-He213 sympathetic activity (dpeaa)DE-He213 Di Rienzo, M. aut Grassi, G. aut Parati, G. aut Enthalten in Aging clinical and experimental research Berlin : Heidelberg : Springer, 2002 7(1995), 1 vom: Feb., Seite 3-9 (DE-627)369554930 (DE-600)2119282-0 1720-8319 nnns volume:7 year:1995 number:1 month:02 pages:3-9 https://dx.doi.org/10.1007/BF03324282 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA AR 7 1995 1 02 3-9 |
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10.1007/BF03324282 doi (DE-627)SPR036588229 (SPR)BF03324282-e DE-627 ger DE-627 rakwb eng Mancia, Giuseppe verfasserin aut Blood pressure variability and reflex control in the elderly 1995 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer Internal Publishing Switzerland 1995 Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) Aging (dpeaa)DE-He213 arterial baroreceptors (dpeaa)DE-He213 blood pressure variability (dpeaa)DE-He213 cardiopulmonary receptors (dpeaa)DE-He213 sympathetic activity (dpeaa)DE-He213 Di Rienzo, M. aut Grassi, G. aut Parati, G. aut Enthalten in Aging clinical and experimental research Berlin : Heidelberg : Springer, 2002 7(1995), 1 vom: Feb., Seite 3-9 (DE-627)369554930 (DE-600)2119282-0 1720-8319 nnns volume:7 year:1995 number:1 month:02 pages:3-9 https://dx.doi.org/10.1007/BF03324282 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA AR 7 1995 1 02 3-9 |
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Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) © Springer Internal Publishing Switzerland 1995 |
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Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) © Springer Internal Publishing Switzerland 1995 |
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Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995) © Springer Internal Publishing Switzerland 1995 |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR036588229</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20230519081440.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201007s1995 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/BF03324282</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR036588229</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)BF03324282-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Mancia, Giuseppe</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Blood pressure variability and reflex control in the elderly</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">1995</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="500" ind1=" " ind2=" "><subfield code="a">© Springer Internal Publishing Switzerland 1995</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Abstract Several lines of experimental and clinical evidence collected over the past ten years suggest that the aging process is characterized not only by profound structural cardiovascular alterations, but also by marked functional changes in the reflex mechanisms involved in the homeostatic control of the circulation. This paper will examine how aging affects baroreceptor control of the heart and sympathetic nerve traffic, providing evidence that while the former is markedly impaired in the elderly, the latter is virtually preserved. It will also discuss the age- related alterations in cardiopulmonary receptor function that, under physiological conditions, tonically inhibit sympathetic vasoconstriction tone and renin release from the kidney. Finally, evidence will be provided showing that short- and long- term blood pressure variabilities (and the different components of blood pressure variability) over 24 hours undergo major changes in the elderly. All these alterations in neural cardiovascular control mechanisms have clearcut clinical implications representing a potential marker of the increased cardiovascular risk which characterizes elderly people. (Aging Clin. Exp. Res. 7: 3–9, 1995)</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Aging</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">arterial baroreceptors</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">blood pressure variability</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">cardiopulmonary receptors</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">sympathetic activity</subfield><subfield code="7">(dpeaa)DE-He213</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Di Rienzo, M.</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Grassi, G.</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Parati, G.</subfield><subfield code="4">aut</subfield></datafield><datafield tag="773" ind1="0" ind2="8"><subfield code="i">Enthalten in</subfield><subfield code="t">Aging clinical and experimental research</subfield><subfield code="d">Berlin : Heidelberg : Springer, 2002</subfield><subfield code="g">7(1995), 1 vom: Feb., Seite 3-9</subfield><subfield code="w">(DE-627)369554930</subfield><subfield code="w">(DE-600)2119282-0</subfield><subfield code="x">1720-8319</subfield><subfield code="7">nnns</subfield></datafield><datafield tag="773" ind1="1" ind2="8"><subfield code="g">volume:7</subfield><subfield code="g">year:1995</subfield><subfield code="g">number:1</subfield><subfield code="g">month:02</subfield><subfield code="g">pages:3-9</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://dx.doi.org/10.1007/BF03324282</subfield><subfield code="z">lizenzpflichtig</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_USEFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SYSFLAG_A</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">GBV_SPRINGER</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">SSG-OLC-PHA</subfield></datafield><datafield tag="951" ind1=" " ind2=" "><subfield code="a">AR</subfield></datafield><datafield tag="952" ind1=" " ind2=" "><subfield code="d">7</subfield><subfield code="j">1995</subfield><subfield code="e">1</subfield><subfield code="c">02</subfield><subfield code="h">3-9</subfield></datafield></record></collection>
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