Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report
Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic...
Ausführliche Beschreibung
Autor*in: |
Aydin, Orkun [verfasserIn] |
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E-Artikel |
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Sprache: |
Englisch |
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2021 |
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Anmerkung: |
© The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 |
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Übergeordnetes Werk: |
Enthalten in: SN comprehensive clinical medicine - [Cham] : Springer International Publishing, 2019, 4(2021), 1 vom: 27. Dez. |
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Übergeordnetes Werk: |
volume:4 ; year:2021 ; number:1 ; day:27 ; month:12 |
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DOI / URN: |
10.1007/s42399-021-01088-8 |
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Katalog-ID: |
SPR045846693 |
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520 | |a Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. | ||
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10.1007/s42399-021-01088-8 doi (DE-627)SPR045846693 (SPR)s42399-021-01088-8-e DE-627 ger DE-627 rakwb eng Aydin, Orkun verfasserin (orcid)0000-0002-3155-540X aut Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. Electrolyte disorders (dpeaa)DE-He213 Intestinal malrotation (dpeaa)DE-He213 Pseudo-Bartter syndrome (dpeaa)DE-He213 Ardicli, Burak aut Kesici, Selman aut Enthalten in SN comprehensive clinical medicine [Cham] : Springer International Publishing, 2019 4(2021), 1 vom: 27. Dez. (DE-627)1037103696 (DE-600)2947252-0 2523-8973 nnns volume:4 year:2021 number:1 day:27 month:12 https://dx.doi.org/10.1007/s42399-021-01088-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 4 2021 1 27 12 |
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10.1007/s42399-021-01088-8 doi (DE-627)SPR045846693 (SPR)s42399-021-01088-8-e DE-627 ger DE-627 rakwb eng Aydin, Orkun verfasserin (orcid)0000-0002-3155-540X aut Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. Electrolyte disorders (dpeaa)DE-He213 Intestinal malrotation (dpeaa)DE-He213 Pseudo-Bartter syndrome (dpeaa)DE-He213 Ardicli, Burak aut Kesici, Selman aut Enthalten in SN comprehensive clinical medicine [Cham] : Springer International Publishing, 2019 4(2021), 1 vom: 27. Dez. (DE-627)1037103696 (DE-600)2947252-0 2523-8973 nnns volume:4 year:2021 number:1 day:27 month:12 https://dx.doi.org/10.1007/s42399-021-01088-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 4 2021 1 27 12 |
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10.1007/s42399-021-01088-8 doi (DE-627)SPR045846693 (SPR)s42399-021-01088-8-e DE-627 ger DE-627 rakwb eng Aydin, Orkun verfasserin (orcid)0000-0002-3155-540X aut Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. Electrolyte disorders (dpeaa)DE-He213 Intestinal malrotation (dpeaa)DE-He213 Pseudo-Bartter syndrome (dpeaa)DE-He213 Ardicli, Burak aut Kesici, Selman aut Enthalten in SN comprehensive clinical medicine [Cham] : Springer International Publishing, 2019 4(2021), 1 vom: 27. Dez. (DE-627)1037103696 (DE-600)2947252-0 2523-8973 nnns volume:4 year:2021 number:1 day:27 month:12 https://dx.doi.org/10.1007/s42399-021-01088-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 4 2021 1 27 12 |
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10.1007/s42399-021-01088-8 doi (DE-627)SPR045846693 (SPR)s42399-021-01088-8-e DE-627 ger DE-627 rakwb eng Aydin, Orkun verfasserin (orcid)0000-0002-3155-540X aut Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report 2021 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. Electrolyte disorders (dpeaa)DE-He213 Intestinal malrotation (dpeaa)DE-He213 Pseudo-Bartter syndrome (dpeaa)DE-He213 Ardicli, Burak aut Kesici, Selman aut Enthalten in SN comprehensive clinical medicine [Cham] : Springer International Publishing, 2019 4(2021), 1 vom: 27. Dez. (DE-627)1037103696 (DE-600)2947252-0 2523-8973 nnns volume:4 year:2021 number:1 day:27 month:12 https://dx.doi.org/10.1007/s42399-021-01088-8 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_152 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2056 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2118 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4328 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 4 2021 1 27 12 |
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Enthalten in SN comprehensive clinical medicine 4(2021), 1 vom: 27. Dez. volume:4 year:2021 number:1 day:27 month:12 |
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Aydin, Orkun @@aut@@ Ardicli, Burak @@aut@@ Kesici, Selman @@aut@@ |
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Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report Electrolyte disorders (dpeaa)DE-He213 Intestinal malrotation (dpeaa)DE-He213 Pseudo-Bartter syndrome (dpeaa)DE-He213 |
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Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report |
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Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report |
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pseudo-bartter syndrome as an atypical presentation of intestinal malrotation: a case report |
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Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report |
abstract |
Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 |
abstractGer |
Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 |
abstract_unstemmed |
Abstract Intestinal malrotation is a congenital intestinal rotation anomaly and can present with various symptoms. Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. Only two patients diagnosed with malrotation have been reported in the literature. We wanted to present this case so that the diagnosis of malrotation should be considered in patients presenting with PBS. © The Author(s), under exclusive licence to Springer Nature Switzerland AG 2021 |
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Pseudo-Bartter Syndrome as an Atypical Presentation of Intestinal Malrotation: a Case Report |
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Pseudo-Bartter syndrome (PBS), which mimics the manifestations of Bartter’s syndrome, can be caused by a severe chloride deficiency and can be seen due to cystic fibrosis, hypertrophic pyloric stenosis, diuretic abuse, and so on. We presented a 3.5-month-old boy who had convulsion after multiple vomiting, followed up in the intensive care unit, and was diagnosed with malrotation. The patient complained of vomiting 10 times and diarrhea 3–4 times a day in the last 15 days. He went to another urban hospital after having a tonic–clonic seizure. Hypochloromic hypokalemic metabolic alkalosis was detected and he was admitted to the intensive care unit of our hospital. He was examined for differential diagnosis. The intestinal malrotation was diagnosed with esophagogastroduodenography. After surgery, he made a good recovery and was discharged symptom free. Cystic fibrosis, Barter syndrome, and hypertrophic pyloric stenosis are known diagnoses in patients presenting with PBS. 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