P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers

Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 ye...
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Autor*in:	Ioakeimidis, Nikolaos [verfasserIn] Terentes-Printzios, Dimitris Georgakopoulos, Christos Vlachopoulos, Charalambos Abdelrassoul, Mohammed Gourgouli, Ioanna Magkas, Nikolaos Tousoulis, Dimitrios

Format:	E-Artikel
Sprache:	Englisch

Erschienen:	2017

Anmerkung:	© Atlantis Press 2017

Übergeordnetes Werk:	Enthalten in: Artery research - Amsterdam : Atlantis Press, 2006, 20(2017), 1 vom: Dez., Seite 89-89
Übergeordnetes Werk:	volume:20 ; year:2017 ; number:1 ; month:12 ; pages:89-89

Links:	Volltext

DOI / URN:	10.1016/j.artres.2017.10.133

Katalog-ID:	SPR054922518

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520			\|a Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method.
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700	1		\|a Tousoulis, Dimitrios \|4 aut
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allfields	10.1016/j.artres.2017.10.133 doi (DE-627)SPR054922518 (SPR)j.artres.2017.10.133-e DE-627 ger DE-627 rakwb eng Ioakeimidis, Nikolaos verfasserin aut P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Atlantis Press 2017 Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. Terentes-Printzios, Dimitris aut Georgakopoulos, Christos aut Vlachopoulos, Charalambos aut Abdelrassoul, Mohammed aut Gourgouli, Ioanna aut Magkas, Nikolaos aut Tousoulis, Dimitrios aut Enthalten in Artery research Amsterdam : Atlantis Press, 2006 20(2017), 1 vom: Dez., Seite 89-89 (DE-627)534057489 (DE-600)2364789-9 1876-4401 nnns volume:20 year:2017 number:1 month:12 pages:89-89 https://dx.doi.org/10.1016/j.artres.2017.10.133 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2004 GBV_ILN_2014 GBV_ILN_2068 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2017 1 12 89-89
spelling	10.1016/j.artres.2017.10.133 doi (DE-627)SPR054922518 (SPR)j.artres.2017.10.133-e DE-627 ger DE-627 rakwb eng Ioakeimidis, Nikolaos verfasserin aut P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Atlantis Press 2017 Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. Terentes-Printzios, Dimitris aut Georgakopoulos, Christos aut Vlachopoulos, Charalambos aut Abdelrassoul, Mohammed aut Gourgouli, Ioanna aut Magkas, Nikolaos aut Tousoulis, Dimitrios aut Enthalten in Artery research Amsterdam : Atlantis Press, 2006 20(2017), 1 vom: Dez., Seite 89-89 (DE-627)534057489 (DE-600)2364789-9 1876-4401 nnns volume:20 year:2017 number:1 month:12 pages:89-89 https://dx.doi.org/10.1016/j.artres.2017.10.133 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2004 GBV_ILN_2014 GBV_ILN_2068 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2017 1 12 89-89
allfields_unstemmed	10.1016/j.artres.2017.10.133 doi (DE-627)SPR054922518 (SPR)j.artres.2017.10.133-e DE-627 ger DE-627 rakwb eng Ioakeimidis, Nikolaos verfasserin aut P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Atlantis Press 2017 Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. Terentes-Printzios, Dimitris aut Georgakopoulos, Christos aut Vlachopoulos, Charalambos aut Abdelrassoul, Mohammed aut Gourgouli, Ioanna aut Magkas, Nikolaos aut Tousoulis, Dimitrios aut Enthalten in Artery research Amsterdam : Atlantis Press, 2006 20(2017), 1 vom: Dez., Seite 89-89 (DE-627)534057489 (DE-600)2364789-9 1876-4401 nnns volume:20 year:2017 number:1 month:12 pages:89-89 https://dx.doi.org/10.1016/j.artres.2017.10.133 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2004 GBV_ILN_2014 GBV_ILN_2068 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2017 1 12 89-89
allfieldsGer	10.1016/j.artres.2017.10.133 doi (DE-627)SPR054922518 (SPR)j.artres.2017.10.133-e DE-627 ger DE-627 rakwb eng Ioakeimidis, Nikolaos verfasserin aut P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Atlantis Press 2017 Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. Terentes-Printzios, Dimitris aut Georgakopoulos, Christos aut Vlachopoulos, Charalambos aut Abdelrassoul, Mohammed aut Gourgouli, Ioanna aut Magkas, Nikolaos aut Tousoulis, Dimitrios aut Enthalten in Artery research Amsterdam : Atlantis Press, 2006 20(2017), 1 vom: Dez., Seite 89-89 (DE-627)534057489 (DE-600)2364789-9 1876-4401 nnns volume:20 year:2017 number:1 month:12 pages:89-89 https://dx.doi.org/10.1016/j.artres.2017.10.133 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2004 GBV_ILN_2014 GBV_ILN_2068 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2017 1 12 89-89
