Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis

Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infect...
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Autor*in:	Bakhtiar, Nayer Mehdizad [verfasserIn] Spotin, Adel [verfasserIn] Mahami-Oskouei, Mahmoud [verfasserIn] Ahmadpour, Ehsan [verfasserIn] Rostami, Ali [verfasserIn]

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2020

Schlagwörter:	Apoptosis Inflammasome Innate immunity Toll-like receptors

Übergeordnetes Werk:	Enthalten in: Parasites & vectors - London : BioMed Central, 2008, 13(2020), 1 vom: 06. Mai
Übergeordnetes Werk:	volume:13 ; year:2020 ; number:1 ; day:06 ; month:05

Links:	Volltext

DOI / URN:	10.1186/s13071-020-04103-4

Katalog-ID:	SPR039626202

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matchkey_str	article:17563305:2020----::eetdacsnnaemueahaseaethsprstcosaknyt
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allfields	10.1186/s13071-020-04103-4 doi (DE-627)SPR039626202 (SPR)s13071-020-04103-4-e DE-627 ger DE-627 rakwb eng 570 ASE Bakhtiar, Nayer Mehdizad verfasserin aut Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis 2020 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis. Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213 Spotin, Adel verfasserin aut Mahami-Oskouei, Mahmoud verfasserin aut Ahmadpour, Ehsan verfasserin aut Rostami, Ali verfasserin aut Enthalten in Parasites & vectors London : BioMed Central, 2008 13(2020), 1 vom: 06. Mai (DE-627)558690076 (DE-600)2409480-8 1756-3305 nnns volume:13 year:2020 number:1 day:06 month:05 https://dx.doi.org/10.1186/s13071-020-04103-4 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2020 1 06 05
spelling	10.1186/s13071-020-04103-4 doi (DE-627)SPR039626202 (SPR)s13071-020-04103-4-e DE-627 ger DE-627 rakwb eng 570 ASE Bakhtiar, Nayer Mehdizad verfasserin aut Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis 2020 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis. Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213 Spotin, Adel verfasserin aut Mahami-Oskouei, Mahmoud verfasserin aut Ahmadpour, Ehsan verfasserin aut Rostami, Ali verfasserin aut Enthalten in Parasites & vectors London : BioMed Central, 2008 13(2020), 1 vom: 06. Mai (DE-627)558690076 (DE-600)2409480-8 1756-3305 nnns volume:13 year:2020 number:1 day:06 month:05 https://dx.doi.org/10.1186/s13071-020-04103-4 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2020 1 06 05
allfields_unstemmed	10.1186/s13071-020-04103-4 doi (DE-627)SPR039626202 (SPR)s13071-020-04103-4-e DE-627 ger DE-627 rakwb eng 570 ASE Bakhtiar, Nayer Mehdizad verfasserin aut Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis 2020 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis. Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213 Spotin, Adel verfasserin aut Mahami-Oskouei, Mahmoud verfasserin aut Ahmadpour, Ehsan verfasserin aut Rostami, Ali verfasserin aut Enthalten in Parasites & vectors London : BioMed Central, 2008 13(2020), 1 vom: 06. Mai (DE-627)558690076 (DE-600)2409480-8 1756-3305 nnns volume:13 year:2020 number:1 day:06 month:05 https://dx.doi.org/10.1186/s13071-020-04103-4 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2020 1 06 05
allfieldsGer	10.1186/s13071-020-04103-4 doi (DE-627)SPR039626202 (SPR)s13071-020-04103-4-e DE-627 ger DE-627 rakwb eng 570 ASE Bakhtiar, Nayer Mehdizad verfasserin aut Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis 2020 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis. Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213 Spotin, Adel verfasserin aut Mahami-Oskouei, Mahmoud verfasserin aut Ahmadpour, Ehsan verfasserin aut Rostami, Ali verfasserin aut Enthalten in Parasites & vectors London : BioMed Central, 2008 13(2020), 1 vom: 06. Mai (DE-627)558690076 (DE-600)2409480-8 1756-3305 nnns volume:13 year:2020 number:1 day:06 month:05 https://dx.doi.org/10.1186/s13071-020-04103-4 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2020 1 06 05
allfieldsSound	10.1186/s13071-020-04103-4 doi (DE-627)SPR039626202 (SPR)s13071-020-04103-4-e DE-627 ger DE-627 rakwb eng 570 ASE Bakhtiar, Nayer Mehdizad verfasserin aut Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis 2020 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis. Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213 Spotin, Adel verfasserin aut Mahami-Oskouei, Mahmoud verfasserin aut Ahmadpour, Ehsan verfasserin aut Rostami, Ali verfasserin aut Enthalten in Parasites & vectors London : BioMed Central, 2008 13(2020), 1 vom: 06. Mai (DE-627)558690076 (DE-600)2409480-8 1756-3305 nnns volume:13 year:2020 number:1 day:06 month:05 https://dx.doi.org/10.1186/s13071-020-04103-4 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 13 2020 1 06 05
