Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats
Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM...
Ausführliche Beschreibung
Autor*in: |
Wang, H. [verfasserIn] |
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E-Artikel |
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Sprache: |
Englisch |
Erschienen: |
2010 |
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Schlagwörter: |
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Anmerkung: |
© Royal Academy of Medicine in Ireland 2010 |
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Übergeordnetes Werk: |
Enthalten in: Irish journal of medical science - London : Springer, 1922, 180(2010), 2 vom: 23. Nov., Seite 483-488 |
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Übergeordnetes Werk: |
volume:180 ; year:2010 ; number:2 ; day:23 ; month:11 ; pages:483-488 |
Links: |
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DOI / URN: |
10.1007/s11845-010-0644-6 |
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Katalog-ID: |
SPR022775897 |
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100 | 1 | |a Wang, H. |e verfasserin |4 aut | |
245 | 1 | 0 | |a Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
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520 | |a Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. | ||
650 | 4 | |a Platelet endothelial cell adhesion molecule-1 |7 (dpeaa)DE-He213 | |
650 | 4 | |a Pulmonary |7 (dpeaa)DE-He213 | |
650 | 4 | |a Ischemia–reperfusion |7 (dpeaa)DE-He213 | |
700 | 1 | |a Yan, Z. |4 aut | |
700 | 1 | |a Qiu, L. |4 aut | |
700 | 1 | |a Hu, Z. |4 aut | |
700 | 1 | |a Qian, W. |4 aut | |
700 | 1 | |a Xu, L. |4 aut | |
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912 | |a GBV_ILN_2048 | ||
912 | |a GBV_ILN_2049 | ||
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912 | |a GBV_ILN_2055 | ||
912 | |a GBV_ILN_2057 | ||
912 | |a GBV_ILN_2059 | ||
912 | |a GBV_ILN_2061 | ||
912 | |a GBV_ILN_2064 | ||
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10.1007/s11845-010-0644-6 doi (DE-627)SPR022775897 (SPR)s11845-010-0644-6-e DE-627 ger DE-627 rakwb eng Wang, H. verfasserin aut Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Royal Academy of Medicine in Ireland 2010 Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 Yan, Z. aut Qiu, L. aut Hu, Z. aut Qian, W. aut Xu, L. aut Enthalten in Irish journal of medical science London : Springer, 1922 180(2010), 2 vom: 23. Nov., Seite 483-488 (DE-627)527569887 (DE-600)2275855-0 0021-1265 nnns volume:180 year:2010 number:2 day:23 month:11 pages:483-488 https://dx.doi.org/10.1007/s11845-010-0644-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 180 2010 2 23 11 483-488 |
spelling |
10.1007/s11845-010-0644-6 doi (DE-627)SPR022775897 (SPR)s11845-010-0644-6-e DE-627 ger DE-627 rakwb eng Wang, H. verfasserin aut Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Royal Academy of Medicine in Ireland 2010 Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 Yan, Z. aut Qiu, L. aut Hu, Z. aut Qian, W. aut Xu, L. aut Enthalten in Irish journal of medical science London : Springer, 1922 180(2010), 2 vom: 23. Nov., Seite 483-488 (DE-627)527569887 (DE-600)2275855-0 0021-1265 nnns volume:180 year:2010 number:2 day:23 month:11 pages:483-488 https://dx.doi.org/10.1007/s11845-010-0644-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 180 2010 2 23 11 483-488 |
allfields_unstemmed |
10.1007/s11845-010-0644-6 doi (DE-627)SPR022775897 (SPR)s11845-010-0644-6-e DE-627 ger DE-627 rakwb eng Wang, H. verfasserin aut Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Royal Academy of Medicine in Ireland 2010 Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 Yan, Z. aut Qiu, L. aut Hu, Z. aut Qian, W. aut Xu, L. aut Enthalten in Irish journal of medical science London : Springer, 1922 180(2010), 2 vom: 23. Nov., Seite 483-488 (DE-627)527569887 (DE-600)2275855-0 0021-1265 nnns volume:180 year:2010 number:2 day:23 month:11 pages:483-488 https://dx.doi.org/10.1007/s11845-010-0644-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 180 2010 2 23 11 483-488 |
allfieldsGer |
