Na fluxes in human mononuclear leucocytes
Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its grad...
Ausführliche Beschreibung
Gespeichert in:
| Autor*in: |
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| Format: |
E-Artikel |
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| Sprache: |
Englisch |
| Erschienen: |
1986 |
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| Umfang: |
3 |
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| Reproduktion: |
Springer Online Journal Archives 1860-2002 |
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| Übergeordnetes Werk: |
in: Cellular and molecular life sciences - 1945, 42(1986) vom: Nov./Dez., Seite 1262-1264 |
| Übergeordnetes Werk: |
volume:42 ; year:1986 ; month:11/12 ; pages:1262-1264 ; extent:3 |
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NLEJ189369892 |
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| 520 | |a Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. | ||
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(DE-627)NLEJ189369892 DE-627 ger DE-627 rakwb eng Na fluxes in human mononuclear leucocytes 1986 3 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. Springer Online Journal Archives 1860-2002 Brossard, M. oth Dagher, G. oth in Cellular and molecular life sciences 1945 42(1986) vom: Nov./Dez., Seite 1262-1264 (DE-627)NLEJ188987495 (DE-600)1458497-9 1420-9071 nnns volume:42 year:1986 month:11/12 pages:1262-1264 extent:3 http://dx.doi.org/10.1007/BF01946414 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 42 1986 11/12 1262-1264 3 |
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(DE-627)NLEJ189369892 DE-627 ger DE-627 rakwb eng Na fluxes in human mononuclear leucocytes 1986 3 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. Springer Online Journal Archives 1860-2002 Brossard, M. oth Dagher, G. oth in Cellular and molecular life sciences 1945 42(1986) vom: Nov./Dez., Seite 1262-1264 (DE-627)NLEJ188987495 (DE-600)1458497-9 1420-9071 nnns volume:42 year:1986 month:11/12 pages:1262-1264 extent:3 http://dx.doi.org/10.1007/BF01946414 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 42 1986 11/12 1262-1264 3 |
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(DE-627)NLEJ189369892 DE-627 ger DE-627 rakwb eng Na fluxes in human mononuclear leucocytes 1986 3 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. Springer Online Journal Archives 1860-2002 Brossard, M. oth Dagher, G. oth in Cellular and molecular life sciences 1945 42(1986) vom: Nov./Dez., Seite 1262-1264 (DE-627)NLEJ188987495 (DE-600)1458497-9 1420-9071 nnns volume:42 year:1986 month:11/12 pages:1262-1264 extent:3 http://dx.doi.org/10.1007/BF01946414 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 42 1986 11/12 1262-1264 3 |
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(DE-627)NLEJ189369892 DE-627 ger DE-627 rakwb eng Na fluxes in human mononuclear leucocytes 1986 3 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. Springer Online Journal Archives 1860-2002 Brossard, M. oth Dagher, G. oth in Cellular and molecular life sciences 1945 42(1986) vom: Nov./Dez., Seite 1262-1264 (DE-627)NLEJ188987495 (DE-600)1458497-9 1420-9071 nnns volume:42 year:1986 month:11/12 pages:1262-1264 extent:3 http://dx.doi.org/10.1007/BF01946414 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 42 1986 11/12 1262-1264 3 |
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(DE-627)NLEJ189369892 DE-627 ger DE-627 rakwb eng Na fluxes in human mononuclear leucocytes 1986 3 nicht spezifiziert zzz rdacontent nicht spezifiziert z rdamedia nicht spezifiziert zu rdacarrier Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. Springer Online Journal Archives 1860-2002 Brossard, M. oth Dagher, G. oth in Cellular and molecular life sciences 1945 42(1986) vom: Nov./Dez., Seite 1262-1264 (DE-627)NLEJ188987495 (DE-600)1458497-9 1420-9071 nnns volume:42 year:1986 month:11/12 pages:1262-1264 extent:3 http://dx.doi.org/10.1007/BF01946414 GBV_USEFLAG_U ZDB-1-SOJ GBV_NL_ARTICLE AR 42 1986 11/12 1262-1264 3 |
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na fluxes in human mononuclear leucocytes |
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Na fluxes in human mononuclear leucocytes |
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Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. |
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Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. |
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Summary Unidirectional22Na fluxes were investigated in human peripheral mononuclear leucocytes. Oua bain inhibited about 60% of Na efflux and the addition of bumetanide further reduced Na efflux rate by about 45%, sugesting the presence of a transport pathway capable of extruding Na against its gradient. Prostaglandins E1 and E2 and exogenous cAMP were found to be potent inhibitors of the bumetanide-sensitive Na efflux without affecting the ouabain-sensitive or the ouabain sensitive or the ouabain and bumetanide resistant Na effluxes. |
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