language	English
source	Enthalten in Parasites & vectors 13(2020), 1 vom: 06. Mai volume:13 year:2020 number:1 day:06 month:05
sourceStr	Enthalten in Parasites & vectors 13(2020), 1 vom: 06. Mai volume:13 year:2020 number:1 day:06 month:05
format_phy_str_mv	Article
institution	findex.gbv.de
topic_facet	Apoptosis Inflammasome Innate immunity Toll-like receptors
dewey-raw	570
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container_title	Parasites & vectors
authorswithroles_txt_mv	Bakhtiar, Nayer Mehdizad @@aut@@ Spotin, Adel @@aut@@ Mahami-Oskouei, Mahmoud @@aut@@ Ahmadpour, Ehsan @@aut@@ Rostami, Ali @@aut@@
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author	Bakhtiar, Nayer Mehdizad
spellingShingle	Bakhtiar, Nayer Mehdizad ddc 570 misc Apoptosis misc Inflammasome misc Innate immunity misc Toll-like receptors Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
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topic_title	570 ASE Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis Apoptosis (dpeaa)DE-He213 Inflammasome (dpeaa)DE-He213 Innate immunity (dpeaa)DE-He213 Toll-like receptors (dpeaa)DE-He213
topic	ddc 570 misc Apoptosis misc Inflammasome misc Innate immunity misc Toll-like receptors
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title	Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
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title_full	Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
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author_browse	Bakhtiar, Nayer Mehdizad Spotin, Adel Mahami-Oskouei, Mahmoud Ahmadpour, Ehsan Rostami, Ali
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title_sort	recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
title_auth	Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
abstract	Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis.
abstractGer	Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis.
abstract_unstemmed	Abstract Cystic echinococcosis (CE) and alveolar echinococcosis (AE) are life-threatening parasitic infections worldwide caused by Echinococcus granulosus (sensu lato) and E. multilocularis, respectively. Very little is known about the factors affecting innate susceptibility and resistance to infection with Echinococcus spp. Although benzimidazolic drugs against CE and AE have definitively improved the treatment of these cestodes; however, the lack of successful control campaigns, including the EG95 vaccine, at a continental level indicates the importance of generating novel therapies. This review represents an update on the latest developments in the regulatory functions of innate immune pathways such as apoptosis, toll-like receptors (TLRs), and inflammasomes against CE and AE. We suggest that apoptosis can reciprocally play a bi-functional role among the host-Echinococcus metabolite relationships in suppressive and survival mechanisms of CE. Based on the available information, further studies are needed to determine whether the orchestrated in silico strategy for designing inhibitors and interfering RNA against anti-apoptotic proteins and TLRs would be effective to improve new treatments as well as therapeutic vaccines against the E. granulosus and E. multilocularis.
collection_details	GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2003 GBV_ILN_2005 GBV_ILN_2009 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2055 GBV_ILN_2111 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700
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title_short	Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis
url	https://dx.doi.org/10.1186/s13071-020-04103-4
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author2	Spotin, Adel Mahami-Oskouei, Mahmoud Ahmadpour, Ehsan Rostami, Ali
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Recent advances on innate immune pathways related to host–parasite cross-talk in cystic and alveolar echinococcosis

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