10.1007/s11845-010-0644-6 doi (DE-627)SPR022775897 (SPR)s11845-010-0644-6-e DE-627 ger DE-627 rakwb eng Wang, H. verfasserin aut Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Royal Academy of Medicine in Ireland 2010 Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 Yan, Z. aut Qiu, L. aut Hu, Z. aut Qian, W. aut Xu, L. aut Enthalten in Irish journal of medical science London : Springer, 1922 180(2010), 2 vom: 23. Nov., Seite 483-488 (DE-627)527569887 (DE-600)2275855-0 0021-1265 nnns volume:180 year:2010 number:2 day:23 month:11 pages:483-488 https://dx.doi.org/10.1007/s11845-010-0644-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 180 2010 2 23 11 483-488 |
allfieldsSound |
10.1007/s11845-010-0644-6 doi (DE-627)SPR022775897 (SPR)s11845-010-0644-6-e DE-627 ger DE-627 rakwb eng Wang, H. verfasserin aut Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats 2010 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Royal Academy of Medicine in Ireland 2010 Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 Yan, Z. aut Qiu, L. aut Hu, Z. aut Qian, W. aut Xu, L. aut Enthalten in Irish journal of medical science London : Springer, 1922 180(2010), 2 vom: 23. Nov., Seite 483-488 (DE-627)527569887 (DE-600)2275855-0 0021-1265 nnns volume:180 year:2010 number:2 day:23 month:11 pages:483-488 https://dx.doi.org/10.1007/s11845-010-0644-6 lizenzpflichtig Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER SSG-OLC-PHA GBV_ILN_11 GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_32 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_70 GBV_ILN_73 GBV_ILN_74 GBV_ILN_90 GBV_ILN_95 GBV_ILN_100 GBV_ILN_101 GBV_ILN_105 GBV_ILN_110 GBV_ILN_120 GBV_ILN_138 GBV_ILN_150 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_171 GBV_ILN_187 GBV_ILN_213 GBV_ILN_224 GBV_ILN_230 GBV_ILN_250 GBV_ILN_281 GBV_ILN_285 GBV_ILN_293 GBV_ILN_370 GBV_ILN_602 GBV_ILN_636 GBV_ILN_702 GBV_ILN_2001 GBV_ILN_2003 GBV_ILN_2004 GBV_ILN_2005 GBV_ILN_2006 GBV_ILN_2007 GBV_ILN_2008 GBV_ILN_2009 GBV_ILN_2010 GBV_ILN_2011 GBV_ILN_2014 GBV_ILN_2015 GBV_ILN_2020 GBV_ILN_2021 GBV_ILN_2025 GBV_ILN_2026 GBV_ILN_2027 GBV_ILN_2031 GBV_ILN_2034 GBV_ILN_2037 GBV_ILN_2038 GBV_ILN_2039 GBV_ILN_2044 GBV_ILN_2048 GBV_ILN_2049 GBV_ILN_2050 GBV_ILN_2055 GBV_ILN_2057 GBV_ILN_2059 GBV_ILN_2061 GBV_ILN_2064 GBV_ILN_2065 GBV_ILN_2068 GBV_ILN_2070 GBV_ILN_2086 GBV_ILN_2088 GBV_ILN_2093 GBV_ILN_2106 GBV_ILN_2107 GBV_ILN_2108 GBV_ILN_2110 GBV_ILN_2111 GBV_ILN_2112 GBV_ILN_2113 GBV_ILN_2116 GBV_ILN_2118 GBV_ILN_2119 GBV_ILN_2122 GBV_ILN_2129 GBV_ILN_2143 GBV_ILN_2144 GBV_ILN_2147 GBV_ILN_2148 GBV_ILN_2152 GBV_ILN_2153 GBV_ILN_2188 GBV_ILN_2190 GBV_ILN_2232 GBV_ILN_2336 GBV_ILN_2446 GBV_ILN_2470 GBV_ILN_2472 GBV_ILN_2507 GBV_ILN_2522 GBV_ILN_2548 GBV_ILN_4012 GBV_ILN_4035 GBV_ILN_4037 GBV_ILN_4046 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4242 GBV_ILN_4246 GBV_ILN_4249 GBV_ILN_4251 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4326 GBV_ILN_4333 GBV_ILN_4334 GBV_ILN_4335 GBV_ILN_4336 GBV_ILN_4338 GBV_ILN_4393 GBV_ILN_4700 AR 180 2010 2 23 11 483-488 |
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Enthalten in Irish journal of medical science 180(2010), 2 vom: 23. Nov., Seite 483-488 volume:180 year:2010 number:2 day:23 month:11 pages:483-488 |
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Enthalten in Irish journal of medical science 180(2010), 2 vom: 23. Nov., Seite 483-488 volume:180 year:2010 number:2 day:23 month:11 pages:483-488 |
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Platelet endothelial cell adhesion molecule-1 Pulmonary Ischemia–reperfusion |
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Irish journal of medical science |
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Wang, H. @@aut@@ Yan, Z. @@aut@@ Qiu, L. @@aut@@ Hu, Z. @@aut@@ Qian, W. @@aut@@ Xu, L. @@aut@@ |
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<?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>01000caa a22002652 4500</leader><controlfield tag="001">SPR022775897</controlfield><controlfield tag="003">DE-627</controlfield><controlfield tag="005">20231006061355.0</controlfield><controlfield tag="007">cr uuu---uuuuu</controlfield><controlfield tag="008">201006s2010 xx |||||o 00| ||eng c</controlfield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/s11845-010-0644-6</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-627)SPR022775897</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(SPR)s11845-010-0644-6-e</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-627</subfield><subfield code="b">ger</subfield><subfield code="c">DE-627</subfield><subfield code="e">rakwb</subfield></datafield><datafield tag="041" ind1=" " ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="100" ind1="1" ind2=" "><subfield code="a">Wang, H.