Etiology of developmental enamel defects in the primary dentition

Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions inclu...
Ausführliche Beschreibung

Gespeichert in:

Autor*in:	Seow, Wan Kim [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2017

Schlagwörter:	Primary teeth Dental enamel Enamel hypoplasia

Anmerkung:	© Springer International Publishing AG 2017

Übergeordnetes Werk:	Enthalten in: Clinical dentistry reviewed - [Cham] : Springer International Publishing, 2017, 1(2017), 1 vom: 09. Aug.
Übergeordnetes Werk:	volume:1 ; year:2017 ; number:1 ; day:09 ; month:08

Links:	Volltext

DOI / URN:	10.1007/s41894-017-0008-y

Katalog-ID:	SPR038328577

Internformat


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520			\|a Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries.
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912			\|a GBV_ILN_95
912			\|a GBV_ILN_100
912			\|a GBV_ILN_101
912			\|a GBV_ILN_105
912			\|a GBV_ILN_110
912			\|a GBV_ILN_120
912			\|a GBV_ILN_138
912			\|a GBV_ILN_150
912			\|a GBV_ILN_151
912			\|a GBV_ILN_161
912			\|a GBV_ILN_165
912			\|a GBV_ILN_170
912			\|a GBV_ILN_171
912			\|a GBV_ILN_187
912			\|a GBV_ILN_213
912			\|a GBV_ILN_224
912			\|a GBV_ILN_230
912			\|a GBV_ILN_250
912			\|a GBV_ILN_266
912			\|a GBV_ILN_281
912			\|a GBV_ILN_285
912			\|a GBV_ILN_293
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912			\|a GBV_ILN_636
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912			\|a GBV_ILN_2001
912			\|a GBV_ILN_2003
912			\|a GBV_ILN_2004
912			\|a GBV_ILN_2005
912			\|a GBV_ILN_2006
912			\|a GBV_ILN_2007
912			\|a GBV_ILN_2008
912			\|a GBV_ILN_2009
912			\|a GBV_ILN_2010
912			\|a GBV_ILN_2011
912			\|a GBV_ILN_2014
912			\|a GBV_ILN_2015
912			\|a GBV_ILN_2020
912			\|a GBV_ILN_2021
912			\|a GBV_ILN_2025
912			\|a GBV_ILN_2026
912			\|a GBV_ILN_2027
912			\|a GBV_ILN_2031
912			\|a GBV_ILN_2034
912			\|a GBV_ILN_2037
912			\|a GBV_ILN_2038
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912			\|a GBV_ILN_2044
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912			\|a GBV_ILN_2055
912			\|a GBV_ILN_2057
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912			\|a GBV_ILN_2088
912			\|a GBV_ILN_2093
912			\|a GBV_ILN_2106
912			\|a GBV_ILN_2107
912			\|a GBV_ILN_2108
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912			\|a GBV_ILN_2153
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912			\|a GBV_ILN_2190
912			\|a GBV_ILN_2232
912			\|a GBV_ILN_2336
912			\|a GBV_ILN_2446
912			\|a GBV_ILN_2470
912			\|a GBV_ILN_2472
912			\|a GBV_ILN_2507
912			\|a GBV_ILN_2522
912			\|a GBV_ILN_2548
912			\|a GBV_ILN_4035
912			\|a GBV_ILN_4037
912			\|a GBV_ILN_4046
912			\|a GBV_ILN_4112
912			\|a GBV_ILN_4125
912			\|a GBV_ILN_4242
912			\|a GBV_ILN_4246
912			\|a GBV_ILN_4249
912			\|a GBV_ILN_4251
912			\|a GBV_ILN_4305
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Indexfelder