allfieldsSound	10.1016/j.artres.2017.10.133 doi (DE-627)SPR054922518 (SPR)j.artres.2017.10.133-e DE-627 ger DE-627 rakwb eng Ioakeimidis, Nikolaos verfasserin aut P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers 2017 Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier © Atlantis Press 2017 Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. Terentes-Printzios, Dimitris aut Georgakopoulos, Christos aut Vlachopoulos, Charalambos aut Abdelrassoul, Mohammed aut Gourgouli, Ioanna aut Magkas, Nikolaos aut Tousoulis, Dimitrios aut Enthalten in Artery research Amsterdam : Atlantis Press, 2006 20(2017), 1 vom: Dez., Seite 89-89 (DE-627)534057489 (DE-600)2364789-9 1876-4401 nnns volume:20 year:2017 number:1 month:12 pages:89-89 https://dx.doi.org/10.1016/j.artres.2017.10.133 kostenfrei Volltext GBV_USEFLAG_A SYSFLAG_A GBV_SPRINGER GBV_ILN_20 GBV_ILN_22 GBV_ILN_23 GBV_ILN_24 GBV_ILN_31 GBV_ILN_39 GBV_ILN_40 GBV_ILN_60 GBV_ILN_62 GBV_ILN_63 GBV_ILN_65 GBV_ILN_69 GBV_ILN_73 GBV_ILN_74 GBV_ILN_95 GBV_ILN_105 GBV_ILN_110 GBV_ILN_151 GBV_ILN_161 GBV_ILN_170 GBV_ILN_206 GBV_ILN_213 GBV_ILN_230 GBV_ILN_285 GBV_ILN_293 GBV_ILN_602 GBV_ILN_2004 GBV_ILN_2014 GBV_ILN_2068 GBV_ILN_4012 GBV_ILN_4037 GBV_ILN_4112 GBV_ILN_4125 GBV_ILN_4126 GBV_ILN_4249 GBV_ILN_4305 GBV_ILN_4306 GBV_ILN_4307 GBV_ILN_4313 GBV_ILN_4322 GBV_ILN_4323 GBV_ILN_4324 GBV_ILN_4325 GBV_ILN_4338 GBV_ILN_4367 GBV_ILN_4700 AR 20 2017 1 12 89-89
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We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method.</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Terentes-Printzios, Dimitris</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Georgakopoulos, Christos</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Vlachopoulos, Charalambos</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Abdelrassoul, Mohammed</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Gourgouli, Ioanna</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Magkas, Nikolaos</subfield><subfield code="4">aut</subfield></datafield><datafield tag="700" 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author	Ioakeimidis, Nikolaos
spellingShingle	Ioakeimidis, Nikolaos P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers
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title	P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers
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title_full	P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers
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author_browse	Ioakeimidis, Nikolaos Terentes-Printzios, Dimitris Georgakopoulos, Christos Vlachopoulos, Charalambos Abdelrassoul, Mohammed Gourgouli, Ioanna Magkas, Nikolaos Tousoulis, Dimitrios
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title_sort	p96: acute effect of electronic cigarette smoking on pulse pressure amplification in young smokers
title_auth	P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers
abstract	Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. © Atlantis Press 2017
abstractGer	Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. © Atlantis Press 2017
abstract_unstemmed	Purpose/Background/Objectives We investigated the acute effect of electronic cigarette (EC) smoking on the aortic pressure waveform amplification. We also sought to compare the effect of EC and combustible cigarette (TC) smoking on central haemodynamics. Methods We studied 24 smokers (age: 30 ± 8 years) on 3 separate occasions: a) tobacco cigarette (nicotine content, 1.2 mg) over 5 minutes, b) EC (18 mg E-liquid) for a period of 30 minutes, and c) nothing (sham procedure) for 60 minutes. Smoking EC for 30 min (15 puffs) was chosen to mimic the common pattern of EC smoking. Results Both TC and EC smoking caused a significant increase in brachial pressures and heart rate (HR), and the differences in blood pressure (BP) and HR responses between the two smoking forms were not significant. The aortic pressures also increased significantly after smoking both TC and EC, with the greatest changes seen in the first 5 minutes after TC smoking and 15 minutes EC smoking (figures 1A- C, all P < 0.05). Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. Given the prognostic role of central haemodynamics on cardiovascular disease risk, EC may still be considered a hazardous smoking method. © Atlantis Press 2017
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title_short	P96: Acute Effect of Electronic Cigarette Smoking on Pulse Pressure Amplification in Young Smokers
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author2	Terentes-Printzios, Dimitris Georgakopoulos, Christos Vlachopoulos, Charalambos Abdelrassoul, Mohammed Gourgouli, Ioanna Magkas, Nikolaos Tousoulis, Dimitrios
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Although AIx, decreased in both two smoking forms, by applying a correction factor for changes in HR, the AIx increased significantly after TC (by 3.0% at 5 minutes, P < 0.05) and EC (by 2.9% at 15 minutes, P < 0.05) (figure 1D). Conclusions Electronic cigarette smoking exerts an unfavourable and comparable to that of TC smoking acute effect on aortic pressure waveform amplification. 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