</subfield><subfield code="e">verfasserin</subfield><subfield code="4">aut</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="c">2010</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="a">Text</subfield><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="a">Computermedien</subfield><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="a">Online-Ressource</subfield><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="500" ind1=" " ind2=" "><subfield code="a">© Royal Academy of Medicine in Ireland 2010</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. 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author |
Wang, H. |
spellingShingle |
Wang, H. misc Platelet endothelial cell adhesion molecule-1 misc Pulmonary misc Ischemia–reperfusion Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
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Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats Platelet endothelial cell adhesion molecule-1 (dpeaa)DE-He213 Pulmonary (dpeaa)DE-He213 Ischemia–reperfusion (dpeaa)DE-He213 |
topic |
misc Platelet endothelial cell adhesion molecule-1 misc Pulmonary misc Ischemia–reperfusion |
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misc Platelet endothelial cell adhesion molecule-1 misc Pulmonary misc Ischemia–reperfusion |
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misc Platelet endothelial cell adhesion molecule-1 misc Pulmonary misc Ischemia–reperfusion |
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Irish journal of medical science |
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Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
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Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
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Wang, H. |
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Irish journal of medical science |
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Wang, H. Yan, Z. Qiu, L. Hu, Z. Qian, W. Xu, L. |
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Wang, H. |
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10.1007/s11845-010-0644-6 |
title_sort |
dynamic changes of platelet endothelial cell adhesion molecule-1 (pecam-1/cd31) on pulmonary injury induced by ischemia–reperfusion in rats |
title_auth |
Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
abstract |
Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. © Royal Academy of Medicine in Ireland 2010 |
abstractGer |
Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. © Royal Academy of Medicine in Ireland 2010 |
abstract_unstemmed |
Objective To evaluate the dynamic changes of PECAM-1/CD31 on pulmonary injury induced by ischemia–reperfusion (IR) in rats. Methods An isolated lung ischemia in rats was performed for 60 min, followed separately by 0, 2, 4, 6 or 8 h for reperfusion. At the end of reperfusion, the expression of PECAM-1/CD31 was detected by using flow cytometry (FCM). Simultaneously, we examined the ultrastructure changes of cells by transmission electron microscope. Results The expressions of PECAM-1/CD31 in neutrophils and lymphocytes of the blood were increased after IR; the peak was at 2 h after IR, then the expressions were gradually decreased to control level at 6 and 8 h after IR. Ultrastructural morphological changes including proliferation of alveolar type II and apoptotic bodies were seen after IR, the most prominently at 2 h after IR. Conclusion PECAM-1/CD31 is involved in the pulmonary injury induced by IR. © Royal Academy of Medicine in Ireland 2010 |
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title_short |
Dynamic changes of platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31) on pulmonary injury induced by ischemia–reperfusion in rats |
url |
https://dx.doi.org/10.1007/s11845-010-0644-6 |
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Yan, Z. Qiu, L. Hu, Z. Qian, W. Xu, L. |
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Yan, Z. Qiu, L. Hu, Z. Qian, W. Xu, L. |
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10.1007/s11845-010-0644-6 |
up_date |
2024-07-03T14:47:10.698Z |
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score |
7.400901 |