author_variant	w k s wk wks
matchkey_str	article:25111965:2017----::tooyfeeomnaeaedfcsnhp
hierarchy_sort_str	2017
publishDate	2017
allfields	10.1007/s41894-017-0008-y doi (DE-627)SPR038328577 (SPR)s41894-017-0008-y-e DE-627 ger DE-627 rakwb eng Seow, Wan Kim verfasserin (orcid)0000-0002-7876-4524 aut Etiology of developmental enamel defects in the primary dentition 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing AG 2017 Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213 Enthalten in Clinical dentistry reviewed [Cham] : Springer International Publishing, 2017 1(2017), 1 vom: 09. Aug. (DE-627)875202942 (DE-600)2878431-5 2511-1965 nnns volume:1 year:2017 number:1 day:09 month:08 https://dx.doi.org/10.1007/s41894-017-0008-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_165 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 1 2017 1 09 08
spelling	10.1007/s41894-017-0008-y doi (DE-627)SPR038328577 (SPR)s41894-017-0008-y-e DE-627 ger DE-627 rakwb eng Seow, Wan Kim verfasserin (orcid)0000-0002-7876-4524 aut Etiology of developmental enamel defects in the primary dentition 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing AG 2017 Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213 Enthalten in Clinical dentistry reviewed [Cham] : Springer International Publishing, 2017 1(2017), 1 vom: 09. Aug. (DE-627)875202942 (DE-600)2878431-5 2511-1965 nnns volume:1 year:2017 number:1 day:09 month:08 https://dx.doi.org/10.1007/s41894-017-0008-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_165 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 1 2017 1 09 08
allfields_unstemmed	10.1007/s41894-017-0008-y doi (DE-627)SPR038328577 (SPR)s41894-017-0008-y-e DE-627 ger DE-627 rakwb eng Seow, Wan Kim verfasserin (orcid)0000-0002-7876-4524 aut Etiology of developmental enamel defects in the primary dentition 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing AG 2017 Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213 Enthalten in Clinical dentistry reviewed [Cham] : Springer International Publishing, 2017 1(2017), 1 vom: 09. Aug. (DE-627)875202942 (DE-600)2878431-5 2511-1965 nnns volume:1 year:2017 number:1 day:09 month:08 https://dx.doi.org/10.1007/s41894-017-0008-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_165 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 1 2017 1 09 08
allfieldsGer	10.1007/s41894-017-0008-y doi (DE-627)SPR038328577 (SPR)s41894-017-0008-y-e DE-627 ger DE-627 rakwb eng Seow, Wan Kim verfasserin (orcid)0000-0002-7876-4524 aut Etiology of developmental enamel defects in the primary dentition 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing AG 2017 Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213 Enthalten in Clinical dentistry reviewed [Cham] : Springer International Publishing, 2017 1(2017), 1 vom: 09. Aug. (DE-627)875202942 (DE-600)2878431-5 2511-1965 nnns volume:1 year:2017 number:1 day:09 month:08 https://dx.doi.org/10.1007/s41894-017-0008-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_165 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 1 2017 1 09 08
allfieldsSound	10.1007/s41894-017-0008-y doi (DE-627)SPR038328577 (SPR)s41894-017-0008-y-e DE-627 ger DE-627 rakwb eng Seow, Wan Kim verfasserin (orcid)0000-0002-7876-4524 aut Etiology of developmental enamel defects in the primary dentition 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Springer International Publishing AG 2017 Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213 Enthalten in Clinical dentistry reviewed [Cham] : Springer International Publishing, 2017 1(2017), 1 vom: 09. Aug. (DE-627)875202942 (DE-600)2878431-5 2511-1965 nnns volume:1 year:2017 number:1 day:09 month:08 https://dx.doi.org/10.1007/s41894-017-0008-y lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_165 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_266 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 1 2017 1 09 08
language	English
source	Enthalten in Clinical dentistry reviewed 1(2017), 1 vom: 09. Aug. volume:1 year:2017 number:1 day:09 month:08
sourceStr	Enthalten in Clinical dentistry reviewed 1(2017), 1 vom: 09. Aug. volume:1 year:2017 number:1 day:09 month:08
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	Primary teeth Dental enamel Enamel hypoplasia
isfreeaccess_bool	false
container_title	Clinical dentistry reviewed
authorswithroles_txt_mv	Seow, Wan Kim @@aut@@
publishDateDaySort_date	2017-08-09T00:00:00Z
hierarchy_top_id	875202942
id	SPR038328577
language_de	englisch
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author	Seow, Wan Kim
spellingShingle	Seow, Wan Kim misc Primary teeth misc Dental enamel misc Enamel hypoplasia Etiology of developmental enamel defects in the primary dentition
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topic_title	Etiology of developmental enamel defects in the primary dentition Primary teeth (dpeaa)DE-He213 Dental enamel (dpeaa)DE-He213 Enamel hypoplasia (dpeaa)DE-He213
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topic_browse	misc Primary teeth misc Dental enamel misc Enamel hypoplasia
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title	Etiology of developmental enamel defects in the primary dentition
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title_full	Etiology of developmental enamel defects in the primary dentition
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title_sort	etiology of developmental enamel defects in the primary dentition
title_auth	Etiology of developmental enamel defects in the primary dentition
abstract	Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. © Springer International Publishing AG 2017
abstractGer	Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. © Springer International Publishing AG 2017
abstract_unstemmed	Abstract Many systemic and local conditions can adversely affect the cells forming the enamel of the primary dentition, resulting in abnormalities that are permanently recorded on the tooth surface as changes in translucency (opacities) or reduced enamel quantity (hypoplasia). These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. Importantly, developmental enamel defects are now recognized as a major risk factor for early childhood caries. © Springer International Publishing AG 2017
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title_short	Etiology of developmental enamel defects in the primary dentition
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These conditions include genetic defects, such as amelogenesis imperfecta, congenital abnormalities of the liver and kidneys, premature birth, nutritional deficiencies, infections and local trauma. Developmental defects of enamel have significant clinical implications, such as reduced esthetics, tooth sensitivity and impaired masticatory function. 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Etiology of developmental enamel defects in the primary dentition

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